GO_REF:0000024
Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
GO_REF:0000033
Annotation inferences using phylogenetic trees
GO_REF:0000044
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
GO_REF:0000052
Gene Ontology annotation based on curation of immunofluorescence data
GO_REF:0000107
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
GO_REF:0000120
Combined Automated Annotation using Multiple IEA Methods
PMID:12127981
Identification of putative mammalian D-lactate dehydrogenase enzymes.
PMID:12507422
The cardiac mechanical stretch sensor machinery involves a Z disc complex that is defective in a subset of human dilated cardiomyopathy.
PMID:15205937
Decreased interactions of mutant muscle LIM protein (MLP) with N-RAP and alpha-actinin and their implication for hypertrophic cardiomyopathy.
PMID:15582318
Tcap gene mutations in hypertrophic cardiomyopathy and dilated cardiomyopathy.
PMID:24860983
Muscle lim protein isoform negatively regulates striated muscle actin dynamics and differentiation.
PMID:32814053
Interactome Mapping Provides a Network of Neurodegenerative Disease Proteins and Uncovers Widespread Protein Aggregation in Affected Brains.
PMID:7954791
Muscle LIM protein, a novel essential regulator of myogenesis, promotes myogenic differentiation.
PMID:9234731
Muscle LIM protein promotes myogenesis by enhancing the activity of MyoD.
PMID:19752190
Muscle LIM protein interacts with cofilin 2 and regulates F-actin dynamics in cardiac and skeletal muscle.
PMID:24934443
Human muscle LIM protein dimerizes along the actin cytoskeleton and cross-links actin filaments.
PMID:27353086
MLP and CARP are linked to chronic PKCα signalling in dilated cardiomyopathy.
PMID:10555147
Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy.