CHEK1

UniProt ID: O14757
Organism: Homo sapiens
Review Status: COMPLETE
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Gene Description

CHEK1 (checkpoint kinase 1, Chk1) is a nuclear serine/threonine-protein kinase (EC 2.7.11.1) of the CAMK group and the principal effector kinase of the ATR-dependent DNA replication-stress and DNA-damage checkpoints. It has an N-terminal catalytic domain and a C-terminal autoinhibitory regulatory region; the isolated kinase domain is markedly more active than the full-length protein. Activated by ATR phosphorylation at Ser317 and Ser345 (with Claspin/CLSPN as mediator and BRCA1 and FEM1B as modulators) followed by Ser296 autophosphorylation, Chk1 restrains cyclin-dependent kinase activity to enforce the intra-S and G2/M checkpoints. Its central outputs are phosphorylation of the CDC25 phosphatases (targeting CDC25A for degradation and creating a 14-3-3 docking site on CDC25C Ser216) and stabilization of WEE1 (indirectly, via phosphorylation of PABIR1/FAM122A and activation of PP2A-B55alpha), which together suppress CDK1/CDK2 activity, limit replication-origin firing, and protect stalled replication forks. Chk1 additionally phosphorylates substrates that coordinate homologous-recombination repair (RAD51 Thr309), the Fanconi anemia/BRCA cross-link pathway (FANCE), the p53 network (p53, MDMX), chromatin/transcription (histone H3 Thr11; TLK1), mitotic fidelity (Aurora B Ser331, NEK6, NEK11) and other targets. It is predominantly nuclear but shuttles to the cytoplasm and localizes to interphase centrosomes, where it shields centrosomal CDK1 from premature activation by CDC25B. Chk1 is an Hsp90/Cdc37 client, and activated Chk1 is turned over by SCF(FBXO6)-mediated ubiquitination to terminate the checkpoint. The gene is essential; complete loss causes early embryonic lethality.

