Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Automatic assignment of GO terms using logical inference, based on on inter-ontology links
Electronic Gene Ontology annotations created by ARBA machine learning models
Combined Automated Annotation using Multiple IEA Methods
Parathyroid hormone-regulated production of stem cell factor in human osteoblasts and osteoblast-like cells.
Structural basis for activation of the receptor tyrosine kinase KIT by stem cell factor.
Developmental changes in human fetal testicular cell numbers and messenger ribonucleic acid levels during the second trimester.
Monocytic cells derived from human embryonic stem cells and fetal liver share common differentiation pathways and homeostatic functions.
Primary structure and functional expression of rat and human stem cell factor DNAs.
Allelic Mutations of KITLG, Encoding KIT Ligand, Cause Asymmetric and Unilateral Hearing Loss and Waardenburg Syndrome Type 2.
Insulin-like growth factor-I augments erythropoietin-induced proliferation through enhanced tyrosine phosphorylation of STAT5.
Processing of SCF isoform 1
GRB2 is indirectly recruited to p-KIT through SHP2
Phosphorylation of GAB2 by SFKs
Activation of RAS by p-KIT bound SOS1
Phosphorylation of SHP2 by SFKs
Interaction of other adapter proteins with p-KIT
Synthesis of PIP3 from PIP2 by PI3K
Phosphorylation and activation of VAV1
Disassociation and translocation of STATs to the nucleus
Phosphorylation of p-KIT on Y900 by Src kinases
LNK binds to p-KIT and inhibit downstream signaling
Indirect recruitment of PI3K to KIT via p(Y)-GAB2
Dimerization of KIT upon SCF binding
Indirect recruitment of GAB2 to p-KIT
SHP2 interacts with p-KIT
Recruitment of CBL to KIT
Direct recruitment of PI3K to p-KIT
GRB2:SOS interacts with p-KIT
Autophosphorylation of KIT
Interaction of SHP1 and KIT
Interaction of APS and p-KIT
Interaction of KIT and sSCF
Interaction of other tyrosine kinases with p-KIT
PI3K phosphorylates PIP2 to PIP3
PI3K inhibitors block PI3K catalytic activity
RAS GEFs promote RAS nucleotide exchange