Gene Ontology annotation through association of InterPro records with GO terms.

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot keyword mapping

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara.

Electronic Gene Ontology annotations created by ARBA machine learning models

Combined Automated Annotation using Multiple IEA Methods.

Intrinsic structural disorder and sequence features of the cell cycle inhibitor p57Kip2.

The cell cycle regulator p27Kip1 interacts with MCM7, a DNA replication licensing factor, to inhibit initiation of DNA replication.

Targeted inhibition of p57 and p15 blocks transforming growth factor beta-inhibited proliferation of primary cultured human limbal epithelial cells.

A new ubiquitin ligase involved in p57KIP2 proteolysis regulates osteoblast cell differentiation.

CDK inhibitors selectively diminish cell cycle controlled activation of the histone H4 gene promoter by p220NPAT and HiNF-P.

Mutations in the PCNA-binding domain of CDKN1C cause IMAGe syndrome.

Novel interactions of the von Hippel-Lindau (pVHL) tumor suppressor with the CDKN1 family of cell cycle inhibitors.

Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.

New functional activities for the p21 family of CDK inhibitors.

Deep research on CDKN1C function

Common and specific roles of the related CDK inhibitors p27 and p57 revealed by a knock-in mouse model.

Cooperation between the Cdk inhibitors p27(KIP1) and p57(KIP2) in the control of tissue growth and development.

p57(Kip2) cooperates with Nurr1 in developing dopamine cells.

Impaired maturation of myeloid progenitors in mice lacking novel Polycomb group protein MBT-1.

Fetal overgrowth in the Cdkn1c mouse model of Beckwith-Wiedemann syndrome.

CDKN1C negatively regulates RNA polymerase II C-terminal domain phosphorylation in an E2F1-dependent manner.

p57KIP2, a structurally distinct member of the p21CIP1 Cdk inhibitor family, is a candidate tumor suppressor gene.

CDKN1C bounded GOA, donor and interaction source evidence