CDH23

UniProt ID: Q9H251
Organism: Homo sapiens
Review Status: COMPLETE
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Gene Description

CDH23 encodes cadherin-23, a large calcium-dependent cadherin family cell-adhesion protein. In sensory hair cells, CDH23 contributes to the upper part of the stereocilia tip-link apparatus and interacts with harmonin and related Usher syndrome proteins to maintain hair-bundle cohesion and mechanotransduction. CDH23 is also expressed in retina, and pathogenic variants cause Usher syndrome and nonsyndromic hearing loss; germline variants have additionally been associated with pituitary adenoma predisposition, but the best-supported normal molecular role remains cadherin-mediated stereocilium adhesion and organization.

Existing Annotations Review

GO Term Evidence Action Reason
GO:0098609 cell-cell adhesion
IBA
GO_REF:0000033
ACCEPT
Summary: Cell-cell adhesion is a core cadherin-23 function, with calcium-dependent cadherin interactions supporting stereocilia cohesion and tip-link anchoring.
Reason: CDH23 is a cadherin family adhesion protein required for stereocilia bundle organization; the broad adhesion terms are appropriate, though stereocilium/tip-link context is the most informative framing.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
Cadherins are calcium-dependent cell adhesion proteins.
PMID:12485990
cadherin 23 are both present in the growing stereocilia and that they bind to each other
GO:0016477 cell migration
IBA
GO_REF:0000033
MARK AS OVER ANNOTATED
Summary: Cell migration is a generic cadherin-family propagation and is not a well-supported core CDH23 function.
Reason: The direct CDH23 evidence in this review supports stereocilium adhesion/organization, hearing, vestibular function, and retinal maintenance rather than cell migration.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
CDH23 is required for establishing and/or maintaining the proper organization of the stereocilia bundle
GO:0008013 beta-catenin binding
IBA
GO_REF:0000033
MARK AS OVER ANNOTATED
Summary: Beta-catenin/catenin-complex annotations appear to be over-propagated from classical cadherin biology and are not central to CDH23 hair-bundle function.
Reason: The reviewed CDH23 evidence emphasizes PCDH15, harmonin/USH1C, USH1G, and MYO7A in stereocilia rather than beta-catenin complex function.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
It is part of the functional network formed by USH1C, USH1G, CDH23 and MYO7A
GO:0016342 catenin complex
IBA
GO_REF:0000033
MARK AS OVER ANNOTATED
Summary: Beta-catenin/catenin-complex annotations appear to be over-propagated from classical cadherin biology and are not central to CDH23 hair-bundle function.
Reason: The reviewed CDH23 evidence emphasizes PCDH15, harmonin/USH1C, USH1G, and MYO7A in stereocilia rather than beta-catenin complex function.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
It is part of the functional network formed by USH1C, USH1G, CDH23 and MYO7A
GO:0031175 neuron projection development
IBA
GO_REF:0000033
MARK AS OVER ANNOTATED
Summary: Neuron projection development is too broad and not the right cellular context for the best-supported CDH23 function.
Reason: CDH23 acts in sensory hair-cell stereocilia bundles, not generic neuron projection development.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
proper organization of the stereocilia bundle of hair cells
GO:0045296 cadherin binding
IBA
GO_REF:0000033
ACCEPT
Summary: Cadherin binding is appropriate because CDH23 participates in cadherin-mediated tip-link/adhesion assemblies, including heterophilic PCDH15 interaction by similarity and related cadherin-family interactions.
Reason: This term captures a real molecular interaction mode better than generic protein binding.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
Cadherin repeats 1 and 2 mediate calcium-dependent heterophilic interaction with PCDH15.
GO:0005509 calcium ion binding
IEA
GO_REF:0000002
ACCEPT
Summary: Calcium ion binding is appropriate for the extracellular cadherin repeats of CDH23.
Reason: Cadherin repeats use calcium to rigidify extracellular-domain interfaces and support adhesion.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
Three calcium ions are usually bound at the interface of each cadherin domain
GO:0005886 plasma membrane
IEA
GO_REF:0000120
ACCEPT
Summary: CDH23 is a single-pass type I membrane protein, so membrane/plasma membrane localization is appropriate.
Reason: The broad membrane annotation is true, with stereocilium/hair-bundle membrane localization being more informative.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
SUBCELLULAR LOCATION: Cell membrane
file:human/CDH23/CDH23-uniprot.txt
Single-pass type I membrane protein
GO:0007155 cell adhesion
IEA
GO_REF:0000002
ACCEPT
Summary: Cell-cell adhesion is a core cadherin-23 function, with calcium-dependent cadherin interactions supporting stereocilia cohesion and tip-link anchoring.
