GO_REF:0000033

Annotation inferences using phylogenetic trees

GO_REF:0000107

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

GO_REF:0000120

Combined Automated Annotation using Multiple IEA Methods

PMID:17474147

Systematic identification of SH3 domain-mediated human protein-protein interactions by peptide array target screening.

PMID:21555454

The Brd4 extraterminal domain confers transcription activation independent of pTEFb by recruiting multiple proteins, including NSD3.

GLTSCR1 associates with BRD4 and contributes to activation of selected target genes.
Selected Results connect the GLTSCR1 interactome with BRD4 and chromatin-remodeling components.

PMID:29374058

Glioma tumor suppressor candidate region gene 1 (GLTSCR1) and its paralog GLTSCR1-like form SWI/SNF chromatin remodeling subcomplexes.

BICRA and BICRAL define mutually exclusive GBAF assemblies with cell-context-dependent effects.

PMID:30510198

A non-canonical BRD9-containing BAF chromatin remodeling complex regulates naive pluripotency in mouse embryonic stem cells.

GBAF contributes to naive mouse ESC regulation, with BRD9 supplying the principal functional perturbations.

PMID:35044719

Proteome-scale mapping of binding sites in the unstructured regions of the human proteome.

Reactome:R-HSA-9932443

BICRA, BICRAL and BRD9 bind SMARCC dimer:SMARCDs

Reactome:R-HSA-9933236

SS18-containing ATPase complex binds SMARCC dimer:SMARCDs:BICRAs:BRD9

An SS18-containing ATPase module joins the BICRA-containing assembly in nucleoplasm.

file:human/BICRA/BICRA-uniprot.txt

UniProt Q9NZM4 BICRA_HUMAN

The reviewed subunit annotation identifies a BRD4-to-GBAF bridge; the interaction section independently records NCK1 and CRK partners.

PMID:33232675

BICRA, a SWI/SNF Complex Member, Is Associated with BAF-Disorder Related Phenotypes in Humans and Model Organisms.

Human BICRA variants cause a neurodevelopmental disorder; zebrafish developmental phenotypes and fly ncBAF association/chromatin phenotypes support conserved organismal relevance.