SEP1 (AGL2, At5g15800, P29382) notes

2026-10-05 — initial review (floral_organ_identity_abce module)

• Identity: UniProt P29382 SEP1_ARATH, gene SEP1/AGL2, At5g15800. Checked against the uniprot file.
• Deep research: just deep-research-falcon ARATH SEP1 failed (HTTP 402 from the provider). No deep-research file was made, so the review uses the cached primary literature.
• E-class function: the sep1 sep2 sep3 triple mutant makes sepals in every whorl PMID:10821278. Adding SEP4 converts organs to leaf-like organs PMID:15530395.
• DNA binding: AGL2 binds CArG DNA as a dimer PMID:8597661.
• Expression is in the whole floral meristem and all four organ primordia PMID:7948914. The GOA IDA "flower development" row rests on this expression study. I changed it (MODIFY) to specification of floral organ identity, which the genetics supports.
• Partners: AGL2 was isolated as an AG K-domain interactor PMID:9418042. Other partners are AP1, ABS/TT16, AGL16, AGL24 and SHP2 (MADS), each MODIFY to GO:0046982. The non-MADS CrY2H hits (At3g24490 Myb/SANT; ARF46) are REMOVE as uninformative.
• Ovule: the SEP1/sep1 sep2 sep3 genotype gives ovule-identity defects PMID:14555696. Marked KEEP_AS_NON_CORE.
• NEW: GO:0010093 IMP from PMID:10821278. The same paper supports the existing SEP3 GO:0010093 IMP, and the triple-mutant genotype removes SEP1.