Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Electronic Gene Ontology annotations created by ARBA machine learning models

Elastic fibres in health and disease.

Three novel collagen VI chains, alpha4(VI), alpha5(VI), and alpha6(VI).

Large-scale proteomics and phosphoproteomics of urinary exosomes.

Proteomics characterization of extracellular space components in the human aorta.

Proteomics analysis of cardiac extracellular matrix remodeling in a porcine model of ischemia/reperfusion injury.

In-depth proteomic analyses of exosomes isolated from expressed prostatic secretions in urine.

Comparative proteomic analysis of supportive and unsupportive extracellular matrix substrates for human embryonic stem cell maintenance.

The expanded collagen VI family: new chains and new questions.

Proteomic analysis of cerebrospinal fluid extracellular vesicles: a comprehensive dataset.

Extracellular matrix signatures of human primary metastatic colon cancers and their metastases to liver.

Extracellular matrix remodelling in response to venous hypertension: proteomics of human varicose veins.

Glycoproteomics Reveals Decorin Peptides With Anti-Myostatin Activity in Human Atrial Fibrillation.

Comprehensive proteomic characterization of stem cell-derived extracellular matrices.

Characterization of the Extracellular Matrix of Normal and Diseased Tissues Using Proteomics.

Type VI collagen mutations in Bethlem myopathy, an autosomal dominant myopathy with contractures.

Collagen type VI degradation by MMP2,9,11

Dimerization of procollagen type VI

Tetramerization of procollagen VI

Procollagen triple helix formation

Secretion of collagens

Formation of collagen networks

Collagen types III, IV, V, VI, VIII, IX, XVI bind integrins alpha1beta1 and alpha2beta1

DCN binds collagen I, II, III, VI fibrils

BGN binds Collagen types I, VI, (IX)

Interaction of NCAM1 with collagens

PDGF binds to extracellular matrix proteins

Collagen types VI, IX bind integrin alpha10beta1

Association of procollagen type VI

The N-terminal N5 subdomain of the alpha 3(VI) chain is important for collagen VI microfibril formation.

The human type VI collagen gene. mRNA and protein variants of the alpha 3 chain generated by alternative splicing of an additional 5-end exon.

Structure of recombinant N-terminal globule of type VI collagen alpha 3 chain and its binding to heparin and hyaluronan.

Complete sequencing and characterization of 21,243 full-length human cDNAs.

The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC).

The C5 domain of the collagen VI alpha3(VI) chain is critical for extracellular microfibril formation and is present in the extracellular matrix of cultured cells.

Mosaic structure of globular domains in the human type VI collagen alpha 3 chain: similarity to von Willebrand factor, fibronectin, actin, salivary proteins and aprotinin type protease inhibitors.

Molecular consequences of dominant Bethlem myopathy collagen VI mutations.

Adipocyte-derived endotrophin promotes malignant tumor progression.

C-terminal proteolysis of the collagen VI α3 chain by BMP-1 and proprotein convertase(s) releases endotrophin in fragments of different sizes.

Lack of evidence for a role of anthrax toxin receptors as surface receptors for collagen VI and for its cleaved-off C5 domain/endotrophin.

The 1.6 A structure of Kunitz-type domain from the alpha 3 chain of human type VI collagen.

Conversion of the Kunitz-type module of collagen VI into a highly active trypsin inhibitor by site-directed mutagenesis.

Structure and multiple conformations of the kunitz-type domain from human type VI collagen alpha3(VI) chain in solution.

Solution structure and backbone dynamics of the human alpha3-chain type VI collagen C-terminal Kunitz domain,

Hyperglycemic glucose concentrations up-regulate the expression of type VI collagen in vitro. Relevance to alterations of peripheral nerves in diabetes mellitus.