Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Gene Ontology annotation based on curation of immunofluorescence data

Batten's disease: clues to neuronal protein catabolism in lysosomes.

Elevated lysosomal pH in neuronal ceroid lipofuscinoses (NCLs).

The gene mutated in variant late-infantile neuronal ceroid lipofuscinosis (CLN6) and in nclf mutant mice encodes a novel predicted transmembrane protein.

Defective endoplasmic reticulum-resident membrane protein CLN6 affects lysosomal degradation of endocytosed arylsulfatase A.

CLN6, which is associated with a lysosomal storage disease, is an endoplasmic reticulum protein.

Gene expression profiling in vLINCL CLN6-deficient fibroblasts: Insights into pathobiology.

Neuronal ceroid lipofuscinosis: a common pathway?

CLN3p impacts galactosylceramide transport, raft morphology, and lipid content.

Protein product of CLN6 gene responsible for variant late-onset infantile neuronal ceroid lipofuscinosis interacts with CRMP-2.

Novel interactions of CLN5 support molecular networking between Neuronal Ceroid Lipofuscinosis proteins.

Defining the membrane proteome of NK cells.

An orthogonal proteomic survey uncovers novel Zika virus host factors.

A reference map of the human binary protein interactome.

Interactome Mapping Provides a Network of Neurodegenerative Disease Proteins and Uncovers Widespread Protein Aggregation in Affected Brains.

A CLN6-CLN8 complex recruits lysosomal enzymes at the ER for Golgi transfer.

Corrigendum to A CLN6-CLN8 complex recruits lysosomal enzymes at the ER for Golgi transfer.

Identification of CLN6 as a molecular entity of endoplasmic reticulum-driven anti-aggregate activity.

CLN3 mediates chloride efflux from lysosomes.