Annotation inferences using phylogenetic trees
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Electronic Gene Ontology annotations created by ARBA machine learning models
Combined Automated Annotation using Multiple IEA Methods
Interaction of Myc-associated zinc finger protein with DCC, the product of a tumor-suppressor gene, during the neural differentiation of P19 EC cells.
Mediation of the DCC apoptotic signal by DIP13 alpha.
The dependence receptor DCC requires lipid raft localization for cell death signaling.
DSCAM is a netrin receptor that collaborates with DCC in mediating turning responses to netrin-1.
Bcl11A/CTIP1 regulates expression of DCC and MAP1b in control of axon branching and dendrite outgrowth.
Netrin-1 up-regulation in inflammatory bowel diseases is required for colorectal cancer progression.
Transmembrane receptor DCC associates with protein synthesis machinery and regulates translation.
Structural basis of cargo recognition by the myosin-X MyTH4-FERM domain.
Different levels of the Tripartite motif protein, Anomalies in sensory axon patterning (Asap), regulate distinct axonal projections of Drosophila sensory neurons.
A Floor-Plate Extracellular Protein-Protein Interaction Screen Identifies Draxin as a Secreted Netrin-1 Antagonist.
A Human IgSF Cell-Surface Interactome Reveals a Complex Network of Protein-Protein Interactions.
Deleted in Colorectal Cancer (DCC) encodes a netrin receptor.
The DCC gene product induces apoptosis by a mechanism requiring receptor proteolysis.
Caspase-9 binds DCC:DIP13alpha complex
Netrin-1 induced DCC clustering
DCC interaction with Netrin-1
DCC heterodimerizes with UNC-5:Netrin-1
DCC interacts with DIP13alpha
DCC interaction with Ezrin
DCC interaction with SIAH1
DCC interaction with SIAH2
Phosphorylation of DCC by Fyn
RhoGTPase GEF's recruited to DCC
Recruitment of Src and Fyn to DCC:FADK1
Recruitment and activation of N-WASP by Cdc42
The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation.
Molecular control of spinal accessory motor neuron/axon development in the mouse spinal cord.
Lrig2 Negatively Regulates Ectodomain Shedding of Axon Guidance Receptors by ADAM Proteases.
DCC expression by neurons regulates synaptic plasticity in the adult brain.
Netrin-1 and DCC mediate axon guidance locally at the optic disc: loss of function leads to optic nerve hypoplasia.
Mammalian homologs of seven in absentia regulate DCC via the ubiquitin-proteasome pathway.
DCC source and assay evidence guide