The annotation-specific decisions use the source limits below. Native cache availability and separately inspected originals are recorded independently.
The purified human enzyme was assayed through PK/LDH coupling. PEP is a coupling reagent; L13943.1 identifies the isoform 1 template.
Source: PMID:37021775. Original Methods and Results; DOI 10.1042/BSR20222258.
The linked correction replaces Figures 8–10 after duplicated graphs entered during revision. The editorial assessment retained the underlying data and conclusions. No superseded numerical kinetics are used.
Source: PMID:37860917. Complete correction text; DOI 10.1042/BSR-2022-2258_COR.
Construct and imaging access limits remain; the named forms do not represent every product.
Source: PMID:15845384. Abstract and selected publisher passages; DOI 10.1016/j.bbrc.2005.03.143.
Patient measurements establish enzyme deficiency and glycerol excess; apparent triglyceride elevation does not establish altered triglyceride flux.
Source: PMID:8651297. Original pages 1205–1211, especially Table 1 and Results/Discussion; PMC1915081.
Table S2 row 798 records the validated pair. Figure S5 describes GK-dependent NR4A1 DNA-binding and reporter effects. These supply contact-specific functional context beyond partner classification.
Source: PMID:21988832. Original Methods/Results, Table S2 row 798, Figure S5 caption; PMID21988832.
Row 67764 names GK P32189-1 and GK2 Q14410. Product grouping and AP-MS association do not establish a functional heteromer.
Source: PMID:33961781. BioPlex3 original table row 67764 and original Methods; PMID33961781.
Network edge 602294 connects GK bait node 602290 to GK2 prey node 602292 in the U2OS AP-MS network.
Source: PMID:40205054. Published NDEx AP-MS network and original Methods; PMID40205054.
Table S1(A), row 412, group 4491 contains GK and has MitoCoP membership. Adjacent mixed GK/GK3P groups cannot establish a unique product. Membrane topology was not independently resolved by this target row.
Source: PMID:34800366. Actual Table S1 and selected original fractionation/grouping methods; PMID34800366.
The author-hosted GK/NP_976325 entry has one peptide and reference 2, linking it to this urinary-vesicle study. The literal urine restriction is retained; no modern product crosswalk or target spectrum was independently verified.
Source: PMID:19056867. Author-hosted table row 463 and original preparation/identification methods; PMID19056867.
Event 75887 reaches human GK member 55717 through catalyst 76115 and set 3769055. The cytosolic representation is not evidence that every splice product is cytosolic.
Source: Reactome:R-HSA-75887. Official reaction/catalyst/member objects; R-HSA-75887.
The pathway explicitly includes the GK reaction as a precursor-forming step. This is separate from patient pseudohypertriglyceridemia evidence.
Source: Reactome:R-HSA-75109. Official pathway event list; R-HSA-75109.
Official mappings are P32189-3(GK+EX18) and P32189-4(GK-EX18). Four product sequences are archived.
The inspected PTHR10196 tree places the exact human sequence below the activity IBD at AN0 and the four process/location IBDs at AN364. Literal donor sets, including self-donors, are retained. A cytosol IRD on the target path limits inherited canonical-product localization; independent product-aware human evidence is assessed separately.