Gene Ontology annotation through association of InterPro records with GO terms
Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Regulators of G protein signaling 6 and 7. Purification of complexes with gbeta5 and assessment of their effects on g protein-mediated signaling pathways.
RGS9-1 is required for normal inactivation of mouse cone phototransduction.
Direct interactions between the heterotrimeric G protein subunit G beta 5 and the G protein gamma subunit-like domain-containing regulator of G protein signaling 11: gain of function of cyan fluorescent protein-tagged G gamma 3.
Palmitoylation regulates plasma membrane-nuclear shuttling of R7BP, a novel membrane anchor for the RGS7 family.
Role of molecular chaperones in G protein beta5/regulator of G protein signaling dimer assembly and G protein betagamma dimer specificity.
RGS6/Gβ5 complex accelerates IKACh gating kinetics in atrial myocytes and modulates parasympathetic regulation of heart rate.
Gβ5-RGS complexes are gatekeepers of hyperactivity involved in control of multiple neurotransmitter systems.
Association of Rgs7/Gβ5 complexes with Girk channels and GABAB receptors in hippocampal CA1 pyramidal neurons.
A quantitative chaperone interaction network reveals the architecture of cellular protein homeostasis pathways.
Using an in situ proximity ligation assay to systematically profile endogenous protein-protein interactions in a pathway network.
Comprehensive analysis of heterotrimeric G-protein complex diversity and their interactions with GPCRs in solution.
LRIT3 is essential to localize TRPM1 to the dendritic tips of depolarizing bipolar cells and may play a role in cone synapse formation.
GNB5 mutation causes a novel neuropsychiatric disorder featuring attention deficit hyperactivity disorder, severely impaired language development and normal cognition.
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Human GNB5 variation impairs R7 RGS-mediated termination of neuronal G protein signaling.
"the S81L mutant form of Gβ5 has significantly
impaired activity in terminating responses that are elicited by dopamine"
Cellular and Subcellular Localization of the RGS7/Gβ5/R7BP Complex in the Cerebellar Cortex.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
Structure of the class C orphan GPCR GPR158 in complex with RGS7-Gβ5.
The G protein beta5 subunit interacts selectively with the Gq alpha subunit.
Cloning and tissue distribution of the human G protein beta 5 cDNA.
TRiC/CCT binds unfolded G-protein beta subunit
ATP hydrolysis promotes folding of G-protein beta by TRiC/CCT
PDCL binds G-protein beta and TRiC/CCT
ATP binds G-protein beta associated TRiC/CCT
RGS proteins bind GNB5 and CCT/TRiC
PDCL promotes G-protein beta 5 folding
Release of GNB5:RGS dimers from CCT/TRiC
Lyubarsky, Mol Vis 2001; 7:71-78.