Existing Annotations Review

GO Term Evidence Action Reason
GO:0000077 DNA damage checkpoint signaling
IDA
PMID:12660173
Human Tousled like kinases are targeted by an ATM- and Chk1-...
ACCEPT
Summary: CHK1 acts in the DNA-damage checkpoint by phosphorylating checkpoint substrates such as TLK1.
Reason: IDA for DNA damage checkpoint signaling; CHK1 is the effector kinase of this pathway. Core function.
Supporting Evidence:
PMID:12660173
Chk1 phosphorylates Tlk1 on serine 695 (S695) in vitro
GO:0000077 DNA damage checkpoint signaling
IDA
PMID:16963448
Repeated phosphopeptide motifs in human Claspin are phosphor...
ACCEPT
Summary: CHK1 is the ATR-activated effector of DNA-damage checkpoint signaling; here via Claspin phosphorylation.
Reason: IDA well supported; core checkpoint-signaling function.
Supporting Evidence:
PMID:16963448
Thr-916 on Claspin is phosphorylated by Chk1
GO:0000077 DNA damage checkpoint signaling
IEA
GO_REF:0000002
ACCEPT
Summary: InterPro-based IEA for DNA damage checkpoint signaling, consistent with CHK1 core function.
Reason: IEA from the Chk1 catalytic-domain InterPro signature; correct and consistent with experimental evidence.
GO:0000077 DNA damage checkpoint signaling
IMP
PMID:19716789
The F box protein Fbx6 regulates Chk1 stability and cellular...
ACCEPT
Summary: CHK1 stability/turnover experiments (Fbx6) place it in DNA-damage checkpoint signaling.
Reason: IMP consistent with CHK1 as the checkpoint effector kinase; core function.
Supporting Evidence:
PMID:19716789
an Fbx6-containing SCF (Skp1-Cul1-F box) E3 ligase, which mediates the ubiquitination and degradation of Chk1
GO:0000781 chromosome, telomeric region
IDA
PMID:15149599
Telomere shortening triggers senescence of human cells throu...
KEEP AS NON CORE
Summary: Colocalizes with the telomeric region in senescent cells; a context-specific localization.
Reason: Telomere-associated DNA-damage foci form in senescence; peripheral to CHK1 core checkpoint-kinase role and not fully resolvable from the cached abstract.
Supporting Evidence:
PMID:15149599
Telomeric foci containing multiple DNA damage response factors were assembled in a subset of senescent cells
GO:0000785 chromatin
IEA
GO_REF:0000107
ACCEPT
Summary: Chromatin localization (IEA from mouse ortholog), consistent with chromatin-associated CHK1.
Reason: CHK1 associates with chromatin in cycling cells; IEA is appropriate.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0000785 chromatin
ISS
GO_REF:0000024
ACCEPT
Summary: Chromatin localization by ISS from mouse ortholog; consistent with chromatin-associated CHK1.
Reason: Supported by experimental chromatin association of human CHK1.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0000794 condensed nuclear chromosome
IDA
PMID:9382850
Atm-dependent interactions of a mammalian chk1 homolog with ...
KEEP AS NON CORE
Summary: CHK1 localizes along condensed meiotic chromosomes in spermatocytes.
Reason: Genuine but context-specific (meiotic) localization; peripheral to the somatic checkpoint-kinase core.
Supporting Evidence:
PMID:9382850
Chk1 accumulates in late zygotene and pachytene spermatocytes and is present along synapsed meiotic chromosomes
GO:0004672 protein kinase activity
IEA
GO_REF:0000120
ACCEPT
Summary: Protein kinase activity (IEA); CHK1 is a bona fide protein kinase.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Broad parent term, correct.
GO:0004672 protein kinase activity
IMP
PMID:22024163
Phosphorylation at serine 331 is required for Aurora B activ...
ACCEPT
Summary: Kinase activity inferred from Aurora B Ser331 phosphorylation by CHK1.
Reason: IMP; CHK1 kinase activity is well established.
Supporting Evidence:
PMID:22024163
Chk1 in vitro kinase assay
GO:0004674 protein serine/threonine kinase activity
IDA
PMID:16963448
Repeated phosphopeptide motifs in human Claspin are phosphor...
ACCEPT
Summary: Protein serine/threonine kinase activity, the core molecular function of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
Supporting Evidence:
PMID:16963448
Thr-916 on Claspin is phosphorylated by Chk1
GO:0004674 protein serine/threonine kinase activity
IDA
PMID:9278511
Conservation of the Chk1 checkpoint pathway in mammals: link...
ACCEPT
Summary: CHK1 phosphorylates CDC25 phosphatases on Ser/Thr; defining catalytic activity.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
Supporting Evidence:
PMID:9278511
Chk1 bound to and phosphorylated the dual-specificity protein phosphatases Cdc25A, Cdc25B, and Cdc25C
GO:0004674 protein serine/threonine kinase activity
IEA
GO_REF:0000120
ACCEPT
Summary: Ser/Thr kinase activity (IEA, EC 2.7.11.1); consistent with all experimental evidence.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-5685230
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-5685242
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-6799246
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-69604
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-75010
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-9007539
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-9943662
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0004674 protein serine/threonine kinase activity
TAS
Reactome:R-HSA-9943678
ACCEPT
Summary: Protein serine/threonine kinase activity (Reactome TAS) for CHK1 checkpoint phosphorylation events.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function. Reactome models CHK1-catalyzed phosphorylation of CDC25A, TP53, RAD51, BRCA2, E2F6, NEK11.
GO:0005515 protein binding
IPI
PMID:11836499
BRCA1 regulates the G2/M checkpoint by activating Chk1 kinas...
REMOVE
Summary: Generic protein binding to CDC25C.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. CDC25C is a CHK1 substrate; substrate binding is implied by the kinase activity.
Supporting Evidence:
PMID:11836499
BRCA1 is essential for activating the Chk1 kinase that regulates DNA damage-induced G2/M arrest
GO:0005515 protein binding
IPI
PMID:11836499
BRCA1 regulates the G2/M checkpoint by activating Chk1 kinas...
REMOVE
Summary: Generic protein binding to BRCA1.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. BRCA1 is an upstream activator of CHK1; interaction is regulatory, not a distinct MF.
Supporting Evidence:
PMID:11836499
BRCA1 is essential for activating the Chk1 kinase that regulates DNA damage-induced G2/M arrest
GO:0005515 protein binding
IPI
PMID:15665856
The cell-cycle checkpoint kinase Chk1 is required for mammal...
REMOVE
Summary: Generic protein binding to RAD51.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. RAD51 is a CHK1 substrate (Thr309); substrate binding implied by kinase activity.
Supporting Evidence:
PMID:15665856
RAD51 is phosphorylated on Thr 309 in a Chk1-dependent manner
GO:0005515 protein binding
IPI
PMID:15798197
Coupling of human circadian and cell cycles by the timeless ...
REMOVE
Summary: Generic protein binding to TIMELESS.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. TIMELESS is a checkpoint scaffold/regulator interacting with CHK1.
Supporting Evidence:
PMID:15798197
human Timeless protein interacts with both the circadian clock protein cryptochrome 2 and with the cell cycle checkpoint proteins Chk1
GO:0005515 protein binding
IPI
PMID:16330544
Chaperoning checkpoint kinase 1 (Chk1), an Hsp90 client, wit...
MODIFY
Summary: CHK1 is an Hsp90/Cdc37 chaperone client.
Reason: Resolve generic protein binding to the informative MF Hsp90 protein binding; CHK1 is an established Hsp90 client.
Proposed replacements: Hsp90 protein binding
Supporting Evidence:
PMID:16330544
is destabilized when heat shock protein 90 (Hsp90) is inhibited, suggesting that Chk1 is an Hsp90 client
GO:0005515 protein binding
IPI
PMID:16330544
Chaperoning checkpoint kinase 1 (Chk1), an Hsp90 client, wit...
REMOVE
Summary: Generic protein binding to CDC25C.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. CDC25C is a CHK1 substrate; substrate binding implied by kinase activity.
Supporting Evidence:
PMID:9278511
Chk1 bound to and phosphorylated the dual-specificity protein phosphatases Cdc25A, Cdc25B, and Cdc25C
GO:0005515 protein binding
IPI
PMID:16511572
14-3-3gamma binds to MDMX that is phosphorylated by UV-activ...
REMOVE
Summary: Generic protein binding to TP53.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. TP53 is a CHK1 substrate; interaction captured by kinase activity on p53.
Supporting Evidence:
PMID:10673501
recombinant hCHK1, but not a kinase-defective version of hCHK1, can phosphorylate p53 in vitro at S20
GO:0005515 protein binding
IPI
PMID:16963448
Repeated phosphopeptide motifs in human Claspin are phosphor...
REMOVE
Summary: Generic protein binding to CLSPN.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. Claspin is the CHK1-activation mediator and a CHK1 substrate.
Supporting Evidence:
PMID:16963448
Thr-916 on Claspin is phosphorylated by Chk1
GO:0005515 protein binding
IPI
PMID:17380128
Phosphorylation of pRB at Ser612 by Chk1/2 leads to a comple...
REMOVE
Summary: Generic protein binding to RB1.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. RB1 is phosphorylated by CHK1 (Ser612); substrate binding implied by kinase activity.
Supporting Evidence:
PMID:17380128
the phosphorylation of pRB at Ser612 was conducted by Chk1/2 after DNA damage
GO:0005515 protein binding
IPI
PMID:19223857
Tumor suppressor protein C53 antagonizes checkpoint kinases ...
REMOVE
Summary: Generic protein binding to CDK5RAP3 (C53/LZAP).
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. C53 antagonizes CHK1; regulatory interaction, no distinct MF term.
Supporting Evidence:
PMID:19223857
C53 interacts with Chk1 and antagonizes its function
GO:0005515 protein binding
IPI
PMID:19330022
Human FEM1B is required for Rad9 recruitment and CHK1 activa...
REMOVE
Summary: Generic protein binding to FEM1B.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. FEM1B is a CHK1-activation adaptor; regulatory interaction.
Supporting Evidence:
PMID:19330022
FEM1B ... is involved in the activation of CHK1
GO:0005515 protein binding
IPI
PMID:19716789
The F box protein Fbx6 regulates Chk1 stability and cellular...
REMOVE
Summary: Generic protein binding to FBXO6.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. FBXO6 (SCF) targets activated CHK1 for degradation; regulatory interaction.
Supporting Evidence:
PMID:19716789
an Fbx6-containing SCF (Skp1-Cul1-F box) E3 ligase, which mediates the ubiquitination and degradation of Chk1
GO:0005515 protein binding
IPI
PMID:20639859
14-3-3gamma mediates Cdc25A proteolysis to block premature m...
MODIFY
Summary: Phospho-CHK1 (Ser296) is bound by 14-3-3 proteins that regulate its localization/activity.
Reason: Resolve generic protein binding to the informative MF 14-3-3 protein binding; a genuine, literature-supported regulatory interaction.
Proposed replacements: 14-3-3 protein binding
Supporting Evidence:
PMID:20639859
14-3-3Ξ³ forms a complex with Chk1 phosphorylated at Ser296
GO:0005515 protein binding
IPI
PMID:20639859
14-3-3gamma mediates Cdc25A proteolysis to block premature m...
MODIFY
Summary: Phospho-CHK1 (Ser296) is bound by 14-3-3 proteins that regulate its localization/activity.
Reason: Resolve generic protein binding to the informative MF 14-3-3 protein binding; a genuine, literature-supported regulatory interaction.
Proposed replacements: 14-3-3 protein binding
Supporting Evidence:
PMID:20639859
14-3-3Ξ³ forms a complex with Chk1 phosphorylated at Ser296
GO:0005515 protein binding
IPI
PMID:22505024
Chk1 phosphorylates the tumour suppressor Mig-6, regulating ...
REMOVE
Summary: Generic protein binding to ERRFI1 (Mig-6).
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. Mig-6 is a CHK1 substrate (Ser251); substrate binding implied by kinase activity.
Supporting Evidence:
PMID:22505024
S251 and S302, but not S273, S276, or S326, were phosphorylated by Chk1 in vitro
GO:0005515 protein binding
IPI
PMID:22939624
Quantitative analysis of HSP90-client interactions reveals p...
MODIFY
Summary: CHK1 is an Hsp90/Cdc37 chaperone client.
Reason: Resolve generic protein binding to the informative MF Hsp90 protein binding; CHK1 is an established Hsp90 client.
Proposed replacements: Hsp90 protein binding
Supporting Evidence:
PMID:22939624
CDC37 provides recognition of the kinase family
GO:0005515 protein binding
IPI
PMID:25249323
ATR/Chk1/Smurf1 pathway determines cell fate after DNA damag...
REMOVE
Summary: Generic protein binding to SMURF1.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. Smurf1 is phosphorylated by CHK1; substrate binding implied by kinase activity.
Supporting Evidence:
PMID:25249323
phosphorylation of Smurf1 that enhances its self-degradation
GO:0005515 protein binding
IPI
PMID:28514442
Architecture of the human interactome defines protein commun...
REMOVE
Summary: Generic protein binding to HLA-DRB5.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. High-throughput (BioPlex) interactome hit; no functional relevance for a nuclear checkpoint kinase.
Supporting Evidence:
PMID:28514442
networks of protein-protein interactions
GO:0005515 protein binding
IPI
PMID:33961781
Dual proteome-scale networks reveal cell-specific remodeling...
REMOVE
Summary: Generic protein binding to HLA-DRB5.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. High-throughput (BioPlex) interactome hit; no functional relevance for a nuclear checkpoint kinase.
Supporting Evidence:
PMID:33961781
cell-specific remodeling of the human interactome
GO:0005515 protein binding
IPI
PMID:35271311
OpenCell: Endogenous tagging for the cartography of human ce...
MODIFY
Summary: Phospho-CHK1 (Ser296) is bound by 14-3-3 proteins that regulate its localization/activity.
Reason: Resolve generic protein binding to the informative MF 14-3-3 protein binding; a genuine, literature-supported regulatory interaction.
Proposed replacements: 14-3-3 protein binding
Supporting Evidence:
PMID:35271311
systematically map the localization and interactions of human proteins
GO:0005515 protein binding
IPI
PMID:35512704
Systematic discovery of mutation-directed neo-protein-protei...
REMOVE
Summary: Generic protein binding to TP53.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. High-throughput neo-PPI screen; TP53 is in any case a CHK1 substrate.
Supporting Evidence:
PMID:35512704
mutation-directed neo-protein-protein interactions in cancer
GO:0005515 protein binding
IPI
PMID:35839996
A Proteomic Approach Identifies Isoform-Specific and Nucleot...
REMOVE
Summary: Generic protein binding to HRAS.
Reason: Generic protein binding (GO:0005515) is uninformative as a molecular function; removal does not assert the interaction is false. High-throughput RAS-interactome hit; not an established functional CHK1 interaction.
Supporting Evidence:
PMID:35839996
HRAS-specific CARM1 and CHK1
GO:0005515 protein binding
IPI
PMID:36931259
A central chaperone-like role for 14-3-3 proteins in human c...
MODIFY
Summary: Phospho-CHK1 (Ser296) is bound by 14-3-3 proteins that regulate its localization/activity.
Reason: Resolve generic protein binding to the informative MF 14-3-3 protein binding; a genuine, literature-supported regulatory interaction.
Proposed replacements: 14-3-3 protein binding
Supporting Evidence:
PMID:36931259
14-3-3 proteins are highly conserved regulatory proteins that interact with hundreds of structurally diverse clients
GO:0005515 protein binding
IPI
PMID:40205054
Multimodal cell maps as a foundation for structural and func...
MODIFY
Summary: Phospho-CHK1 (Ser296) is bound by 14-3-3 proteins that regulate its localization/activity.
Reason: Resolve generic protein binding to the informative MF 14-3-3 protein binding; a genuine, literature-supported regulatory interaction.
Proposed replacements: 14-3-3 protein binding
Supporting Evidence:
PMID:40205054
joint measurement of biophysical interactions
GO:0005524 ATP binding
IEA
GO_REF:0000002
ACCEPT
Summary: ATP binding, required for CHK1 phosphotransfer catalysis.
Reason: IEA from kinase InterPro signatures; CHK1 binds ATP in its catalytic cleft. Correct and consistent with kinase activity.
GO:0005576 extracellular region
HDA
PMID:23580065
Shotgun proteomics reveals specific modulated protein patter...
REMOVE
Summary: Extracellular detection in a shotgun proteomics survey of glaucoma tears.
Reason: HDA biofluid proteomics (tears); CHK1 is a nuclear checkpoint kinase, not an extracellular protein. This detection does not reflect a site of function.
Supporting Evidence:
PMID:23580065
shotgun proteomics approach to tears of patients with POAG
GO:0005634 nucleus
EXP
PMID:11836499
BRCA1 regulates the G2/M checkpoint by activating Chk1 kinas...
ACCEPT
Summary: Nuclear localization (EXP); the nucleus is the principal site of CHK1 function.
Reason: CHK1 is predominantly nuclear; core location.
GO:0005634 nucleus
EXP
PMID:12676962
Regulation of Chk1 includes chromatin association and 14-3-3...
ACCEPT
Summary: Nuclear localization (EXP); the nucleus is the principal site of CHK1 function.
Reason: CHK1 is predominantly nuclear; core location.
GO:0005634 nucleus
EXP
PMID:15710331
Lack of PTEN sequesters CHK1 and initiates genetic instabili...
ACCEPT
Summary: Nuclear localization (EXP); the nucleus is the principal site of CHK1 function.
Reason: CHK1 is predominantly nuclear; core location.
GO:0005634 nucleus
EXP
PMID:9278511
Conservation of the Chk1 checkpoint pathway in mammals: link...
ACCEPT
Summary: Nuclear localization (EXP); the nucleus is the principal site of CHK1 function.
Reason: CHK1 is predominantly nuclear; core location.
GO:0005634 nucleus
IDA
PMID:15311285
Centrosome-associated Chk1 prevents premature activation of ...
ACCEPT
Summary: Nuclear localization (IDA).
Reason: Core CHK1 location.
GO:0005634 nucleus
IDA
PMID:20932473
DNA damage activates a spatially distinct late cytoplasmic c...
ACCEPT
Summary: Nuclear localization (IDA, MK2 checkpoint study).
Reason: Nuclear CHK1 activity establishes the G2/M checkpoint.
Supporting Evidence:
PMID:20932473
nuclear Chk1 activity is essential to establish a G(2)/M checkpoint
GO:0005634 nucleus
IDA
PMID:26296656
hHR23A is required to control the basal turnover of Chk1.
ACCEPT
Summary: Nuclear localization (IDA).
Reason: Core CHK1 location.
GO:0005634 nucleus
IEA
GO_REF:0000120
ACCEPT
Summary: Nuclear localization (IEA, subcellular-location mapping).
Reason: Consistent with experimental nuclear localization.
GO:0005654 nucleoplasm
IDA
GO_REF:0000052
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-176116
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-5684882
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-5684887
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-5685230
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-5685242
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-6799246
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-69604
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-75010
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-9007539
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-9943662
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005654 nucleoplasm
TAS
Reactome:R-HSA-9943678
ACCEPT
Summary: Nucleoplasm localization (IDA/Reactome TAS).
Reason: CHK1 acts in the nucleoplasm; consistent with its nuclear checkpoint role.
GO:0005657 replication fork
IEA
GO_REF:0000107
ACCEPT
Summary: Replication fork localization (IEA from mouse ortholog).
Reason: CHK1 acts at stalled/active replication forks in the intra-S checkpoint; consistent with chromatin association.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0005694 chromosome
EXP
PMID:12676962
Regulation of Chk1 includes chromatin association and 14-3-3...
ACCEPT
Summary: Chromosome/chromatin localization (EXP).
Reason: CHK1 is chromatin-associated; consistent location.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0005694 chromosome
EXP
PMID:31316063
SPRTN protease and checkpoint kinase 1 cross-activation loop...
ACCEPT
Summary: Chromosome/chromatin localization (EXP).
Reason: CHK1 is chromatin-associated; consistent location.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0005694 chromosome
EXP
PMID:9382850
Atm-dependent interactions of a mammalian chk1 homolog with ...
ACCEPT
Summary: Chromosome/chromatin localization (EXP).
Reason: CHK1 is chromatin-associated; consistent location.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0005694 chromosome
IEA
GO_REF:0000044
ACCEPT
Summary: Chromosome localization (IEA).
Reason: Consistent with experimental chromatin association.
GO:0005737 cytoplasm
EXP
PMID:12676962
Regulation of Chk1 includes chromatin association and 14-3-3...
ACCEPT
Summary: Cytoplasmic localization (EXP); CHK1 shuttles between nucleus and cytoplasm.
Reason: CHK1 undergoes CRM1/XPO1-dependent nuclear export; cytoplasmic pool is real.
Supporting Evidence:
PMID:12676962
Chk1 is associated with chromatin in cycling cells
GO:0005737 cytoplasm
IDA
PMID:26296656
hHR23A is required to control the basal turnover of Chk1.
ACCEPT
Summary: Cytoplasmic localization (IDA).
Reason: CHK1 shuttles to the cytoplasm; consistent location.
GO:0005737 cytoplasm
IEA
GO_REF:0000044
ACCEPT
Summary: Cytoplasmic localization (IEA).
Reason: Consistent with nucleocytoplasmic shuttling of CHK1.
GO:0005813 centrosome
IDA
PMID:15311285
Centrosome-associated Chk1 prevents premature activation of ...
ACCEPT
Summary: Interphase centrosome localization (IDA); centrosomal CHK1 shields CDK1 from premature activation.
Reason: Well documented; core aspect of CHK1 control of mitotic timing.
Supporting Evidence:
PMID:15311285
human Chk1 kinase localizes to interphase, but not mitotic, centrosomes
GO:0005813 centrosome
IEA
GO_REF:0000044
ACCEPT
Summary: Centrosome localization (IEA).
Reason: Consistent with experimental centrosomal CHK1.
Supporting Evidence:
PMID:15311285
human Chk1 kinase localizes to interphase, but not mitotic, centrosomes
GO:0005829 cytosol
TAS
Reactome:R-HSA-205328
REMOVE
Summary: Cytosol localization derived from Reactome R-HSA-205328.
Reason: R-HSA-205328 ("Interaction of other tyrosine kinases with p-KIT") explicitly concerns "CHK1 (Csk homologous kinase or MATK)", a cytoplasmic tyrosine kinase, NOT CHEK1/Chk1 checkpoint kinase. This is a gene-name collision; the annotation is mis-attributed to the wrong protein.
Supporting Evidence:
Reactome:R-HSA-205328
CHK1 (Csk homologous kinase or MATK), FER and FES associate with p-KIT
GO:0006260 DNA replication
TAS
Reactome:R-HSA-176187
MODIFY
Summary: CHK1 restrains DNA replication (origin firing, fork progression) but does not perform replication.
Reason: GO:0006260 (DNA replication) implies the replication process itself; CHK1 regulates it. Reactome models CHK1 in ATR-dependent reduction of origin firing.
Proposed replacements: regulation of DNA replication
Supporting Evidence:
Reactome:R-HSA-176187
reduction of replication origin firing
GO:0006281 DNA repair
IMP
PMID:19716789
The F box protein Fbx6 regulates Chk1 stability and cellular...
KEEP AS NON CORE
Summary: CHK1 is required for efficient DNA repair (HRR) after replication stress.
Reason: IMP for DNA repair (broad term). CHK1 regulates rather than executes repair; retain as a genuine but non-core, downstream role.
Supporting Evidence:
PMID:15665856
Chk1 is a key regulator of genome maintenance by the homologous recombination repair (HRR) system
GO:0006338 chromatin remodeling
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Chromatin remodeling (IEA) linked to CHK1 histone H3T11 phosphorylation / transcriptional repression.
Reason: CHK1 modulates chromatin-templated transcription via H3T11ph; "chromatin remodeling" is a broad, downstream framing. Non-core.
Supporting Evidence:
PMID:18243098
H3-T11 phosphorylation occurs throughout the cell cycle and is Chk1 dependent in vivo
GO:0006338 chromatin remodeling
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Chromatin remodeling (ISS from mouse ortholog).
Reason: Same downstream H3T11-linked role; non-core.
Supporting Evidence:
PMID:18243098
H3-T11 phosphorylation occurs throughout the cell cycle and is Chk1 dependent in vivo
GO:0006974 DNA damage response
IMP
PMID:20932473
DNA damage activates a spatially distinct late cytoplasmic c...
ACCEPT
Summary: CHK1 is central to the DNA damage response (IMP).
Reason: Broad but correct; CHK1 is a core DDR effector.
Supporting Evidence:
PMID:20932473
nuclear Chk1 activity is essential to establish a G(2)/M checkpoint
GO:0006974 DNA damage response
ISS
GO_REF:0000024
ACCEPT
Summary: DNA damage response (ISS from mouse ortholog).
Reason: Consistent with core DDR role.
GO:0007095 mitotic G2 DNA damage checkpoint signaling
IBA
GO_REF:0000033
ACCEPT
Summary: IBA places CHK1 in mitotic G2 DNA damage checkpoint signaling, its core process.
Reason: PAINT node PTN002388913 infers the G2 DNA-damage checkpoint; CHK1 itself is among the descendant evidences (correct, not circular). Core function.
Supporting Evidence:
file:human/CHEK1/CHEK1-deep-research-falcon.md
Its primary function is to phosphorylate proteins that restrain cyclin-dependent kinase activity, suppress inappropriate replication-origin firing, stabilize or protect stressed replication forks, coordinate homologous recombination
GO:0007095 mitotic G2 DNA damage checkpoint signaling
IMP
PMID:20932473
DNA damage activates a spatially distinct late cytoplasmic c...
ACCEPT
Summary: Nuclear CHK1 establishes the mitotic G2 DNA-damage checkpoint (IMP).
Reason: Core checkpoint function.
Supporting Evidence:
PMID:20932473
nuclear Chk1 activity is essential to establish a G(2)/M checkpoint
GO:0010569 regulation of double-strand break repair via homologous recombination
IDA
PMID:15665856
The cell-cycle checkpoint kinase Chk1 is required for mammal...
ACCEPT
Summary: CHK1 regulates DSB repair via homologous recombination by phosphorylating RAD51 (Thr309).
Reason: Direct, specific role: CHK1 promotes RAD51-dependent HRR. Well supported.
Supporting Evidence:
PMID:15665856
RAD51 is phosphorylated on Thr 309 in a Chk1-dependent manner
PMID:15665856
Chk1 is a key regulator of genome maintenance by the homologous recombination repair (HRR) system
GO:0019904 protein domain specific binding
IPI
PMID:26296656
hHR23A is required to control the basal turnover of Chk1.
KEEP AS NON CORE
Summary: Domain-specific binding to the hHR23A/RAD23A UBA domains, regulating CHK1 turnover.
Reason: A more informative term than generic protein binding; reflects regulation of CHK1 basal turnover. Non-core.
Supporting Evidence:
PMID:26296656
hHR23A associates with Chk1 through its UBA domains
GO:0032991 protein-containing complex
IDA
PMID:26296656
hHR23A is required to control the basal turnover of Chk1.
REMOVE
Summary: Membership in an unspecified protein-containing complex (hHR23A study).
Reason: GO:0032991 is the root complex term and is uninformative; CHK1 acts as a largely monomeric kinase with transient interactions rather than as a defined stable complex subunit. Removal does not assert the interaction is false.
Supporting Evidence:
PMID:26296656
hHR23A associates with Chk1 through its UBA domains
GO:0035402 histone H3T11 kinase activity
IBA
GO_REF:0000033
ACCEPT
Summary: Histone H3T11 kinase activity (IBA).
Reason: PAINT-inferred, supported by direct human/mouse evidence; a genuine specific MF.
Supporting Evidence:
PMID:18243098
Chk1 is a histone H3 threonine 11 kinase that regulates DNA damage-induced transcriptional repression
GO:0035402 histone H3T11 kinase activity
IDA
PMID:18243098
Chk1 is a histone H3 threonine 11 kinase that regulates DNA ...
ACCEPT
Summary: CHK1 phosphorylates histone H3 on Thr11 (IDA).
Reason: Direct experimental demonstration.
Supporting Evidence:
PMID:18243098
Chk1 is a histone H3 threonine 11 kinase that regulates DNA damage-induced transcriptional repression
GO:0035402 histone H3T11 kinase activity
IEA
GO_REF:0000107
ACCEPT
Summary: Histone H3T11 kinase activity (IEA from mouse ortholog).
Reason: Consistent with human IDA evidence.
Supporting Evidence:
PMID:18243098
Chk1 is a histone H3 threonine 11 kinase that regulates DNA damage-induced transcriptional repression
GO:0042770 signal transduction in response to DNA damage
IDA
PMID:16963448
Repeated phosphopeptide motifs in human Claspin are phosphor...
ACCEPT
Summary: Signal transduction in response to DNA damage (IDA) via Claspin.
Reason: CHK1 is a core DNA-damage signal transducer.
Supporting Evidence:
PMID:16963448
Thr-916 on Claspin is phosphorylated by Chk1
GO:0042770 signal transduction in response to DNA damage
IEA
GO_REF:0000117
ACCEPT
Summary: Signal transduction in response to DNA damage (IEA/ARBA).
Reason: Consistent with core CHK1 signaling role.
GO:0044818 mitotic G2/M transition checkpoint
IDA
PMID:9278511
Conservation of the Chk1 checkpoint pathway in mammals: link...
ACCEPT
Summary: CHK1 enforces the mitotic G2/M transition checkpoint by inhibiting CDC25.
Reason: Core function; CHK1 phosphorylates CDC25C Ser216 to block CDK1 activation. Modeled in gocams/653b0ce600000623 and modules/g2_m_transition.yaml.
Supporting Evidence:
PMID:9278511
Chk1 phosphorylates Cdc25C on serine-216
GO:0044818 mitotic G2/M transition checkpoint
IMP
PMID:33108758
CHK1 Inhibitor Blocks Phosphorylation of FAM122A and Promote...
ACCEPT
Summary: G2/M transition checkpoint via CHK1-FAM122A-PP2A-WEE1 axis (IMP with CHK1 inhibitor).
Reason: Core checkpoint function; CHK1 phosphorylates FAM122A to stabilize WEE1 and activate the G2/M checkpoint.
Supporting Evidence:
PMID:33108758
CHK1 directly phosphorylated FAM122A on a highly conserved site, Ser37
PMID:33108758
CHK1 is known to directly phosphorylate WEE1 thereby activating a G2/M checkpoint
GO:0045787 positive regulation of cell cycle
IDA
PMID:26296656
hHR23A is required to control the basal turnover of Chk1.
MARK AS OVER ANNOTATED
Summary: Positive regulation of cell cycle (IDA) from an hHR23A/CHK1-turnover study.
Reason: CHK1 is fundamentally a checkpoint/negative regulator; the cited paper concerns hHR23A control of Chk1 protein turnover, not a positive cell-cycle-driving activity. Over-annotation.
Supporting Evidence:
PMID:26296656
hHR23A acts as a carrier to promote Chk1 degradation through the Ubiquitin Proteasome System
GO:0045814 negative regulation of gene expression, epigenetic
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Epigenetic negative regulation of gene expression via H3T11 phosphorylation.
Reason: Genuine but downstream/context-specific transcriptional-repression role; non-core relative to checkpoint-kinase action.
Supporting Evidence:
PMID:18243098
Chk1 is a histone H3 threonine 11 kinase that regulates DNA damage-induced transcriptional repression
GO:0045814 negative regulation of gene expression, epigenetic
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Epigenetic negative regulation of gene expression (ISS).
Reason: Same H3T11-linked role; non-core.
Supporting Evidence:
PMID:18243098
Chk1 is a histone H3 threonine 11 kinase that regulates DNA damage-induced transcriptional repression
GO:0045839 negative regulation of mitotic nuclear division
IDA
PMID:15311285
Centrosome-associated Chk1 prevents premature activation of ...
ACCEPT
Summary: Centrosomal CHK1 negatively regulates mitotic nuclear division by preventing premature CDK1 activation.
Reason: Direct evidence: forced/active centrosomal CHK1 blocks centrosomal CDK1 activation and premature mitosis.
Supporting Evidence:
PMID:15311285
wild-type Chk1 impaired activation of centrosome-associated Cdk1, thereby resulting in DNA endoreplication and centrosome amplification
GO:0046602 regulation of mitotic centrosome separation
IDA
PMID:15311285
Centrosome-associated Chk1 prevents premature activation of ...
ACCEPT
Summary: CHK1 regulates mitotic centrosome separation; its inhibition causes premature separation.
Reason: Direct IDA; centrosome-associated CHK1 restrains premature centrosome separation.
Supporting Evidence:
PMID:15311285
Chemical inhibition of Chk1 resulted in premature centrosome separation
GO:0070317 negative regulation of G0 to G1 transition
TAS
Reactome:R-HSA-8953750
KEEP AS NON CORE
Summary: Negative regulation of G0-to-G1 transition (Reactome, via E2F6 phosphorylation).
Reason: CHK1 phosphorylates E2F6 during replication stress to modulate E2F-target transcription; a downstream, context-specific role. Non-core.
Supporting Evidence:
Reactome:R-HSA-9007539
CHEK1-mediated phosphorylation prevents association of E2F6 with its target promoters
GO:0071260 cellular response to mechanical stimulus
IEP
PMID:19593445
Expression of the Bcl-2 protein BAD promotes prostate cancer...
MARK AS OVER ANNOTATED
Summary: Cellular response to mechanical stimulus (IEP) from a BAD/prostate-cancer study.
Reason: IEP (expression-pattern) evidence; the cited paper concerns BAD in prostate cancer and does not establish a mechanical-stimulus response role for CHK1. Peripheral over-annotation.
Supporting Evidence:
PMID:19593445
Expression of the Bcl-2 protein BAD promotes prostate cancer growth
GO:0090399 replicative senescence
NAS
PMID:15149599
Telomere shortening triggers senescence of human cells throu...
KEEP AS NON CORE
Summary: Replicative senescence (NAS) associated with telomeric DNA-damage signaling.
Reason: CHK1 participates in checkpoint signaling that can contribute to senescence; NAS and peripheral to the core checkpoint-kinase function.
Supporting Evidence:
PMID:15149599
Telomeric foci containing multiple DNA damage response factors were assembled in a subset of senescent cells
GO:0106310 protein serine kinase activity
EXP
PMID:10673501
The human homologs of checkpoint kinases Chk1 and Cds1 (Chk2...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:12660173
Human Tousled like kinases are targeted by an ATM- and Chk1-...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:14559997
Chk1 kinase negatively regulates mitotic function of Cdc25A ...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:15659650
p53 C-terminal phosphorylation by CHK1 and CHK2 participates...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:15665856
The cell-cycle checkpoint kinase Chk1 is required for mammal...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:16511572
14-3-3gamma binds to MDMX that is phosphorylated by UV-activ...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:16963448
Repeated phosphopeptide motifs in human Claspin are phosphor...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:17296736
Chk1-mediated phosphorylation of FANCE is required for the F...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:17380128
Phosphorylation of pRB at Ser612 by Chk1/2 leads to a comple...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:18317453
The checkpoint kinases Chk1 and Chk2 regulate the functional...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:18728393
Nek6 is involved in G2/M phase cell cycle arrest through DNA...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:31316063
SPRTN protease and checkpoint kinase 1 cross-activation loop...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
EXP
PMID:9278511
Conservation of the Chk1 checkpoint pathway in mammals: link...
ACCEPT
Summary: Protein serine kinase activity (EXP); the core catalytic activity of CHK1.
Reason: CHK1 is a serine/threonine-protein kinase (EC 2.7.11.1); catalytic kinase activity is its defining, core molecular function.
GO:0106310 protein serine kinase activity
IEA
GO_REF:0000116
ACCEPT
Summary: Protein serine kinase activity (IEA, Rhea reaction mapping).
Reason: Consistent with the EC 2.7.11.1 catalytic activity.
GO:1901796 regulation of signal transduction by p53 class mediator
TAS
Reactome:R-HSA-6804756
KEEP AS NON CORE
Summary: Regulation of p53-class signaling (Reactome TAS) via CHK1 phosphorylation of TP53.
Reason: CHK1 phosphorylates p53 (e.g. S20) to stabilize/activate it; a genuine but downstream, context-specific role. Non-core.
Supporting Evidence:
Reactome:R-HSA-6804756
CHEK1-mediated phosphorylation of TP53 at S20
GO:1901988 negative regulation of cell cycle phase transition
IEA
GO_REF:0000117
ACCEPT
Summary: Negative regulation of cell cycle phase transition (IEA/ARBA).
Reason: Correct high-level BP: CHK1 restrains CDK-driven phase transitions at checkpoints.

