Gene Ontology annotation through association of InterPro records with GO terms
Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on curation of immunofluorescence data
Automatic transfer of experimentally verified manual GO annotation data to orthologs using Ensembl Compara
Gene Ontology annotation of human sequence-specific DNA binding transcription factors (DbTFs) based on the TFClass database
Electronic Gene Ontology annotations created by ARBA machine learning models
Combined Automated Annotation using Multiple IEA Methods
Coactivators for the orphan nuclear receptor RORalpha.
Transcriptional regulation of apolipoprotein C-III gene expression by the orphan nuclear receptor RORalpha.
The orphan nuclear receptor ROR alpha is a negative regulator of the inflammatory response.
Transcription factors and nuclear receptors interact with the SWI/SNF complex through the BAF60c subunit.
A protein-protein interaction network for human inherited ataxias and disorders of Purkinje cell degeneration.
Cytosolic aspartate aminotransferase, a new partner in adipocyte glyceroneogenesis and an atypical target of thiazolidinedione.
Isoform-specific inhibition of ROR alpha-mediated transcriptional activation by human FOXP3.
Transcriptional activation of HIF-1 by RORalpha and its role in hypoxia signaling.
The nuclear receptor ROR(alpha) exerts a bi-directional regulation of IL-6 in resting and reactive astrocytes.
Modulation of retinoic acid receptor-related orphan receptor alpha and gamma activity by 7-oxygenated sterol ligands.
Modulation of clock gene expression by the transcriptional coregulator receptor interacting protein 140 (RIP140).
Cryptochromes mediate rhythmic repression of the glucocorticoid receptor.
A proteome-scale map of the human interactome network.
Isoform-specific amino-terminal domains dictate DNA-binding properties of ROR alpha, a novel family of orphan hormone nuclear receptors.
Transcriptional activation and repression by RORalpha, an orphan nuclear receptor required for cerebellar development.
Exogenous expression of a dominant negative RORalpha1 vector in muscle cells impairs differentiation: RORalpha1 directly interacts with p300 and myoD.
Formation of NR-MED1 Coactivator Complex
Formation of NR-NCOR Corepressor Complex
PIAS2,3,4 SUMOylate RORA with SUMO2
PIAS2,3,4 SUMOylate RORA with SUMO1
Expression of STAT3-upregulated nuclear proteins
Dual Molecular Effects of Dominant RORA Mutations Cause Two Variants of Syndromic Intellectual Disability with Either Autism or Cerebellar Ataxia.
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Dominant human RORA variants cause neurodevelopmental phenotypes that can include cerebellar ataxia and cerebellar atrophy, while functional complementation supports an essential cerebellar-development role.
"RORĪ±, the RAR-related orphan nuclear receptor alpha, is essential for cerebellar development."