Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Cell division: plant-like properties of animal cell cytokinesis.
Receptor-mediated endocytosis in the Caenorhabditis elegans oocyte.
Completion of cytokinesis in C. elegans requires a brefeldin A-sensitive membrane accumulation at the cleavage furrow apex.
Genome-wide analysis identifies a general requirement for polarity proteins in endocytic traffic.
RAB-11 permissively regulates spindle alignment by modulating metaphase microtubule dynamics in Caenorhabditis elegans early embryos.
Regulated trafficking of the MSP/Eph receptor during oocyte meiotic maturation in C. elegans.
Rab11 is required for synchronous secretion of chondroitin proteoglycans after fertilization in Caenorhabditis elegans.
RACK-1 directs dynactin-dependent RAB-11 endosomal recycling during mitosis in Caenorhabditis elegans.
Caenorhabditis elegans screen reveals role of PAR-5 in RAB-11-recycling endosome positioning and apicobasal cell polarity.
The small GTPase RAB-11 directs polarized exocytosis of the intracellular pathogen N. parisii for fecal-oral transmission from C. elegans.
REI-1 Is a Guanine Nucleotide Exchange Factor Regulating RAB-11 Localization and Function in C. elegans Embryos.
The ArfGEF GBF-1 Is Required for ER Structure, Secretion and Endocytic Transport in C. elegans.
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RAB-11.1 is described as the Rab essential for endosome-to-plasma-membrane recycling in oocytes, and RAB-11.1::GFP recycling endosomes reach the oocyte plasma membrane independently of GBF-1.
"RAB-11.1, which is the Rab GTPase essential for recycling from endosomes to the plasma membrane was not affected by the reduction of GBF-1"
UniProt entry O01803 (RB11A_CAEEL)
Manual literature synthesis for rab-11.1
RAB-5- and RAB-11-dependent vesicle-trafficking pathways are required for plasma membrane repair after attack by bacterial pore-forming toxin.
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rab-11.1 RNAi makes animals hypersensitive to the pore-forming toxin Cry5B and impairs membrane-repair vesicle trafficking in intestinal cells.
"Vector-RNAi control animals are only mildly affected by a low dose of PFT, but animals grown on rab-5 or rab-11.1 RNAi are significantly more intoxicated"
WormBase gene description for rab-11.1 (worm)