Reason: CDH23 is a cadherin family adhesion protein required for stereocilia bundle organization; the broad adhesion terms are appropriate, though stereocilium/tip-link context is the most informative framing.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
Cadherins are calcium-dependent cell adhesion proteins.
PMID:12485990
cadherin 23 are both present in the growing stereocilia and that they bind to each other
GO:0007156 homophilic cell-cell adhesion
IEA
GO_REF:0000002
MARK AS OVER ANNOTATED
Summary: CDH23 mediates heterophilic, not homophilic, cell-cell adhesion. The functional tip-link is a CDH23-PCDH15 heterodimer, so the InterPro-propagated homophilic annotation from the generic cadherin domain is an over-annotation for this paralog.
Reason: The InterPro2GO mapping propagated a generic cadherin-family homophilic adhesion term, but CDH23 specifically forms an antiparallel heterodimer with PCDH15 via cadherin repeats 1-2, defining the upper part of the stereocilia tip link. The broader cell adhesion term (GO:0007155) and stereocilium-specific terms are retained as accurate; homophilic adhesion is not supported for this gene.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
SUBUNIT: antiparallel heterodimer with PCDH15
file:human/CDH23/CDH23-uniprot.txt
Cadherin repeats 1 and 2 mediate calcium-dependent heterophilic interaction with PCDH15.
GO:0016020 membrane
IEA
GO_REF:0000002
ACCEPT
Summary: CDH23 is a single-pass type I membrane protein, so membrane/plasma membrane localization is appropriate.
Reason: The broad membrane annotation is true, with stereocilium/hair-bundle membrane localization being more informative.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
SUBCELLULAR LOCATION: Cell membrane
file:human/CDH23/CDH23-uniprot.txt
Single-pass type I membrane protein
GO:0005515 protein binding
IPI
PMID:22879593
Large protein assemblies formed by multivalent interactions ...
MARK AS OVER ANNOTATED
Summary: The cited interaction evidence is meaningful but generic protein binding is too vague for CDH23 curation.
Reason: The interaction should be interpreted as harmonin/USH protein binding and stereocilium tip-link anchoring rather than generic protein binding.
Supporting Evidence:
PMID:22879593
cadherin23/harmonin complexes
GO:0042802 identical protein binding
IPI
PMID:22879593
Large protein assemblies formed by multivalent interactions ...
KEEP AS NON CORE
Summary: Identical protein binding is supported by evidence that an exon68-encoded CDH23 peptide can self-dimerize.
Reason: Self-dimerization contributes to polymeric CDH23-harmonin assemblies but is not the whole gene-level core function.
Supporting Evidence:
PMID:22879593
the exon68-encoded peptide of cadherin23 can either interact with the N-terminal domain (NTD) of harmonin or form a homodimer
GO:0006816 calcium ion transport
IMP
PMID:17234811
A functional study of plasma-membrane calcium-pump isoform 2...
REMOVE
Summary: CDH23 is not a calcium transporter; the cited calcium-transport study centers on PMCA2/ATP2B2 with CDH23 as a hearing modifier.
Reason: The annotation conflates hair-cell calcium physiology with CDH23 cadherin function.
Supporting Evidence:
PMID:17234811
Ca2+ enters the stereocilia of hair cells through mechanoelectrical transduction channels
PMID:17234811
exported back to endolymph by an unusual splicing isoform
GO:0007605 sensory perception of sound
IMP
PMID:17234811
A functional study of plasma-membrane calcium-pump isoform 2...
ACCEPT
Summary: Sensory perception of sound is a core physiological process supported by CDH23 stereocilium/hair-bundle function and hearing-loss genetics.
Reason: CDH23 is required for normal hearing through its role in stereocilia bundle organization, so sound perception should be treated as the core biological-process context for the cadherin/tip-link function.
Supporting Evidence:
PMID:15537665
CDH23 and PCDH15 play an essential long-term role in maintaining the normal organization of the stereocilia bundle
file:human/CDH23/CDH23-uniprot.txt
Required for normal hearing.
GO:0051480 regulation of cytosolic calcium ion concentration
IMP
PMID:17234811
A functional study of plasma-membrane calcium-pump isoform 2...
MARK AS OVER ANNOTATED
Summary: Regulation of cytosolic calcium concentration is an indirect hearing-physiology consequence rather than a direct CDH23 function.
Reason: The direct molecular role of CDH23 is cadherin-mediated stereocilium adhesion; PMCA2 is the calcium pump in the cited work.
Supporting Evidence:
PMID:17234811
stereociliary Ca2+ transients induced by Ca2+ uncaging was compromised
GO:0045494 photoreceptor cell maintenance
IMP
PMID:16679490
Survey of the frequency of USH1 gene mutations in a cohort o...
KEEP AS NON CORE
Summary: This phenotype-level sensory annotation is supported but should be kept as non-core relative to the molecular cadherin/stereocilium function.
Reason: CDH23 mutations cause hearing/vestibular/retinal phenotypes, but the core gene product role is stereocilium adhesion and hair-bundle organization.
Supporting Evidence:
PMID:15537665
CDH23 and PCDH15 play an essential long-term role in maintaining the normal organization of the stereocilia bundle
file:human/CDH23/CDH23-uniprot.txt
Required for normal hearing.
GO:0050953 sensory perception of light stimulus
IMP
PMID:16679490
Survey of the frequency of USH1 gene mutations in a cohort o...
KEEP AS NON CORE
Summary: This phenotype-level sensory annotation is supported but should be kept as non-core relative to the molecular cadherin/stereocilium function.
Reason: CDH23 mutations cause hearing/vestibular/retinal phenotypes, but the core gene product role is stereocilium adhesion and hair-bundle organization.
Supporting Evidence:
PMID:15537665
CDH23 and PCDH15 play an essential long-term role in maintaining the normal organization of the stereocilia bundle
file:human/CDH23/CDH23-uniprot.txt
Required for normal hearing.
GO:0005515 protein binding
IPI
PMID:12485990
Myosin VIIa, harmonin and cadherin 23, three Usher I gene pr...
MARK AS OVER ANNOTATED
Summary: The cited interaction evidence is meaningful but generic protein binding is too vague for CDH23 curation.
Reason: The interaction should be interpreted as harmonin/USH protein binding and stereocilium tip-link anchoring rather than generic protein binding.
Supporting Evidence:
PMID:12485990
cadherin 23 are both present in the growing stereocilia and that they bind to each other
GO:0007605 sensory perception of sound
IMP
PMID:16679490
Survey of the frequency of USH1 gene mutations in a cohort o...
ACCEPT
Summary: Sensory perception of sound is a core physiological process supported by CDH23 stereocilium/hair-bundle function and hearing-loss genetics.
Reason: CDH23 is required for normal hearing through its role in stereocilia bundle organization, so sound perception should be treated as the core biological-process context for the cadherin/tip-link function.
Supporting Evidence:
PMID:15537665
CDH23 and PCDH15 play an essential long-term role in maintaining the normal organization of the stereocilia bundle
file:human/CDH23/CDH23-uniprot.txt
Required for normal hearing.
GO:0032420 stereocilium
ISS
PMID:12485990
Myosin VIIa, harmonin and cadherin 23, three Usher I gene pr...
ACCEPT
Summary: Stereocilium localization is directly supported and central to CDH23 function in hair cells.
Reason: CDH23 is present in developing stereocilia and participates in tip-link/hair-bundle architecture.
Supporting Evidence:
PMID:12485990
cadherin 23 are both present in the growing stereocilia
GO:0050957 equilibrioception
IMP
PMID:16679490
Survey of the frequency of USH1 gene mutations in a cohort o...
KEEP AS NON CORE
Summary: This phenotype-level sensory annotation is supported but should be kept as non-core relative to the molecular cadherin/stereocilium function.
Reason: CDH23 mutations cause hearing/vestibular/retinal phenotypes, but the core gene product role is stereocilium adhesion and hair-bundle organization.
Supporting Evidence:
PMID:15537665
CDH23 and PCDH15 play an essential long-term role in maintaining the normal organization of the stereocilia bundle
file:human/CDH23/CDH23-uniprot.txt
Required for normal hearing.
GO:0007605 sensory perception of sound
IMP
PMID:15537665
Digenic inheritance of deafness caused by mutations in genes...
ACCEPT
Summary: Sensory perception of sound is a core physiological process supported by CDH23 stereocilium/hair-bundle function and hearing-loss genetics.
Reason: CDH23 is required for normal hearing through its role in stereocilia bundle organization, so sound perception should be treated as the core biological-process context for the cadherin/tip-link function.
Supporting Evidence:
PMID:15537665
CDH23 and PCDH15 play an essential long-term role in maintaining the normal organization of the stereocilia bundle
file:human/CDH23/CDH23-uniprot.txt
Required for normal hearing.
GO:0016020 membrane
NAS
PMID:11138009
Mutation of CDH23, encoding a new member of the cadherin gen...
ACCEPT
Summary: CDH23 is a single-pass type I membrane protein, so membrane/plasma membrane localization is appropriate.
Reason: The broad membrane annotation is true, with stereocilium/hair-bundle membrane localization being more informative.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
SUBCELLULAR LOCATION: Cell membrane
file:human/CDH23/CDH23-uniprot.txt
Single-pass type I membrane protein
GO:0016339 calcium-dependent cell-cell adhesion
NAS
PMID:11138009
Mutation of CDH23, encoding a new member of the cadherin gen...
ACCEPT
Summary: Cell-cell adhesion is a core cadherin-23 function, with calcium-dependent cadherin interactions supporting stereocilia cohesion and tip-link anchoring.
Reason: CDH23 is a cadherin family adhesion protein required for stereocilia bundle organization; the broad adhesion terms are appropriate, though stereocilium/tip-link context is the most informative framing.
Supporting Evidence:
file:human/CDH23/CDH23-uniprot.txt
Cadherins are calcium-dependent cell adhesion proteins.
PMID:12485990
cadherin 23 are both present in the growing stereocilia and that they bind to each other

Core Functions

CDH23 mediates calcium-dependent cadherin interactions in the stereocilium tip-link/hair-bundle apparatus, anchoring CDH23-harmonin and related Usher protein assemblies needed for stereocilia cohesion and mechanotransduction.

Molecular Function:
cadherin binding
Directly Involved In:
Supporting Evidence:
  • PMID:12485990
    cadherin 23 are both present in the growing stereocilia and that they bind to each other
  • PMID:22879593
    cadherin23/harmonin complexes
  • file:human/CDH23/CDH23-uniprot.txt
    CDH23 is required for establishing and/or maintaining the proper organization of the stereocilia bundle

References

Gene Ontology annotation through association of InterPro records with GO terms
Annotation inferences using phylogenetic trees
Combined Automated Annotation using Multiple IEA Methods
Mutation of CDH23, encoding a new member of the cadherin gene family, causes Usher syndrome type 1D.
Myosin VIIa, harmonin and cadherin 23, three Usher I gene products that cooperate to shape the sensory hair cell bundle.
Digenic inheritance of deafness caused by mutations in genes encoding cadherin 23 and protocadherin 15 in mice and humans.
Survey of the frequency of USH1 gene mutations in a cohort of Usher patients shows the importance of cadherin 23 and protocadherin 15 genes and establishes a detection rate of above 90%.
A functional study of plasma-membrane calcium-pump isoform 2 mutants causing digenic deafness.
Large protein assemblies formed by multivalent interactions between cadherin23 and harmonin suggest a stable anchorage structure at the tip link of stereocilia.
file:human/CDH23/CDH23-uniprot.txt
UniProt record for CDH23

Suggested Questions for Experts

Q: Which CDH23 domains or isoforms, if any, are mechanistically relevant to pituitary adenoma predisposition rather than hearing/retinal biology?

Q: Can PITA5-associated CDH23 variants be shown to perturb cadherin adhesion, mechanotransduction-related assemblies, or a pituitary-specific interaction network?

Suggested Experiments

Experiment: Test PITA5-associated CDH23 variants in cadherin adhesion and harmonin/PCDH15 interaction assays alongside known hearing-loss variants.

Hypothesis: PITA5-associated variants may perturb CDH23 adhesion or scaffold interactions differently from classic hearing-loss variants.

Experiment: Assess CDH23 isoform expression and subcellular localization in normal pituitary and pituitary adenoma samples.

Hypothesis: Pituitary-relevant isoform expression or localization would explain how CDH23 variation contributes to adenoma predisposition.

📚 Additional Documentation

Notes

(CDH23-notes.md)

CDH23 notes

2026-06-02

PITA context: CDH23 corresponds to PITA5 / pituitary adenoma 5, multiple types. UniProt notes that PITA5 includes GH-, PRL-, ACTH-, TSH-secreting and plurihormonal tumors and that familial transmission is consistent with autosomal dominant inheritance with reduced penetrance [file:human/CDH23/CDH23-uniprot.txt "PITA5 is consistent with autosomal dominant inheritance with reduced penetrance"].

Deep research status: just deep-research-falcon human CDH23 --fallback perplexity-lite timed out on Falcon after 600 seconds, and the fallback failed with a Perplexity quota error. I proceeded using fetched UniProt, GOA, and cached publications.

Functional summary: CDH23 is a cadherin-family adhesion protein whose best-supported normal function is in stereocilia/hair-bundle architecture. UniProt states that "Cadherins are calcium-dependent cell adhesion proteins" and that CDH23 is required for "proper organization of the stereocilia bundle" [file:human/CDH23/CDH23-uniprot.txt "Cadherins are calcium-dependent cell adhesion proteins."; file:human/CDH23/CDH23-uniprot.txt "proper organization of the stereocilia bundle of hair cells"]. Experimental evidence shows cadherin 23 is present in growing stereocilia and binds harmonin PMID:12485990, and CDH23/PCDH15 maintain normal stereocilia bundle organization PMID:15537665.

Annotation decisions: I accepted cadherin adhesion, calcium binding, membrane/stereocilium localization, and sensory phenotypes as supported, with sensory phenotypes kept non-core where they are phenotype-level consequences. I removed the calcium ion transport annotation because the cited PMCA2 paper describes stereociliary calcium entry/export biology rather than CDH23 transporter activity [PMID:17234811 "Ca2+ enters the stereocilia of hair cells through mechanoelectrical transduction channels"; PMID:17234811 "exported back to endolymph by an unusual splicing isoform"].

📄 View Raw YAML

id: Q9H251
gene_symbol: CDH23
product_type: PROTEIN
status: COMPLETE
taxon:
  id: NCBITaxon:9606
  label: Homo sapiens
description: CDH23 encodes cadherin-23, a large calcium-dependent cadherin family cell-adhesion
  protein. In sensory hair cells, CDH23 contributes to the upper part of the stereocilia tip-link
  apparatus and interacts with harmonin and related Usher syndrome proteins to maintain hair-bundle
  cohesion and mechanotransduction. CDH23 is also expressed in retina, and pathogenic variants
  cause Usher syndrome and nonsyndromic hearing loss; germline variants have additionally been
  associated with pituitary adenoma predisposition, but the best-supported normal molecular role
  remains cadherin-mediated stereocilium adhesion and organization.
alternative_products:
- name: '1'
  id: Q9H251-1
- name: '2'
  id: Q9H251-2
  sequence_note: VSP_000645
- name: '3'
  id: Q9H251-3
  sequence_note: VSP_000646
- name: '4'
  id: Q9H251-4
  sequence_note: VSP_000647
- name: '5'
  id: Q9H251-5
  sequence_note: VSP_013268, VSP_013269
- name: '6'
  id: Q9H251-6
  sequence_note: VSP_035289, VSP_035290
- name: 7 (B1)
  id: Q9H251-7
  sequence_note: VSP_044260
- name: '8'
  id: Q9H251-8
  sequence_note: VSP_044261, VSP_000645
- name: 9 (B2)
  id: Q9H251-9
  sequence_note: VSP_044260, VSP_000647
- name: 10 (C1)
  id: Q9H251-10
  sequence_note: VSP_047923, VSP_047924
- name: 11 (C2)
  id: Q9H251-11
  sequence_note: VSP_047923, VSP_047924, VSP_000647
existing_annotations:
- term:
    id: GO:0098609
    label: cell-cell adhesion
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: involved_in
  review:
    summary: Cell-cell adhesion is a core cadherin-23 function, with calcium-dependent cadherin
      interactions supporting stereocilia cohesion and tip-link anchoring.
    action: ACCEPT
    reason: CDH23 is a cadherin family adhesion protein required for stereocilia bundle organization;
      the broad adhesion terms are appropriate, though stereocilium/tip-link context is the most
      informative framing.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Cadherins are calcium-dependent cell adhesion proteins.
    - reference_id: PMID:12485990
      supporting_text: cadherin 23 are both present in the growing stereocilia and that they bind
        to each other
- term:
    id: GO:0016477
    label: cell migration
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: involved_in
  review:
    summary: Cell migration is a generic cadherin-family propagation and is not a well-supported
      core CDH23 function.
    action: MARK_AS_OVER_ANNOTATED
    reason: The direct CDH23 evidence in this review supports stereocilium adhesion/organization,
      hearing, vestibular function, and retinal maintenance rather than cell migration.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: CDH23 is required for establishing and/or maintaining the proper organization
        of the stereocilia bundle
- term:
    id: GO:0008013
    label: beta-catenin binding
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: enables
  review:
    summary: Beta-catenin/catenin-complex annotations appear to be over-propagated from classical
      cadherin biology and are not central to CDH23 hair-bundle function.
    action: MARK_AS_OVER_ANNOTATED
    reason: The reviewed CDH23 evidence emphasizes PCDH15, harmonin/USH1C, USH1G, and MYO7A in
      stereocilia rather than beta-catenin complex function.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: It is part of the functional network formed by USH1C, USH1G, CDH23 and
        MYO7A
- term:
    id: GO:0016342
    label: catenin complex
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: part_of
  review:
    summary: Beta-catenin/catenin-complex annotations appear to be over-propagated from classical
      cadherin biology and are not central to CDH23 hair-bundle function.
    action: MARK_AS_OVER_ANNOTATED
    reason: The reviewed CDH23 evidence emphasizes PCDH15, harmonin/USH1C, USH1G, and MYO7A in
      stereocilia rather than beta-catenin complex function.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: It is part of the functional network formed by USH1C, USH1G, CDH23 and
        MYO7A
- term:
    id: GO:0031175
    label: neuron projection development
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: involved_in
  review:
    summary: Neuron projection development is too broad and not the right cellular context for
      the best-supported CDH23 function.
    action: MARK_AS_OVER_ANNOTATED
    reason: CDH23 acts in sensory hair-cell stereocilia bundles, not generic neuron projection
      development.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: proper organization of the stereocilia bundle of hair cells
- term:
    id: GO:0045296
    label: cadherin binding
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: enables
  review:
    summary: Cadherin binding is appropriate because CDH23 participates in cadherin-mediated tip-link/adhesion
      assemblies, including heterophilic PCDH15 interaction by similarity and related cadherin-family
      interactions.
    action: ACCEPT
    reason: This term captures a real molecular interaction mode better than generic protein binding.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Cadherin repeats 1 and 2 mediate calcium-dependent heterophilic interaction
        with PCDH15.
- term:
    id: GO:0005509
    label: calcium ion binding
  evidence_type: IEA
  original_reference_id: GO_REF:0000002
  qualifier: enables
  review:
    summary: Calcium ion binding is appropriate for the extracellular cadherin repeats of CDH23.
    action: ACCEPT
    reason: Cadherin repeats use calcium to rigidify extracellular-domain interfaces and support
      adhesion.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Three calcium ions are usually bound at the interface of each cadherin
        domain
- term:
    id: GO:0005886
    label: plasma membrane
  evidence_type: IEA
  original_reference_id: GO_REF:0000120
  qualifier: located_in
  review:
    summary: CDH23 is a single-pass type I membrane protein, so membrane/plasma membrane localization
      is appropriate.
    action: ACCEPT
    reason: The broad membrane annotation is true, with stereocilium/hair-bundle membrane localization
      being more informative.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: 'SUBCELLULAR LOCATION: Cell membrane'
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Single-pass type I membrane protein
- term:
    id: GO:0007155
    label: cell adhesion
  evidence_type: IEA
  original_reference_id: GO_REF:0000002
  qualifier: involved_in
  review:
    summary: Cell-cell adhesion is a core cadherin-23 function, with calcium-dependent cadherin
      interactions supporting stereocilia cohesion and tip-link anchoring.
    action: ACCEPT
    reason: CDH23 is a cadherin family adhesion protein required for stereocilia bundle organization;
      the broad adhesion terms are appropriate, though stereocilium/tip-link context is the most
      informative framing.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Cadherins are calcium-dependent cell adhesion proteins.
    - reference_id: PMID:12485990
      supporting_text: cadherin 23 are both present in the growing stereocilia and that they bind
        to each other
- term:
    id: GO:0007156
    label: homophilic cell-cell adhesion
  evidence_type: IEA
  original_reference_id: GO_REF:0000002
  qualifier: involved_in
  review:
    summary: CDH23 mediates heterophilic, not homophilic, cell-cell adhesion. The functional
      tip-link is a CDH23-PCDH15 heterodimer, so the InterPro-propagated homophilic annotation
      from the generic cadherin domain is an over-annotation for this paralog.
    action: MARK_AS_OVER_ANNOTATED
    reason: The InterPro2GO mapping propagated a generic cadherin-family homophilic adhesion term,
      but CDH23 specifically forms an antiparallel heterodimer with PCDH15 via cadherin repeats
      1-2, defining the upper part of the stereocilia tip link. The broader cell adhesion term
      (GO:0007155) and stereocilium-specific terms are retained as accurate; homophilic adhesion
      is not supported for this gene.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: 'SUBUNIT: antiparallel heterodimer with PCDH15'
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Cadherin repeats 1 and 2 mediate calcium-dependent heterophilic interaction
        with PCDH15.
- term:
    id: GO:0016020
    label: membrane
  evidence_type: IEA
  original_reference_id: GO_REF:0000002
  qualifier: located_in
  review:
    summary: CDH23 is a single-pass type I membrane protein, so membrane/plasma membrane localization
      is appropriate.
    action: ACCEPT
    reason: The broad membrane annotation is true, with stereocilium/hair-bundle membrane localization
      being more informative.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: 'SUBCELLULAR LOCATION: Cell membrane'
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Single-pass type I membrane protein
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:22879593
  qualifier: enables
  review:
    summary: The cited interaction evidence is meaningful but generic protein binding is too vague
      for CDH23 curation.
    action: MARK_AS_OVER_ANNOTATED
    reason: The interaction should be interpreted as harmonin/USH protein binding and stereocilium
      tip-link anchoring rather than generic protein binding.
    supported_by:
    - reference_id: PMID:22879593
      supporting_text: cadherin23/harmonin complexes
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IPI
  original_reference_id: PMID:22879593
  qualifier: enables
  review:
    summary: Identical protein binding is supported by evidence that an exon68-encoded CDH23 peptide
      can self-dimerize.
    action: KEEP_AS_NON_CORE
    reason: Self-dimerization contributes to polymeric CDH23-harmonin assemblies but is not the
      whole gene-level core function.
    supported_by:
    - reference_id: PMID:22879593
      supporting_text: the exon68-encoded peptide of cadherin23 can either interact with the N-terminal
        domain (NTD) of harmonin or form a homodimer
- term:
    id: GO:0006816
    label: calcium ion transport
  evidence_type: IMP
  original_reference_id: PMID:17234811
  qualifier: involved_in
  review:
    summary: CDH23 is not a calcium transporter; the cited calcium-transport study centers on
      PMCA2/ATP2B2 with CDH23 as a hearing modifier.
    action: REMOVE
    reason: The annotation conflates hair-cell calcium physiology with CDH23 cadherin function.
    supported_by:
    - reference_id: PMID:17234811
      supporting_text: Ca2+ enters the stereocilia of hair cells through mechanoelectrical transduction
        channels
    - reference_id: PMID:17234811
      supporting_text: exported back to endolymph by an unusual splicing isoform
- term:
    id: GO:0007605
    label: sensory perception of sound
  evidence_type: IMP
  original_reference_id: PMID:17234811
  qualifier: involved_in
  review:
    summary: Sensory perception of sound is a core physiological process supported by CDH23 stereocilium/hair-bundle
      function and hearing-loss genetics.
    action: ACCEPT
    reason: CDH23 is required for normal hearing through its role in stereocilia bundle organization,
      so sound perception should be treated as the core biological-process context for the cadherin/tip-link
      function.
    supported_by:
    - reference_id: PMID:15537665
      supporting_text: CDH23 and PCDH15 play an essential long-term role in maintaining the normal
        organization of the stereocilia bundle
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Required for normal hearing.
- term:
    id: GO:0051480
    label: regulation of cytosolic calcium ion concentration
  evidence_type: IMP
  original_reference_id: PMID:17234811
  qualifier: involved_in
  review:
    summary: Regulation of cytosolic calcium concentration is an indirect hearing-physiology consequence
      rather than a direct CDH23 function.
    action: MARK_AS_OVER_ANNOTATED
    reason: The direct molecular role of CDH23 is cadherin-mediated stereocilium adhesion; PMCA2
      is the calcium pump in the cited work.
    supported_by:
    - reference_id: PMID:17234811
      supporting_text: stereociliary Ca2+ transients induced by Ca2+ uncaging was compromised
- term:
    id: GO:0045494
    label: photoreceptor cell maintenance
  evidence_type: IMP
  original_reference_id: PMID:16679490
  qualifier: involved_in
  review:
    summary: This phenotype-level sensory annotation is supported but should be kept as non-core
      relative to the molecular cadherin/stereocilium function.
    action: KEEP_AS_NON_CORE
    reason: CDH23 mutations cause hearing/vestibular/retinal phenotypes, but the core gene product
      role is stereocilium adhesion and hair-bundle organization.
    supported_by:
    - reference_id: PMID:15537665
      supporting_text: CDH23 and PCDH15 play an essential long-term role in maintaining the normal
        organization of the stereocilia bundle
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Required for normal hearing.
- term:
    id: GO:0050953
    label: sensory perception of light stimulus
  evidence_type: IMP
  original_reference_id: PMID:16679490
  qualifier: involved_in
  review:
    summary: This phenotype-level sensory annotation is supported but should be kept as non-core
      relative to the molecular cadherin/stereocilium function.
    action: KEEP_AS_NON_CORE
    reason: CDH23 mutations cause hearing/vestibular/retinal phenotypes, but the core gene product
      role is stereocilium adhesion and hair-bundle organization.
    supported_by:
    - reference_id: PMID:15537665
      supporting_text: CDH23 and PCDH15 play an essential long-term role in maintaining the normal
        organization of the stereocilia bundle
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Required for normal hearing.
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:12485990
  qualifier: enables
  review:
    summary: The cited interaction evidence is meaningful but generic protein binding is too vague
      for CDH23 curation.
    action: MARK_AS_OVER_ANNOTATED
    reason: The interaction should be interpreted as harmonin/USH protein binding and stereocilium
      tip-link anchoring rather than generic protein binding.
    supported_by:
    - reference_id: PMID:12485990
      supporting_text: cadherin 23 are both present in the growing stereocilia and that they bind
        to each other
- term:
    id: GO:0007605
    label: sensory perception of sound
  evidence_type: IMP
  original_reference_id: PMID:16679490
  qualifier: involved_in
  review:
    summary: Sensory perception of sound is a core physiological process supported by CDH23 stereocilium/hair-bundle
      function and hearing-loss genetics.
    action: ACCEPT
    reason: CDH23 is required for normal hearing through its role in stereocilia bundle organization,
      so sound perception should be treated as the core biological-process context for the cadherin/tip-link
      function.
    supported_by:
    - reference_id: PMID:15537665
      supporting_text: CDH23 and PCDH15 play an essential long-term role in maintaining the normal
        organization of the stereocilia bundle
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Required for normal hearing.
- term:
    id: GO:0032420
    label: stereocilium
  evidence_type: ISS
  original_reference_id: PMID:12485990
  qualifier: located_in
  review:
    summary: Stereocilium localization is directly supported and central to CDH23 function in
      hair cells.
    action: ACCEPT
    reason: CDH23 is present in developing stereocilia and participates in tip-link/hair-bundle
      architecture.
    supported_by:
    - reference_id: PMID:12485990
      supporting_text: cadherin 23 are both present in the growing stereocilia
- term:
    id: GO:0050957
    label: equilibrioception
  evidence_type: IMP
  original_reference_id: PMID:16679490
  qualifier: involved_in
  review:
    summary: This phenotype-level sensory annotation is supported but should be kept as non-core
      relative to the molecular cadherin/stereocilium function.
    action: KEEP_AS_NON_CORE
    reason: CDH23 mutations cause hearing/vestibular/retinal phenotypes, but the core gene product
      role is stereocilium adhesion and hair-bundle organization.
    supported_by:
    - reference_id: PMID:15537665
      supporting_text: CDH23 and PCDH15 play an essential long-term role in maintaining the normal
        organization of the stereocilia bundle
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Required for normal hearing.
- term:
    id: GO:0007605
    label: sensory perception of sound
  evidence_type: IMP
  original_reference_id: PMID:15537665
  qualifier: acts_upstream_of_or_within
  review:
    summary: Sensory perception of sound is a core physiological process supported by CDH23 stereocilium/hair-bundle
      function and hearing-loss genetics.
    action: ACCEPT
    reason: CDH23 is required for normal hearing through its role in stereocilia bundle organization,
      so sound perception should be treated as the core biological-process context for the cadherin/tip-link
      function.
    supported_by:
    - reference_id: PMID:15537665
      supporting_text: CDH23 and PCDH15 play an essential long-term role in maintaining the normal
        organization of the stereocilia bundle
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Required for normal hearing.
- term:
    id: GO:0016020
    label: membrane
  evidence_type: NAS
  original_reference_id: PMID:11138009
  qualifier: located_in
  review:
    summary: CDH23 is a single-pass type I membrane protein, so membrane/plasma membrane localization
      is appropriate.
    action: ACCEPT
    reason: The broad membrane annotation is true, with stereocilium/hair-bundle membrane localization
      being more informative.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: 'SUBCELLULAR LOCATION: Cell membrane'
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Single-pass type I membrane protein
- term:
    id: GO:0016339
    label: calcium-dependent cell-cell adhesion
  evidence_type: NAS
  original_reference_id: PMID:11138009
  qualifier: involved_in
  review:
    summary: Cell-cell adhesion is a core cadherin-23 function, with calcium-dependent cadherin
      interactions supporting stereocilia cohesion and tip-link anchoring.
    action: ACCEPT
    reason: CDH23 is a cadherin family adhesion protein required for stereocilia bundle organization;
      the broad adhesion terms are appropriate, though stereocilium/tip-link context is the most
      informative framing.
    supported_by:
    - reference_id: file:human/CDH23/CDH23-uniprot.txt
      supporting_text: Cadherins are calcium-dependent cell adhesion proteins.
    - reference_id: PMID:12485990
      supporting_text: cadherin 23 are both present in the growing stereocilia and that they bind
        to each other
references:
- id: GO_REF:0000002
  title: Gene Ontology annotation through association of InterPro records with GO terms
  findings: []
- id: GO_REF:0000033
  title: Annotation inferences using phylogenetic trees
  findings: []
- id: GO_REF:0000120
  title: Combined Automated Annotation using Multiple IEA Methods
  findings: []
- id: PMID:11138009
  title: "Mutation of CDH23, encoding a new member of the cadherin gene family, causes Usher syndrome type 1D."
  findings: []
- id: PMID:12485990
  title: "Myosin VIIa, harmonin and cadherin 23, three Usher I gene products that cooperate to shape the sensory hair cell bundle."
  findings: []
- id: PMID:15537665
  title: Digenic inheritance of deafness caused by mutations in genes encoding cadherin 23 and
    protocadherin 15 in mice and humans.
  findings: []
- id: PMID:16679490
  title: Survey of the frequency of USH1 gene mutations in a cohort of Usher patients shows the
    importance of cadherin 23 and protocadherin 15 genes and establishes a detection rate of above
    90%.
  findings: []
- id: PMID:17234811
  title: A functional study of plasma-membrane calcium-pump isoform 2 mutants causing digenic
    deafness.
  findings: []
- id: PMID:22879593
  title: "Large protein assemblies formed by multivalent interactions between cadherin23 and harmonin suggest a stable anchorage structure at the tip link of stereocilia."
  findings: []
- id: file:human/CDH23/CDH23-uniprot.txt
  title: UniProt record for CDH23
  findings: []
core_functions:
- molecular_function:
    id: GO:0045296
    label: cadherin binding
  description: CDH23 mediates calcium-dependent cadherin interactions in the stereocilium tip-link/hair-bundle
    apparatus, anchoring CDH23-harmonin and related Usher protein assemblies needed for stereocilia
    cohesion and mechanotransduction.
  directly_involved_in:
  - id: GO:0007605
    label: sensory perception of sound
  locations:
  - id: GO:0032420
    label: stereocilium
  - id: GO:0005886
    label: plasma membrane
  supported_by:
  - reference_id: PMID:12485990
    supporting_text: cadherin 23 are both present in the growing stereocilia and that they bind
      to each other
  - reference_id: PMID:22879593
    supporting_text: cadherin23/harmonin complexes
  - reference_id: file:human/CDH23/CDH23-uniprot.txt
    supporting_text: CDH23 is required for establishing and/or maintaining the proper organization
      of the stereocilia bundle
proposed_new_terms: []
suggested_questions:
- question: Which CDH23 domains or isoforms, if any, are mechanistically relevant to pituitary
    adenoma predisposition rather than hearing/retinal biology?
- question: Can PITA5-associated CDH23 variants be shown to perturb cadherin adhesion, mechanotransduction-related
    assemblies, or a pituitary-specific interaction network?
suggested_experiments:
- description: Test PITA5-associated CDH23 variants in cadherin adhesion and harmonin/PCDH15 interaction
    assays alongside known hearing-loss variants.
  hypothesis: PITA5-associated variants may perturb CDH23 adhesion or scaffold interactions differently
    from classic hearing-loss variants.
- description: Assess CDH23 isoform expression and subcellular localization in normal pituitary
    and pituitary adenoma samples.
  hypothesis: Pituitary-relevant isoform expression or localization would explain how CDH23 variation
    contributes to adenoma predisposition.