Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Gene Ontology annotation based on curation of immunofluorescence data
Electronic Gene Ontology annotations created by ARBA machine learning models
Identification and functional characterization of the p66/p68 components of the MeCP1 complex.
Two highly related p66 proteins comprise a new family of potent transcriptional repressors interacting with MBD2 and MBD3.
Suppression of FOXO1 activity by FHL2 through SIRT1-mediated deacetylation.
Distinct regulatory elements mediate similar expression patterns in the excretory cell of Caenorhabditis elegans.
Heterogeneous nuclear ribonucleoprotein C1/C2, MeCP1, and SWI/SNF form a chromatin remodeling complex at the beta-globin locus control region.
p66alpha and p66beta of the Mi-2/NuRD complex mediate MBD2 and histone interaction.
MBD2/NuRD and MBD3/NuRD, two distinct complexes with different biochemical and functional properties.
ZIP: a novel transcription repressor, represses EGFR oncogene and suppresses breast carcinogenesis.
NuRD mediates activating and repressive functions of GATA-1 and FOG-1 during blood development.
NuRD suppresses pluripotency gene expression to promote transcriptional heterogeneity and lineage commitment.
Protein interaction network of alternatively spliced isoforms from brain links genetic risk factors for autism.
Towards elucidating the stability, dynamics and architecture of the nucleosome remodeling and deacetylase complex by using quantitative interaction proteomics.
NuRD-ZNF827 recruitment to telomeres creates a molecular scaffold for homologous recombination.
A proteome-scale map of the human interactome network.
Identification of Novel Proteins Co-Purifying with Cockayne Syndrome Group B (CSB) Reveals Potential Roles for CSB in RNA Metabolism and Chromatin Dynamics.
Transcription factors LRF and BCL11A independently repress expression of fetal hemoglobin.
ZMYND8 Co-localizes with NuRD on Target Genes and Regulates Poly(ADP-Ribose)-Dependent Recruitment of GATAD2A/NuRD to Sites of DNA Damage.
CHD3 and CHD4 form distinct NuRD complexes with different yet overlapping functionality.
Endogenous Cell Type-Specific Disrupted in Schizophrenia 1 Interactomes Reveal Protein Networks Associated With Neurodevelopmental Disorders.
HENA, heterogeneous network-based data set for Alzheimer's disease.
Extensive disruption of protein interactions by genetic variants across the allele frequency spectrum in human populations.
A reference map of the human binary protein interactome.
Cross-linking mass spectrometry reveals the structural topology of peripheral NuRD subunits relative to the core complex.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
Systematic discovery of mutation-directed neo-protein-protein interactions in cancer.
The dermatomyositis-specific autoantigen Mi2 is a component of a complex containing histone deacetylase and nucleosome remodeling activities.
MBD2, MBD3 and GATAD2A, GATAD2B bind core NuRD complex
CHD and CDK2AP subunits are recruited to NuRD
MBD3L2 binds MBD3 NuRD core complex
MBD3L1 binds MBD2 NuRD core complex
ZNF827 and NR2C2,NR2F2 recruit NuRD to telomeric chromatin
NuRD deacetylates telomeric nucleosomes
NuRD hydrolyzes ATP to affect nucleosome organization
NuRD deacetylates nucleosomes
NuRD binds TCF19 at FBP1 gene
NuRD binds TCF19 at G6PC gene
NuRD binds TCF19 at PCK1 gene
NuRD binds Ikaros family member proteins
UniProtKB O95983 — Methyl-CpG-binding domain protein 3
UniProtKB Q14192 — Four and a half LIM domains protein 2
UniProtKB Q8N5A5 — Zinc finger CCCH-type and G-patch domain-containing protein
UniProtKB Q8WXI9 — Transcriptional repressor p66-beta
Characterization of cDNA clones selected by the GeneMark analysis from size-fractionated cDNA libraries from human brain.
Improved titanium dioxide enrichment of phosphopeptides from HeLa cells and high confident phosphopeptide identification by cross-validation of MS/MS and MS/MS/MS spectra.
Diagnostic exome sequencing in persons with severe intellectual disability.
GATAD2B loss-of-function mutations cause a recognisable syndrome with intellectual disability and are associated with learning deficits and synaptic undergrowth in Drosophila.