IAA12–ARF5 interaction is directly reported by PMID:12101120; EAR-dependent TPL recruitment and required TPL repression are explicit in PMID:18258861. The latter remains abstract-only after real refresh attempts, so no additional unseen assay is asserted. The bdl developmental phenotypes reflect a stabilized gain-of-function allele, not a gene deletion. Native root/vascular anatomy is not generalized to Marchantia. Inherited NEW signaling and ligase-binding rows are preserved on their direct primary evidence (PMID:15992545; co-receptor specificity PMID:22466420), not counted as new coverage.
All 61 baseline rows were assessed, with original term/evidence/reference/partner fields and flags preserved. Bare GO:0005515 removal follows the explicit repository exception: it does not deny the measured interaction. Each affected reason identifies its original source, partner and assay context. Independently grounded functional ARF/SCF interactions are refined; homotypic binding is retained as non-core. Neither receptor-dependent degradation nor a loss-of-function phenotype alone licenses a new protein-catabolic-process annotation on an Aux/IAA substrate. No new rows were added in this audit.
The prior cis-regulatory-binding rejection inferred an unproved ARF bridge in a yeast one-hybrid screen. Normal publisher supplement retrieval found IAA12/AT1G04550 → AT5G12870 (MYB46 promoter), SupplementaryTable2 Sheet1 row445, with one of LacZ/HIS reporters activated. Table5 confirms the AUX-IAA clone. The Methods state: PMID:25533953. This supports retaining non-core promoter association, with no claim that every Aux/IAA is an autonomous DNA-binding transcription factor. PAINT inheritance is respected; no node defect or lineage-specific loss is inferred from a missing canonical DNA-binding domain.
Provider provenance: parent ran actual Falcon and Perplexity-lite attempts; both authentication/provider attempts failed. Logs are .cache/module-audit-logs/auxin-fetch/ARATH-IAA12-provider.log. No provider report was fabricated. Normal gene-publication caching succeeded. Primary sources, not provider prose or GOA rows, support the decisions. CrY2H-seq correction PMID:36690743 changes the first author surname, not the interaction data. The detailed row-by-row audit is generated under IAA12-source-analysis/annotation-audit.yaml.
Final actions: ACCEPT=4, KEEP_AS_NON_CORE=8, MODIFY=6, NEW=2, REMOVE=41.
Original source distribution: