Gene Ontology annotation through association of InterPro records with GO terms

Gene Ontology annotation by the MGI curatorial staff, curated orthology

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Automated transfer of experimentally-verified manual GO annotation data to mouse-rat orthologs

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Automated transfer of experimentally-verified manual GO annotation data to mouse-human orthologs

Combined Automated Annotation using Multiple IEA Methods

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore.

Integrated analysis of protein composition, tissue diversity, and gene regulation in mouse mitochondria.

Activation of Ras up-regulates pro-apoptotic BNIP3 in nitric oxide-induced cell death.

Qualitative and quantitative analyses of protein phosphorylation in naive and stimulated mouse synaptosomal preparations.

Wdnm1-like, a new adipokine with a role in MMP-2 activation.

p53-TIGAR axis attenuates mitophagy to exacerbate cardiac damage after ischemia.

Impact of lactobacilli on orally acquired listeriosis.

Adenovirus E1B 19 kDa and Bcl-2 proteins interact with a common set of cellular proteins.

Microtubule-associated protein 1 light chain 3 (LC3) interacts with Bnip3 protein to selectively remove endoplasmic reticulum and mitochondria via autophagy.

Modulation of serines 17 and 24 in the LC3-interacting region of Bnip3 determines pro-survival mitophagy versus apoptosis.

Hypoxia-induced autophagy is mediated through hypoxia-inducible factor induction of BNIP3 and BNIP3L via their BH3 domains.

BNip3 regulates mitochondrial function and lipid metabolism in the liver.

A mitochondrial SCF-FBXL4 ubiquitin E3 ligase complex degrades BNIP3 and NIX to restrain mitophagy and prevent mitochondrial disease.

A mitophagy sensor PPTC7 controls BNIP3 and NIX degradation to regulate mitochondrial mass.

Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice.

The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis.

The BH3-only Bnip3 binds to the dynamin Opa1 to promote mitochondrial fragmentation and apoptosis by distinct mechanisms.

BNIP3 and NIX mediate Mieap-induced accumulation of lysosomal proteins within mitochondria.

UniProt entry O55003 (BNIP3_MOUSE)