Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Gene Ontology annotation based on curation of immunofluorescence data

Gene Ontology annotation based on curation of intracellular localizations of expressed fusion proteins in living cells

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Combined Automated Annotation using Multiple IEA Methods

Purification and characterization of the human elongator complex.

Human Elongator facilitates RNA polymerase II transcription through chromatin.

Elongator controls the migration and differentiation of cortical neurons through acetylation of alpha-tubulin.

Systematic analysis of protein pools, isoforms, and modifications affecting turnover and subcellular localization.

DERP6 (ELP5) and C3ORF75 (ELP6) regulate tumorigenicity and migration of melanoma cells as subunits of Elongator.

The Many Faces of Elongator in Neurodevelopment and Disease.

Structural insights into the function of Elongator.

Elongator subunit 3 (ELP3) modifies ALS through tRNA modification.

Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.

Archaeal Elp3 catalyzes tRNA wobble uridine modification at C5 via a radical mechanism.

The Elongator subunit Elp3 is a non-canonical tRNA acetyltransferase.

Structural basis for tRNA modification by Elp3 from Dehalococcoides mccartyi.

An early step in wobble uridine tRNA modification requires the Elongator complex.

Elevated levels of two tRNA species bypass the requirement for elongator complex in transcription and exocytosis.

Variants of the elongator protein 3 (ELP3) gene are associated with motor neuron degeneration.

ELP3 controls active zone morphology by acetylating the ELKS family member Bruchpilot.

Loss of Elp3 Impairs the Acetylation and Distribution of Connexin-43 in the Developing Cerebral Cortex.

UniProt entry for human ELP3 (Q9H9T3)