Overall Architecture of the Intraflagellar Transport (IFT)-B Complex Containing Cluap1/IFT38 as an Essential Component of the IFT-B Peripheral Subcomplex.
RABL2 interacts with the intraflagellar transport-B complex and CEP19 and participates in ciliary assembly.
The CEP19-RABL2 GTPase Complex Binds IFT-B to Initiate Intraflagellar Transport at the Ciliary Base.
Combined Automated Annotation using Multiple IEA Methods
Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Electronic Gene Ontology annotations created by ARBA machine learning models
Defects in the IFT-B component IFT172 cause Jeune and Mainzer-Saldino syndromes in humans.
Assembly of the anterograde IFT train
The retrograde IFT train dissociates
Characterization of the intraflagellar transport complex B core: direct interaction of the IFT81 and IFT74/72 subunits.
Destabilization of the IFT-B cilia core complex due to mutations in IFT81 causes a Spectrum of Short-Rib Polydactyly Syndrome.
Annotation inferences using phylogenetic trees
Molecular basis of tubulin transport within the cilium by IFT74 and IFT81.
Gene Ontology annotation through association of InterPro records with GO terms
Intraflagellar transport-A complex mediates ciliary entry and retrograde trafficking of ciliary G protein-coupled receptors.
Chlamydomonas IFT88 and its mouse homologue, polycystic kidney disease gene tg737, are required for assembly of cilia and flagella.
Biochemically validated structural model of the 15-subunit intraflagellar transport complex IFT-B.
CCDC66 regulates primary cilium length and signaling via interactions with transition zone and axonemal proteins.
Assembly of the retrograde IFT train
The anterograde IFT train dissociates
Deep research report on IFT81 (Falcon)
Intrinsic heterogeneity of primary cilia revealed through spatial proteomics.