Gene Ontology annotation through association of InterPro records with GO terms

Annotation inferences using phylogenetic trees

Gene Ontology annotation based on UniProtKB/Swiss-Prot keyword mapping

Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping

Combined Automated Annotation using Multiple IEA Methods

Regulation of longevity in Caenorhabditis elegans by heat shock factor and molecular chaperones.

The L-type cyclin CYL-1 and the heat-shock-factor HSF-1 are required for heat-shock-induced protein expression in Caenorhabditis elegans.

Heat-shock transcription factor (HSF)-1 pathway required for Caenorhabditis elegans immunity.

Ca(2+)/Calmodulin-binding proteins from the C. elegans proteome.

An ALS-linked mutant SOD1 produces a locomotor defect associated with aggregation and synaptic dysfunction when expressed in neurons of Caenorhabditis elegans.

Conditioning protects C. elegans from lethal effects of enteropathogenic E. coli by activating genes that regulate lifespan and innate immunity.

Diversity in DNA recognition by heat shock transcription factors (HSFs) from model organisms.

HSF-1 regulators DDL-1/2 link insulin-like signaling to heat-shock responses and modulation of longevity.

Caenorhabditis elegans HSF-1 is an essential nuclear protein that forms stress granule-like structures following heat shock.

A novel interaction between aging and ER overload in a protein conformational dementia.

Neuronal serotonin release triggers the heat shock response in C. elegans in the absence of temperature increase.

Repression of the Heat Shock Response Is a Programmed Event at the Onset of Reproduction.

HSF-1 is involved in regulation of ascaroside pheromone biosynthesis by heat stress in Caenorhabditis elegans.

HSF-1 activates the ubiquitin proteasome system to promote non-apoptotic developmental cell death in C. elegans.

Transcriptional control of non-apoptotic developmental cell death in C. elegans.

E2F coregulates an essential HSF developmental program that is distinct from the heat-shock response.

Hormetic heat stress and HSF-1 induce autophagy to improve survival and proteostasis in C. elegans.

HSF-1 is a regulator of miRNA expression in Caenorhabditis elegans.

Olfactory experience primes the heat shock transcription factor HSF-1 to enhance the expression of molecular chaperones in C. elegans.

Deep research report on hsf-1