Gene Ontology annotation through association of InterPro records with GO terms
Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
Annotation inferences using phylogenetic trees
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Electronic Gene Ontology annotations created by ARBA machine learning models
Collagen types I and III propeptides as markers of healing in chronic leg ulcers. A noninvasive method for the determination of procollagen propeptides in wound fluid--influence of growth hormone.
The effects of single doses of beta radiation on the wound healing behaviour of human Tenon's capsule fibroblasts.
Human fibroblasts with mutations in COL5A1 and COL3A1 genes do not organize collagens and fibronectin in the extracellular matrix, down-regulate alpha2beta1 integrin, and recruit alphavbeta3 Instead of alpha5beta1 integrin.
Transforming growth factor-beta1-induced hypertrophy and matrix expression in human bladder smooth muscle cells.
G to T transversion at position +5 of a splice donor site causes skipping of the preceding exon in the type III procollagen transcripts of a patient with Ehlers-Danlos syndrome type IV.
Collagens are functional, high affinity ligands for the inhibitory immune receptor LAIR-1.
A single high-affinity binding site for von Willebrand factor in collagen III, identified using synthetic triple-helical peptides.
RNAi-mediated inhibition of COL1A1 and COL3A1 in human skin fibroblasts.
Crystal structure of human type III collagen Gly991-Gly1032 cystine knot-containing peptide shows both 7/2 and 10/3 triple helical symmetries.
MMP-12 catalytic domain recognizes and cleaves at multiple sites in human skin collagen type I and type III.
Aegyptin displays high-affinity for the von Willebrand factor binding site (RGQOGVMGF) in collagen and inhibits carotid thrombus formation in vivo.
Proteomics analysis of cardiac extracellular matrix remodeling in a porcine model of ischemia/reperfusion injury.
Extracellular matrix signatures of human primary metastatic colon cancers and their metastases to liver.
Extracellular matrix remodelling in response to venous hypertension: proteomics of human varicose veins.
Glycoproteomics Reveals Decorin Peptides With Anti-Myostatin Activity in Human Atrial Fibrillation.
Bi-allelic variants in COL3A1 encoding the ligand to GPR56 are associated with cobblestone-like cortical malformation, white matter changes and cerebellar cysts.
Comprehensive proteomic characterization of stem cell-derived extracellular matrices.
Proteomic characterization of human multiple myeloma bone marrow extracellular matrix.
Evidence that the primary binding site of von Willebrand factor that mediates platelet adhesion on subendothelium is not collagen.
Characterization of the Extracellular Matrix of Normal and Diseased Tissues Using Proteomics.
Abnormal type III collagen produced by an exon-17-skipping mutation of the COL3A1 gene in Ehlers-Danlos syndrome type IV is not incorporated into the extracellular matrix.
Myocardial infarction resulting from coronary artery dissection in an adolescent with Ehlers-Danlos syndrome type IV due to a type III collagen mutation.
Type I, II, III, IV, V, and VI collagens serve as extracellular ligands for the isoforms of platelet-derived growth factor (AA, BB, and AB).
Mutations in the COL3A1 gene result in the Ehlers-Danlos syndrome type IV and alterations in the size and distribution of the major collagen fibrils of the dermis.
Interferon-gamma regulates collagen and fibronectin gene expression by transcriptional and post-transcriptional mechanisms.
Simple collagen-like peptides support platelet adhesion under static but not under flow conditions: interaction via alpha2 beta1 and von Willebrand factor with specific sequences in native collagen is a requirement to resist shear forces.
Gelatin degradation by MMP1, 2, 3, 7, 8, 9, 12, 13
Collagen type III degradation by MMP14
Collagen type III degradation by MMP1,8,9,13
Formation of collagen fibrils
Prolyl 4-hydroxylase converts collagen prolines to 4-hydroxyprolines
Removal of fibrillar collagen N-propeptides
Removal of fibrillar collagen C-propeptides
P4HB binds Collagen chains
Procollagen triple helix formation
Formation of collagen fibres
Collagen types III, IV, V, VI, VIII, IX, XVI bind integrins alpha1beta1 and alpha2beta1
FN1 binds Collagen types I-V, VII
DDR1 binds collagen type I, II, III, IV, V, XI fibrils
DCN binds collagen I, II, III, VI fibrils
VTN binds collagens II, III and V
DDR2 binds collagen type I, II, III, V, X fibrils
Collagen type III degradation by MMP15
Interaction of NCAM1 with collagens
PDGF binds to extracellular matrix proteins
OSCAR binds collagen and SP-D
Association of procollagen type III
Interaction of Smads with collagen types I, III, and V.
Regulation of collagen gene expression in the Tsk2 mouse.
The haploinsufficient Col3a1 mouse as a model for vascular Ehlers-Danlos syndrome.
Haploinsufficiency of the murine Col3a1 locus causes aortic dissection: a novel model of the vascular type of Ehlers-Danlos syndrome.
G protein-coupled receptor 56 and collagen III, a receptor-ligand pair, regulates cortical development and lamination.
Loss of Col3a1, the gene for Ehlers-Danlos syndrome type IV, results in neocortical dyslamination.
Angiotensin II promotes thoracic aortic dissections and ruptures in Col3a1 haploinsufficient mice.
Tight skin 2 mice exhibit a novel time line of events leading to increased extracellular matrix deposition and dermal fibrosis.
The Tsk2/+ mouse fibrotic phenotype is due to a gain-of-function mutation in the PIIINP segment of the Col3a1 gene.
Type III collagen affects dermal and vascular collagen fibrillogenesis and tissue integrity in a mutant Col3a1 transgenic mouse model.
Type III collagen is a key regulator of the collagen fibrillar structure and biomechanics of articular cartilage and meniscus.
A novel transgenic mouse model for corneal scar visualization.