Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on curation of immunofluorescence data

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Gene Ontology annotation of human sequence-specific DNA binding transcription factors (DbTFs) based on the TFClass database

Electronic Gene Ontology annotations created by ARBA machine learning models

Combined Automated Annotation using Multiple IEA Methods

Context-dependent transcriptional cooperation mediated by cardiac transcription factors Csx/Nkx-2.5 and GATA-4.

GATA4 mutations cause human congenital heart defects and reveal an interaction with TBX5.

Hairy-related transcription factors inhibit GATA-dependent cardiac gene expression through a signal-responsive mechanism.

Hey basic helix-loop-helix transcription factors are repressors of GATA4 and GATA6 and restrict expression of the GATA target gene ANF in fetal hearts.

The Kruppel-like transcription factor KLF13 is a novel regulator of heart development.

Developmental changes in human fetal testicular cell numbers and messenger ribonucleic acid levels during the second trimester.

A novel mutation in GATA4 gene associated with dominant inherited familial atrial septal defect.

Involvement of the atrial natriuretic peptide transcription factor GATA4 in alcohol dependence, relapse risk and treatment response to acamprosate.

Loss-of-function mutation in GATA4 causes anomalies of human testicular development.

Transcription factor genes Smad4 and Gata4 cooperatively regulate cardiac valve development. [corrected].

GATA4 loss-of-function mutations underlie familial tetralogy of fallot.

The formation and function of the cardiac conduction system.

Impact of cytosine methylation on DNA binding specificities of human transcription factors.

GATA4 Loss-of-Function Mutation and the Congenitally Bicuspid Aortic Valve.

Transcription factor protein interactomes reveal genetic determinants in heart disease.

The cardiac transcription factors Nkx2-5 and GATA-4 are mutual cofactors.

Distinct functions are implicated for the GATA-4, -5, and -6 transcription factors in the regulation of intestine epithelial cell differentiation.

Identification of the in vivo casein kinase II phosphorylation site within the homeodomain of the cardiac tisue-specifying homeobox gene product Csx/Nkx2.5.

ZFPM proteins bind GATA proteins

Proper coronary vascular development and heart morphogenesis depend on interaction of GATA-4 with FOG cofactors.

Opening of compacted chromatin by early developmental transcription factors HNF3 (FoxA) and GATA-4.

Gonadal differentiation, sex determination and normal Sry expression in mice require direct interaction between transcription partners GATA4 and FOG2.

GATA4 is a dosage-sensitive regulator of cardiac morphogenesis.

Morphogenesis of the right ventricle requires myocardial expression of Gata4.

Development of the mammalian liver and ventral pancreas is dependent on GATA4.

Disease Model of GATA4 Mutation Reveals Transcription Factor Cooperativity in Human Cardiogenesis.

GATA4 transcription factor is required for ventral morphogenesis and heart tube formation.

Requirement of the transcription factor GATA4 for heart tube formation and ventral morphogenesis.

The cardiac tissue-restricted homeobox protein Csx/Nkx2.5 physically associates with the zinc finger protein GATA4 and cooperatively activates atrial natriuretic factor gene expression.

GATA-4:FOG interactions regulate gastric epithelial development in the mouse.