IAA17 domain-I repression is directly tested in PMID:14742873. PMID:11283339 reports sevenfold stabilization of axr3-1 and interaction/localization experiments. PMID:22912871 actually images IAA17-GFP as a nuclear positive control, despite its IAA8-focused title; the old reference note saying presumably was superseded. PMID:24706860 now has normally fetched full text and supports the ARF7 PB1 interface. PMID:25324183 supports a contextual positive leaf-senescence role with overexpression and knockout phenotypes. The inherited NEW negative-transcription row remains true but adds no coverage beyond the newly refined broad transcription rows; no replacement NEW is created.
All 125 baseline rows were assessed, with original term/evidence/reference/partner fields and flags preserved. Bare GO:0005515 removal follows the explicit repository exception: it does not deny the measured interaction. Each affected reason identifies its original source, partner and assay context. Independently grounded functional ARF/SCF interactions are refined; homotypic binding is retained as non-core. Neither receptor-dependent degradation nor a loss-of-function phenotype alone licenses a new protein-catabolic-process annotation on an Aux/IAA substrate. No new rows were added in this audit.
The prior cis-regulatory-binding rejection inferred an unproved ARF bridge in a yeast one-hybrid screen. Normal publisher supplement retrieval found IAA12/AT1G04550 → AT5G12870 (MYB46 promoter), SupplementaryTable2 Sheet1 row445, with one of LacZ/HIS reporters activated. Table5 confirms the AUX-IAA clone. The Methods state: PMID:25533953. This supports retaining non-core promoter association, with no claim that every Aux/IAA is an autonomous DNA-binding transcription factor. PAINT inheritance is respected; no node defect or lineage-specific loss is inferred from a missing canonical DNA-binding domain.
Provider provenance: parent ran actual Falcon and Perplexity-lite attempts; both authentication/provider attempts failed. Logs are .cache/module-audit-logs/auxin-fetch/ARATH-IAA17-provider.log. No provider report was fabricated. Normal gene-publication caching succeeded. Primary sources, not provider prose or GOA rows, support the decisions. CrY2H-seq correction PMID:36690743 changes the first author surname, not the interaction data. The detailed row-by-row audit is generated under IAA17-source-analysis/annotation-audit.yaml.
Final actions: ACCEPT=6, KEEP_AS_NON_CORE=4, MODIFY=10, NEW=1, REMOVE=104.
Original source distribution: