Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on curation of immunofluorescence data
Electronic Gene Ontology annotations created by ARBA machine learning models
Combined Automated Annotation using Multiple IEA Methods
The human DNA methyltransferases (DNMTs) 1, 3a and 3b: coordinate mRNA expression in normal tissues and overexpression in tumors.
Cloning, expression and chromosome locations of the human DNMT3 gene family.
DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development.
Genetic variation in ICF syndrome: evidence for genetic heterogeneity.
Dnmt3b, de novo DNA methyltransferase, interacts with SUMO-1 and Ubc9 through its N-terminal region and is subject to modification by SUMO-1.
The Polycomb group protein EZH2 directly controls DNA methylation.
Mutations in DNA methyltransferase DNMT3B in ICF syndrome affect its regulation by DNMT3L.
Recruitment of the de novo DNA methyltransferase Dnmt3a by Kaposi's sarcoma-associated herpesvirus LANA.
Zinc-fingers and homeoboxes 1 (ZHX1) binds DNA methyltransferase (DNMT) 3B to enhance DNMT3B-mediated transcriptional repression.
DNA methyltransferase 1 and 3B activate BAG-1 expression via recruitment of CTCFL/BORIS and modulation of promoter histone methylation.
DNA methyltransferase 3B acts as a co-repressor of the human polycomb protein hPc2 to repress fibroblast growth factor receptor 3 transcription.
De novo DNA methylation promoted by G9a prevents reprogramming of embryonically silenced genes.
Np95 interacts with de novo DNA methyltransferases, Dnmt3a and Dnmt3b, and mediates epigenetic silencing of the viral CMV promoter in embryonic stem cells.
Oxidative damage targets complexes containing DNA methyltransferases, SIRT1, and polycomb members to promoter CpG Islands.
Dnmt3L antagonizes DNA methylation at bivalent promoters and favors DNA methylation at gene bodies in ESCs.
Enhancing the Cytotoxic Effects of PARP Inhibitors with DNA Demethylating Agents - A Potential Therapy for Cancer.
Structural insights into CpG-specific DNA methylation by human DNA methyltransferase 3B.
Structure of nucleosome-bound DNA methyltransferases DNMT3A and DNMT3B.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
PRC2 recruits DNA methyltransferases
DNMT1,3A,3B:PRC2 methylates cytosine and histone H3
SUMOylation of DNMT3B with SUMO1
The GSDME gene promoter is hypermethylated
Modulation of Dnmt3b function in vitro by interactions with Dnmt3L, Dnmt3a and Dnmt3b splice variants.
The DNA methyltransferase DNMT3C protects male germ cells from transposon activity.
Loss of Dnmt3b function upregulates the tumor modifier Ment and accelerates mouse lymphomagenesis.
DNA methyltransferase 3B mutant in ICF syndrome interacts non-covalently with SUMO-1.
Mutations in DNMT3B Modify Epigenetic Repression of the D4Z4 Repeat and the Penetrance of Facioscapulohumeral Dystrophy.