Gene Ontology annotation through association of InterPro records with GO terms

Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt

Gene Ontology annotation based on curation of immunofluorescence data

Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara

Automatic assignment of GO terms using logical inference, based on on inter-ontology links

Electronic Gene Ontology annotations created by ARBA machine learning models

Combined Automated Annotation using Multiple IEA Methods

UCP2 inhibits ROS-mediated apoptosis in A549 under hypoxic conditions.

Toward understanding the mechanism of ion transport activity of neuronal uncoupling proteins UCP2, UCP4, and UCP5.

UCP2 transports C4 metabolites out of mitochondria, regulating glucose and glutamine oxidation.

A proteome-scale map of the human interactome network.

A biophysical study on molecular physiology of the uncoupling proteins of the central nervous system.

PRMT1-mediated methylation of MICU1 determines the UCP2/3 dependency of mitochondrial Ca(2+) uptake in immortalized cells.

Loss of UCP2 impairs cold-induced non-shivering thermogenesis by promoting a shift toward glucose utilization in brown adipose tissue.

Functional Oligomeric Forms of Uncoupling Protein 2: Strong Evidence for Asymmetry in Protein and Lipid Bilayer Systems.

Quantitative high-confidence human mitochondrial proteome and its dynamics in cellular context.

Uncoupling protein-2: a novel gene linked to obesity and hyperinsulinemia.

UCP3: an uncoupling protein homologue expressed preferentially and abundantly in skeletal muscle and brown adipose tissue.

FA anion flip-flops to the opposite surface

FA anion diffuses laterally to UCP

The FA anion diffuses away laterally from UCP

Mutations in UCP2 in congenital hyperinsulinism reveal a role for regulation of insulin secretion.