Gene Ontology annotation through association of InterPro records with GO terms

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniPathway vocabulary mapping

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Electronic Gene Ontology annotations created by ARBA machine learning models

XAB2, a novel tetratricopeptide repeat protein involved in transcription-coupled DNA repair and transcription.

Translocation of Cockayne syndrome group A protein to the nuclear matrix: possible relevance to transcription-coupled DNA repair.

The ubiquitin ligase activity in the DDB2 and CSA complexes is differentially regulated by the COP9 signalosome in response to DNA damage.

CSA-dependent degradation of CSB by the ubiquitin-proteasome pathway establishes a link between complementation factors of the Cockayne syndrome.

A family of diverse Cul4-Ddb1-interacting proteins includes Cdt2, which is required for S phase destruction of the replication factor Cdt1.

The role of CSA in the response to oxidative DNA damage in human cells.

The molecular basis of CRL4DDB2/CSA ubiquitin ligase architecture, targeting, and activation.

Mutations in UVSSA cause UV-sensitive syndrome and destabilize ERCC6 in transcription-coupled DNA repair.

The C-terminal Region and SUMOylation of Cockayne Syndrome Group B Protein Play Critical Roles in Transcription-coupled Nucleotide Excision Repair.

CSA and CSB play a role in the response to DNA breaks.

Ubiquitination of DNA Damage-Stalled RNAPII Promotes Transcription-Coupled Repair.

The cooperative action of CSB, CSA, and UVSSA target TFIIH to DNA damage-stalled RNA polymerase II.

Structural basis of human transcription-DNA repair coupling.

Structural basis for RNA polymerase II ubiquitylation and inactivation in transcription-coupled repair.

Transcription-coupled repair of DNA-protein cross-links depends on CSA and CSB.

ERCC8 (CSA) binds stalled RNA Pol II

ERCC8:DDB1:CUL4:RBX1 ubiquitinates ERCC6 and RNA Pol II

Assembly of the pre-incision complex in TC-NER

UVSSA:USP7 deubiquitinates ERCC6

ERCC2-facilitated RNA Pol II backtracking in TC-NER

ERCC5 and RPA bind TC-NER site

Binding of ERCC1:ERCC4 (ERCC1:XPF) to pre-incision complex in TC-NER

5' incision of damaged DNA strand by ERCC1:ERCC4 in TC-NER

Repair DNA synthesis of ~27-30 bases long patch by POLD, POLE or POLK in TC-NER

DNA polymerases delta, epsilon or kappa bind the TC-NER site

3' incision by ERCC5 (XPG) in TC-NER

Ligation of newly synthesized repair patch to incised DNA in TC-NER

Recovery of RNA synthesis after TC-NER

AcM-UBE2M transfers NEDD8 to CRL4 E3 ubiquitin ligase complex

NEDD8:AcM-UBE2M binds CRL4 E3 ubiquitin ligase complex

CAND1 binds CRL4 E3 ubiquitin ligase in the nucleus

COMMDs displace CAND1 from CRL4 E3 ubiquitin ligase complex

COP9 signalosome deneddylates nuclear CRL4 E3 ubiquitin ligase complex

ERCC3-facilitated RNA Pol II backtracking in TC-NER

Dynamic compartmentalization of DNA repair proteins within spiral ganglion neurons in response to noise stress.

Noise exposure potentiates the subcellular distribution of nucleotide excision repair proteins within spiral ganglion neurons.

Schizosaccharomyces pombe Ddb1 recruits substrate-specific adaptor proteins through a novel protein motif, the DDB-box.