Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on curation of immunofluorescence data

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Automatic assignment of GO terms using logical inference, based on on inter-ontology links

Electronic Gene Ontology annotations created by ARBA machine learning models

Combined Automated Annotation using Multiple IEA Methods

Cooperation between engulfment receptors: the case of ABCA1 and MEGF10.

Mutations in MEGF10, a regulator of satellite cell myogenesis, cause early onset myopathy, areflexia, respiratory distress and dysphagia (EMARDD).

MEGF10 and MEGF11 mediate homotypic interactions required for mosaic spacing of retinal neurons.

Megf10 Is a Receptor for C1Q That Mediates Clearance of Apoptotic Cells by Astrocytes.

Consequences of MEGF10 deficiency on myoblast function and Notch1 interactions.

Selective serotonin reuptake inhibitors ameliorate MEGF10 myopathy.

The mammalian Ced-1 ortholog MEGF10/KIAA1780 displays a novel adhesion pattern.

MEGF10 is a mammalian ortholog of CED-1 that interacts with clathrin assembly protein complex 2 medium chain and induces large vacuole formation.

MEGF10 functions as a receptor for the uptake of amyloid-β.

Cysteine mutations cause defective tyrosine phosphorylation in MEGF10 myopathy.

Astrocytes mediate synapse elimination through MEGF10 and MERTK pathways.

Megf10 regulates the progression of the satellite cell myogenic program.