Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Automatic assignment of GO terms using logical inference, based on on inter-ontology links
Combined Automated Annotation using Multiple IEA Methods
Hypodermal expression of Caenorhabditis elegans TGF-beta type I receptor SMA-6 is essential for the growth and maintenance of body length.
lon-1 regulates Caenorhabditis elegans body size downstream of the dbl-1 TGF beta signaling pathway.
The expression of TGFbeta signal transducers in the hypodermis regulates body size in C. elegans.
Cyclic GMP-dependent protein kinase EGL-4 controls body size and lifespan in C elegans.
The roles of an ephrin and a semaphorin in patterning cell-cell contacts in C. elegans sensory organ development.
Genetic screen for small body size mutants in C. elegans reveals many TGFbeta pathway components.
C. elegans serine-threonine kinase KIN-29 modulates TGFbeta signaling and regulates body size formation.
Glypican LON-2 is a conserved negative regulator of BMP-like signaling in Caenorhabditis elegans.
Neuroimmune regulation of antimicrobial peptide expression by a noncanonical TGF-beta signaling pathway in Caenorhabditis elegans epidermis.
CEH-28 activates dbl-1 expression and TGF-β signaling in the C. elegans M4 neuron.
Orthosiphon stamineus protects Caenorhabditis elegans against Staphylococcus aureus infection through immunomodulation.
Promotion of bone morphogenetic protein signaling by tetraspanins and glycosphingolipids.
Two Paralogous Tetraspanins TSP-12 and TSP-14 Function with the ADAM10 Metalloprotease SUP-17 to Promote BMP Signaling in Caenorhabditis elegans.
Specificity of TGFbeta signaling is conferred by distinct type I receptors and their associated SMAD proteins in Caenorhabditis elegans.
BMP signaling requires retromer-dependent recycling of the type I receptor.
Caenorhabditis elegans SMA-10/LRIG is a conserved transmembrane protein that enhances bone morphogenetic protein signaling.
Deep research report on sma-6 (Falcon)
TGF-beta Sma/Mab signaling mutations uncouple reproductive aging from somatic aging.
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sma-6 mutants, like other Sma/Mab pathway mutants, substantially extend reproductive span.
"the rest of the mutants in this pathway (sma-3, sma-4, sma-6, and sma-9) also increased reproductive span substantially"
WormBase gene description for sma-6 (worm)