Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot keyword mapping
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping
Complementary signaling pathways regulate the unfolded protein response and are required for C. elegans development.
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XBP-1 is activated by IRE-1-mediated unconventional splicing
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XBP-1 is required for UPR gene transcription
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xbp-1 and pek-1 have complementary essential functions in development
Genetic interactions due to constitutive and inducible gene regulation mediated by the unfolded protein response in C. elegans.
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IRE-1 and XBP-1 regulate most inducible UPR genes
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PEK-1 contributes to approximately 23% of i-UPR gene induction
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ATF-6 complements IRE-1/XBP-1 pathway
An essential role for XBP-1 in host protection against immune activation in C. elegans.
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XBP-1 protects against ER stress from innate immune activation
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xbp-1 mutants show disrupted ER morphology on P. aeruginosa
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XBP-1 role is to protect host from self-inflicted ER stress during immune response
Physiological IRE-1-XBP-1 and PEK-1 signaling in Caenorhabditis elegans larval development and immunity.
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XBP-1 deficiency causes constitutive ER stress
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Temperature-dependent requirement for XBP-1 and PEK-1
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UPR signaling protects against immune activation and heat stress
XBP-1 is a cell-nonautonomous regulator of stress resistance and longevity.
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Neuronal XBP-1s activates UPR in distal tissues
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XBP-1s increases longevity through cell-nonautonomous signaling
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XBP-1s rescues age-onset loss of ER proteostasis
Integration of the unfolded protein and oxidative stress responses through SKN-1/Nrf.
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SKN-1 and XBP-1 bind common downstream targets
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ChIP evidence for XBP-1 at UPR gene promoters
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Integration of UPR and oxidative stress responses
Developmental defects in a Caenorhabditis elegans model for type III galactosemia.
Deep research report on xbp-1