Core Functions

ATR-activated effector serine/threonine kinase that phosphorylates the CDC25 phosphatases (CDC25A for degradation; CDC25C on Ser216 to create a 14-3-3 docking site) to restrain cyclin-dependent kinase activity and enforce the mitotic G2/M transition checkpoint.

Supporting Evidence:

Effector kinase of the intra-S/replication checkpoint that limits origin firing and protects stalled replication forks, acting at chromatin and replication forks; also stabilizes WEE1 via phosphorylation of PABIR1/FAM122A and activation of PP2A-B55alpha.

Supporting Evidence:
  • PMID:33108758
    CHK1 directly phosphorylated FAM122A on a highly conserved site, Ser37
  • PMID:31316063
    SPRTN cleaves, evicts and activates CHK1 from replicative chromatin

Promotes homologous-recombination repair of DNA double-strand breaks by phosphorylating RAD51 on Thr309 (releasing RAD51 from BRCA2 and enhancing its chromatin association).

Supporting Evidence:
  • PMID:15665856
    RAD51 is phosphorylated on Thr 309 in a Chk1-dependent manner
  • PMID:15665856
    Chk1 is a key regulator of genome maintenance by the homologous recombination repair (HRR) system

Histone H3 Threonine-11 kinase that couples DNA-damage checkpoint signaling to transcriptional repression of cell-cycle genes (e.g. CCNB1, CDK1) via loss of H3T11 phosphorylation and reduced histone acetylation.

Supporting Evidence:
  • PMID:18243098
    Chk1 is a histone H3 threonine 11 kinase that regulates DNA damage-induced transcriptional repression
  • PMID:18243098
    H3-T11 phosphorylation occurs throughout the cell cycle and is Chk1 dependent in vivo

References

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Suggested Questions for Experts

Q: Which human CHK1 substrates identified by analog-sensitive phosphoproteomics (e.g. KAP1/TRIM28 Ser473, FEN1, RIF1, Treslin/TICRR) are direct in vivo targets with defined checkpoint consequences, versus in vitro candidates?

Q: Is WEE1 a direct human CHK1 substrate, or is CHK1 regulation of WEE1 principally indirect through the FAM122A-PP2A-B55alpha axis?

Q: How is CHK1 signaling terminated during checkpoint recovery, and how do SCF(FBXO6) degradation, hHR23A-mediated basal turnover, and PPM1D/PP1/PP2A dephosphorylation quantitatively partition this control?

Suggested Experiments

Experiment: Analog-sensitive CHK1 (gatekeeper mutant) with a bulky ATP analog plus rapid inhibition and quantitative phosphoproteomics in synchronized cells to distinguish direct, cell-cycle-resolved CHK1 substrates from indirect targets.

Experiment: Degron-based acute CHK1 depletion combined with DNA fiber assays and origin-mapping to separate CHK1 control of origin firing from its control of fork stability in unperturbed versus stressed S phase.

Experiment: Separation-of-function CHK1 phosphosite/localization mutants (Ser317, Ser345, Ser296, centrosome-tethered) to map how compartmentalization directs distinct substrate sets (nuclear CDC25A/C versus centrosomal CDK1 shielding).

Deep Research

Falcon

(CHEK1-deep-research-falcon.md)

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πŸ“š Additional Documentation

Notes

(CHEK1-notes.md)

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