Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Gene Ontology annotation based on curation of immunofluorescence data
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Gene Ontology annotation of human sequence-specific DNA binding transcription factors (DbTFs) based on the TFClass database
A protein-protein interaction network for human inherited ataxias and disorders of Purkinje cell degeneration.
Opposing effects of polyglutamine expansion on native protein complexes contribute to SCA1.
Quantitative interaction proteomics of neurodegenerative disease proteins.
Architecture of the human interactome defines protein communities and disease networks.
An AP-MS- and BioID-compatible MAC-tag enables comprehensive mapping of protein interactions and subcellular localizations.
A reference map of the human binary protein interactome.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
Disruption of the ATXN1-CIC complex causes a spectrum of neurobehavioral phenotypes in mice and humans.
A new mode of DNA binding distinguishes Capicua from other HMG-box factors and explains its mutation patterns in cancer.
Molecular basis of DNA recognition by the HMG-box-C1 module of capicua.
ATAXIN-1 interacts with the repressor Capicua in its native complex to cause SCA1 neuropathology.
Partial loss of ataxin-1 function contributes to transcriptional dysregulation in spinocerebellar ataxia type 1 pathogenesis.
ATXN1 protein family and CIC regulate extracellular matrix remodeling and lung alveolarization.