Falcon (Edison Scientific) deep research report for WRKY33
Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
The WRKY superfamily of plant transcription factors.
Arabidopsis transcription factors: genome-wide comparative analysis among eukaryotes.
The MAP kinase substrate MKS1 is a regulator of plant defense responses.
Arabidopsis WRKY33 transcription factor is required for resistance to necrotrophic fungal pathogens.
Arabidopsis MAP kinase 4 regulates gene expression through transcription factor release in the nucleus.
Functional characterization of Arabidopsis NaCl-inducible WRKY25 and WRKY33 transcription factors in abiotic stresses.
Arabidopsis MKS1 is involved in basal immunity and requires an intact N-terminal domain for proper function.
Arabidopsis thaliana WRKY25, WRKY26, and WRKY33 coordinate induction of plant thermotolerance.
A critical role of autophagy in plant resistance to necrotrophic fungal pathogens.
Arabidopsis sigma factor binding proteins are activators of the WRKY33 transcription factor in plant defense.
Structural and functional analysis of VQ motif-containing proteins in Arabidopsis as interacting proteins of WRKY transcription factors.
Promoter-based integration in plant defense regulation.
An Arabidopsis gene regulatory network for secondary cell wall synthesis.
Establishment of Expression in the SHORTROOT-SCARECROW Transcriptional Cascade through Opposing Activities of Both Activators and Repressors.
A MPK3/6-WRKY33-ALD1-Pipecolic Acid Regulatory Loop Contributes to Systemic Acquired Resistance.
Transcriptional regulation of nitrogen-associated metabolism and growth.
Phosphorylation of a WRKY transcription factor by two pathogen-responsive MAPKs drives phytoalexin biosynthesis in Arabidopsis.
Arabidopsis WRKY33 is a key transcriptional regulator of hormonal and metabolic responses toward Botrytis cinerea infection.