The Drosophila caspase inhibitor DIAP1 is essential for cell survival and is negatively regulated by HID.

Metamorphosis in drosophila and other insects: the fate of neurons throughout the stages.

The damage-responsive Drosophila gene sickle encodes a novel IAP binding protein similar to but distinct from reaper, grim, and hid.

GH3, a novel proapoptotic domain in Drosophila Grim, promotes a mitochondrial death pathway.

Molecular mechanisms of DrICE inhibition by DIAP1 and removal of inhibition by Reaper, Hid and Grim.

Neuronal expression of p53 dominant-negative proteins in adult Drosophila melanogaster extends life span.

Crystal cell rupture after injury in Drosophila requires the JNK pathway, small GTPases and the TNF homolog Eiger.

DIAP2 functions as a mechanism-based regulator of drICE that contributes to the caspase activity threshold in living cells.

grim promotes programmed cell death of Drosophila microchaete glial cells.

Differential localization and processing of apoptotic proteins in Malpighian tubules of Drosophila during metamorphosis.

Coordinated expression of cell death genes regulates neuroblast apoptosis.

Essential role of grim-led programmed cell death for the establishment of corazonin-producing peptidergic nervous system during embryogenesis and metamorphosis in Drosophila melanogaster.

Mechanisms of postecdysis-associated programmed cell death of peptidergic neurons in Drosophila melanogaster.

Caspase-dependent regulation of the ubiquitin-proteasome system through direct substrate targeting.

grim, a novel cell death gene in Drosophila.

Drosophila grim induces apoptosis in mammalian cells.

grim UniProtKB record Q24570

Dredd, a novel effector of the apoptosis activators reaper, grim, and hid in Drosophila.

FlyBase gene description for grim (DROME)