Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Electronic Gene Ontology annotations created by ARBA machine learning models

Combined Automated Annotation using Multiple IEA Methods

Falcon deep research report for MYO7A (human)

Mutations of the protocadherin gene PCDH15 cause Usher syndrome type 1F.

MyRIP, a novel Rab effector, enables myosin VIIa recruitment to retinal melanosomes.

Impaired calmodulin binding of myosin-7A causes autosomal dominant hearing loss (DFNA11).

The unconventional myosin-VIIa associates with lysosomes.

Function of MYO7A in the human RPE and the validity of shaker1 mice as a model for Usher syndrome 1B.

Functional characterization of the human myosin-7a motor domain.

Myosin VIIa and sans localization at stereocilia upper tip-link density implicates these Usher syndrome proteins in mechanotransduction.

Alterations of the CIB2 calcium- and integrin-binding protein cause Usher syndrome type 1J and nonsyndromic deafness DFNB48.

The giant spectrin βV couples the molecular motors to phototransduction and Usher syndrome type I proteins along their trafficking route.

Defective myosin VIIA gene responsible for Usher syndrome type 1B.

Human Usher 1B/mouse shaker-1: the retinal phenotype discrepancy explained by the presence/absence of myosin VIIA in the photoreceptor cells.