Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Gene Ontology annotation based on curation of immunofluorescence data
Gene Ontology annotation based on curation of intracellular localizations of expressed fusion proteins in living cells
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Electronic Gene Ontology annotations created by ARBA machine learning models
Combined Automated Annotation using Multiple IEA Methods
Membrane targeting of a Rab GTPase that fails to associate with Rab escort protein (REP) or guanine nucleotide dissociation inhibitor (GDI).
Identification and characterization of Iporin as a novel interaction partner for rab1.
MHC class II-associated proteins in B-cell exosomes and potential functional implications for exosome biogenesis.
Autophagosome formation depends on the small GTPase Rab1 and functional ER exit sites.
Family-wide characterization of the DENN domain Rab GDP-GTP exchange factors.
A structural basis for Lowe syndrome caused by mutations in the Rab-binding domain of OCRL1.
Analysis of Rab GTPase-activating proteins indicates that Rab1a/b and Rab43 are important for herpes simplex virus 1 secondary envelopment.
Protein LidA from Legionella is a Rab GTPase supereffector.
Catalytic mechanism of a mammalian Rab·RabGAP complex in atomic detail.
JAM-A associates with ZO-2, afadin, and PDZ-GEF1 to activate Rap2c and regulate epithelial barrier function.
A role of OCRL in clathrin-coated pit dynamics and uncoating revealed by studies of Lowe syndrome cells.
Small GTPase Rab2B and Its Specific Binding Protein Golgi-associated Rab2B Interactor-like 4 (GARI-L4) Regulate Golgi Morphology.
Phosphoproteomics reveals that Parkinson's disease kinase LRRK2 regulates a subset of Rab GTPases.
PITPNC1 Recruits RAB1B to the Golgi Network to Drive Malignant Secretion.
Architecture of the human interactome defines protein communities and disease networks.
Parkinson's disease-associated LRRK2-G2019S mutant acts through regulation of SERCA activity to control ER stress in astrocytes.
A reference map of the human binary protein interactome.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
OpenCell: Endogenous tagging for the cartography of human cellular organization.
The putative "switch 2" domain of the Ras-related GTPase, Rab1B, plays an essential role in the interaction with Rab escort protein.
CSNK1D phosphorylates SEC23
RAB1:GTP recruits GBF1 to the ERGIC
GBF1 recruits ARF:GDP to the ERGIC
GBF1 stimulates ARF nucleotide exchange
Active ARF recruits coatomer
ARFGAP, cargo, v-SNAREs and p24 proteins bind nascent COPI complex
ARFGAPs stimulate ARF GTPase activity
ERGIC-to-Golgi vesicles bind dynein:dynactin
Vesicle is tethered through binding GOLGA2:GORASP1, GOLGB1 and the COG complex
cis-Golgi t-SNAREs bind YKT6 on tethered vesicle
GBF1 recruits ARF:GDP to the Golgi
Active ARF recruits coatomer to the Golgi
GBF1 stimulates nucleotide exchange on ARF
RAB:GTP recruits GBF1 to the Golgi membrane
ARFGAP, cargo, vSNARES and p24 proteins bind COPI vesicles at Golgi
ARFGAPs stimulate ARF GTPase activity at the Golgi membrane
Retrograde vesicle is tethered at the ER by the NRZ complex and t-SNAREs
Retrograde COPI vesicles bind kinesin and microtubules
COPI vesicle uncoating at the ER
TBC1D20 stimulates GTPase activity of RAB1, resulting in hydrolysis of GTP
RGGT geranylgeranylates RAB proteins