Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on curation of immunofluorescence data

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Automatic assignment of GO terms using logical inference, based on on inter-ontology links

Gene Ontology annotation of human sequence-specific DNA binding transcription factors (DbTFs) based on the TFClass database

Electronic Gene Ontology annotations created by ARBA machine learning models

Combined Automated Annotation using Multiple IEA Methods

MSX1 mutation is associated with orofacial clefting and tooth agenesis in humans.

Evidence of a sex-dependent association between the MSX1 locus and nonsyndromic cleft lip with or without cleft palate in the Chilean population.

A nonsense mutation in MSX1 causes Witkop syndrome.

MSX1 and TGFB3 contribute to clefting in South America.

Homeobox Msx1 interacts with p53 tumor suppressor and inhibits tumor growth by inducing apoptosis.

The formation and function of the cardiac conduction system.

Impact of cytosine methylation on DNA binding specificities of human transcription factors.

A reference map of the human binary protein interactome.

A human MSX1 homeodomain missense mutation causes selective tooth agenesis.

Regulation of Msx genes by a Bmp gradient is essential for neural crest specification.

Msx1 and Pax3 cooperate to mediate FGF8 and WNT signals during Xenopus neural crest induction.

Msx1 and Msx2 have shared essential functions in neural crest but may be dispensable in epidermis and axis formation in Xenopus.

Combined deficiencies of Msx1 and Msx2 cause impaired patterning and survival of the cranial neural crest.

Msx1 deficient mice exhibit cleft palate and abnormalities of craniofacial and tooth development.

Haploinsufficiency of MSX1: a mechanism for selective tooth agenesis.

Transcriptional repression by Msx-1 does not require homeodomain DNA-binding sites.

Comparison of MSX-1 and MSX-2 suggests a molecular basis for functional redundancy.

PIAS1 confers DNA-binding specificity on the Msx1 homeoprotein.

Msx1 and Msx2 promote meiosis initiation.

BMP4 rescues a non-cell-autonomous function of Msx1 in tooth development.

Msx1 regulates proliferation and differentiation of mouse dental mesenchymal cells in culture.

Homeobox protein MSX-1 inhibits expression of bone morphogenetic protein 2, bone morphogenetic protein 4, and lymphoid enhancer-binding factor 1 via Wnt/β-catenin signaling to prevent differentiation of dental mesenchymal cells during the late bell stage.

Msx1 and Msx2 are functional interacting partners of T-box factors in the regulation of Connexin43.

The posteriorizing gene Gbx2 is a direct target of Wnt signalling and the earliest factor in neural crest induction.

Insights from the amphioxus genome on the origin of vertebrate neural crest.

Shared features of blastula and neural crest stem cells evolved at the base of vertebrates.

Pax3 and Zic1 drive induction and differentiation of multipotent, migratory, and functional neural crest in Xenopus embryos.

Pax3 and Zic1 trigger the early neural crest gene regulatory network by the direct activation of multiple key neural crest specifiers.

Deep research report on human MSX1 (Falcon)