Gene Ontology annotation through association of InterPro records with GO terms

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Automatic assignment of GO terms using logical inference, based on on inter-ontology links

CED-1 is a transmembrane receptor that mediates cell corpse engulfment in C. elegans.

Interaction of CED-6/GULP, an adapter protein involved in engulfment of apoptotic cells with CED-1 and CD91/low density lipoprotein receptor-related protein (LRP).

Two pathways converge at CED-10 to mediate actin rearrangement and corpse removal in C. elegans.

C. elegans Dynamin mediates the signaling of phagocytic receptor CED-1 for the engulfment and degradation of apoptotic cells.

Phagocytic receptor CED-1 initiates a signaling pathway for degrading engulfed apoptotic cells.

Unfolded protein response genes regulated by CED-1 are required for Caenorhabditis elegans innate immunity.

Genes required for the engulfment of cell corpses during programmed cell death in Caenorhabditis elegans.

An essential role for calcium flux in phagocytes for apoptotic cell engulfment and the anti-inflammatory response.

The Wnt pathway controls cell death engulfment, spindle orientation, and migration through CED-10/Rac.

Retromer is required for apoptotic cell clearance by phagocytic receptor recycling.

Caenorhabditis elegans transthyretin-like protein TTR-52 mediates recognition of apoptotic cells by the CED-1 phagocyte receptor.

Three sorting nexins drive the degradation of apoptotic cells in response to PtdIns(3)P signaling.

Structural study of TTR-52 reveals the mechanism by which a bridging molecule mediates apoptotic cell engulfment.

Both the caspase CSP-1 and a caspase-independent pathway promote programmed cell death in parallel to the canonical pathway for apoptosis in Caenorhabditis elegans.

Caspase-mediated activation of Caenorhabditis elegans CED-8 promotes apoptosis and phosphatidylserine externalization.

trim-21 promotes proteasomal degradation of CED-1 for apoptotic cell clearance in C. elegans.

Mutations affecting programmed cell deaths in the nematode Caenorhabditis elegans.

Phagocytosis promotes programmed cell death in C. elegans.

Engulfment genes cooperate with ced-3 to promote cell death in Caenorhabditis elegans.