Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Combined Automated Annotation using Multiple IEA Methods

Role of Ninjurin-1 in the migration of myeloid cells to central nervous system inflammatory lesions.

A proteome-scale map of the human interactome network.

Ninjurin1 regulates lipopolysaccharide-induced inflammation through direct binding.

A reference map of the human binary protein interactome.

Ninjurin 1 dodecamer peptide containing the N-terminal adhesion motif (N-NAM) exerts proangiogenic effects in HUVECs and in the postischemic brain.

NINJ1 mediates plasma membrane rupture during lytic cell death.

Glycine inhibits NINJ1 membrane clustering to suppress plasma membrane rupture in cell death.

Inhibiting membrane rupture with NINJ1 antibodies limits tissue injury.

Structural basis of NINJ1-mediated plasma membrane rupture in cell death.

NINJ1 induces plasma membrane rupture and release of damage-associated molecular pattern molecules during ferroptosis.

NINJ1 mediates plasma membrane rupture by cutting and releasing membrane disks.

How NINJ1 mediates plasma membrane rupture and why NINJ2 cannot.

Infection-induced glucose starvation triggers NINJ1-dependent macrophage lysis and Candida escape.

Surveying the roles of NINJ1 in plasma membrane rupture.

Ninjurins regulate hematopoietic stem cell amplification through canonical WNT signaling independent of plasma membrane rupture.

Mechanism of homophilic binding mediated by ninjurin, a novel widely expressed adhesion molecule.