Gene Ontology annotation through association of InterPro records with GO terms

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Gene Ontology annotation based on curation of immunofluorescence data

Gene Ontology annotation based on curation of intracellular localizations of expressed fusion proteins in living cells

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Combined Automated Annotation using Multiple IEA Methods

UniProt text export for G6PD (P11413)

Cell growth and cholesterol metabolism in human glucose-6-phosphate dehydrogenase deficient lymphomononuclear cells.

Structural studies of glucose-6-phosphate and NADP+ binding to human glucose-6-phosphate dehydrogenase.

Production of inflammatory molecules in peripheral blood mononuclear cells from severely glucose-6-phosphate dehydrogenase-deficient subjects.

Mutation in G6PD gene leads to loss of cellular control of protein glutathionylation: mechanism and implication.

Large-scale proteomics and phosphoproteomics of urinary exosomes.

Defining the membrane proteome of NK cells.

ATM activates the pentose phosphate pathway promoting anti-oxidant defence and DNA repair.

Quantitative proteomic analysis of human substantia nigra in Alzheimer's disease, Huntington's disease and Multiple sclerosis.

Sex steroid hormone modulation of NADPH pathways in MCF-7 cells.

In-depth proteomic analyses of exosomes isolated from expressed prostatic secretions in urine.

Ribose metabolism and nucleic acid synthesis in normal and glucose-6-phosphate dehydrogenase-deficient human erythrocytes infected with Plasmodium falciparum.

Regulation of G6PD acetylation by SIRT2 and KAT9 modulates NADPH homeostasis and cell survival during oxidative stress.

A proteome-scale map of the human interactome network.

Severe G6PD Deficiency Due to a New Missense Mutation in an Infant of Northern European Descent.

Aldolase B suppresses hepatocellular carcinogenesis by inhibiting G6PD and pentose phosphate pathways.

Substitution of arginine 219 by glycine compromises stability, dimerization, and catalytic activity in a G6PD mutant.

Biochemical variants of glucose-6-phosphate dehydrogenase giving rise to congenital nonspherocytic hemolytic disease.

Glucose 6-phosphate dehydrogenase activity in membranes of erythrocytes from normal individuals and subjects with Mediterranean G6PD deficiency.

G6PD multimers dehydrogenate G6P

AcK-NFE2L2-dependent P6GD gene expression