IP6K3

UniProt ID: Q96PC2
Organism: Homo sapiens
Review Status: COMPLETE
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Gene Description

IP6K3 encodes inositol hexakisphosphate kinase 3, a tissue-enriched member of the inositol phosphokinase family that phosphorylates soluble inositol polyphosphates. Its core activity converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/5-PP-InsP5) and can also phosphorylate InsP5/inositol pyrophosphate substrates, contributing to inositol pyrophosphate signaling. The experimentally supported localization is predominantly cytoplasmic (with detectable nuclear pool by GFP fusion in HEK293/C2C12). In addition to its catalytic role, IP6K3 has a noncatalytic scaffolding function in cerebellar Purkinje cells where it binds spectrin and adducin and is required for normal Purkinje dendritic architecture and synaptogenesis; in non-neural tissues (skeletal muscle, heart, thyroid) IP6K3 contributes to systemic metabolic regulation. Annotations implying protein phosphorylation or phosphatidylinositol-lipid metabolism should be interpreted as misapplied to a soluble inositol phosphate kinase.

Existing Annotations Review

GO Term Evidence Action Reason
GO:0005634 nucleus
IBA
GO_REF:0000033
KEEP AS NON CORE
Summary: Nuclear localization is not the main experimentally supported localization for human IP6K3; the original characterization describes IP6K3 as predominantly cytoplasmic. The falcon deep research summary explicitly states IP6K3 predominates in the cytoplasm, though a GFP-IP6K3 fusion can show a detectable nuclear pool in HEK293/C2C12 cells, suggesting partial nuclear accessibility rather than a primary nuclear role.
Reason: Per PR #848 review feedback: a detectable nuclear pool is experimentally supported (PMID:27577108, AcGFP-IP6K3 in both cytoplasm and nucleus in C2C12 cells). The predominant/core localization remains cytoplasm/cytosol, so nucleus is retained as a valid non-core localization (changed from MARK_AS_OVER_ANNOTATED).
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
file:human/IP6K3/IP6K3-deep-research-manual.md
PMID:11502751 reports that InsP(6)K3 is enriched in brain and predominates in the cytoplasm, whereas InsP(6)K2 is exclusively nuclear and InsP(6)K1 has comparable nuclear and cytosolic densities.
file:human/IP6K3/IP6K3-deep-research-falcon.md
A GFP-tagged IP6K3 fusion protein localized to **both cytoplasm and nucleus** in HEK293 cells, consistent with the idea that IP6K3-derived inositol pyrophosphates can affect both cytosolic and nuclear signaling processes.
PMID:27577108
Furthermore, when an AcGFP-human IP6K3 fusion protein construct was transfected into mouse C2C12 cells, the resultant AcGFP-IP6K3 was localized to both the cytoplasm and the nucleus (Fig. S3).
GO:0005737 cytoplasm
IBA
GO_REF:0000033
ACCEPT
Summary: cytoplasm localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
file:human/IP6K3/IP6K3-deep-research-falcon.md
A GFP-tagged IP6K3 fusion protein localized to **both cytoplasm and nucleus** in HEK293 cells, consistent with the idea that IP6K3-derived inositol pyrophosphates can affect both cytosolic and nuclear signaling processes.
GO:0046854 phosphatidylinositol phosphate biosynthetic process
IBA
GO_REF:0000033
MODIFY
Summary: phosphatidylinositol phosphate biosynthetic process describes phosphatidylinositol lipid metabolism, but the evidence for IP6K3 is soluble inositol phosphate/pyrophosphate metabolism.
Reason: IP6K3 is not primarily a phosphatidylinositol lipid kinase. The supported biological process is soluble inositol phosphate biosynthesis/metabolism.
Supporting Evidence:
PMID:11502751
We now report the identification, cloning, and characterization of a third InsP(7) forming enzyme designated InsP(6)K3.
GO:0000828 inositol hexakisphosphate kinase activity
IBA
GO_REF:0000033
ACCEPT
Summary: inositol hexakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
file:human/IP6K3/IP6K3-deep-research-manual.md
UniProt describes the curated function as conversion of InsP6 to InsP7/PP-InsP5, and conversion of InsP5 to PP-InsP4.
file:human/IP6K3/IP6K3-deep-research-falcon.md
Across IP6Ks (including IP6K3), the best-supported catalytic reaction is: **IP6 β†’ 5-IP7 (5-InsP7 / 5-diphosphoinositol pentakisphosphate)**, and further phosphorylation such as **1-IP7 β†’ InsP8**.
GO:0032958 inositol phosphate biosynthetic process
IBA
GO_REF:0000033
ACCEPT
Summary: inositol phosphate biosynthetic process is supported because IP6K3 biosynthesizes/metabolizes soluble inositol pyrophosphates from inositol phosphate substrates.
Reason: This biological-process term matches the characterized IP6K3 reaction products and Reactome inositol phosphate pathway context.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5).
file:human/IP6K3/IP6K3-deep-research-falcon.md
A primary study describing IP6K (family) substrate scope notes that the **5-kinase activity** can act on multiple highly phosphorylated inositols, including: **InsP5 β†’ 5-PP-InsP4**, **InsP6 β†’ 5-PP-InsP5 (5-InsP7)**, **1-PP-InsP5 β†’ InsP8**.
GO:0000166 nucleotide binding
IEA
GO_REF:0000043
MARK AS OVER ANNOTATED
Summary: nucleotide binding is a broad parent term that does not convey IP6K3 substrate specificity.
Reason: The annotation should be interpreted through the specific inositol hexakisphosphate/inositol pyrophosphate kinase activity terms. Broad kinase, transferase, and nucleotide-binding terms are over-general.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
The curated function identifies IP6K3 as an inositol hexakisphosphate kinase with specific inositol polyphosphate substrates.
GO:0000827 inositol-1,3,4,5,6-pentakisphosphate kinase activity
IEA
GO_REF:0000117
ACCEPT
Summary: inositol-1,3,4,5,6-pentakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0000828 inositol hexakisphosphate kinase activity
IEA
GO_REF:0000117
ACCEPT
Summary: inositol hexakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0000829 diphosphoinositol pentakisphosphate kinase activity
IEA
GO_REF:0000117
ACCEPT
Summary: diphosphoinositol pentakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0000832 inositol hexakisphosphate 5-kinase activity
IEA
GO_REF:0000116
ACCEPT
Summary: inositol hexakisphosphate 5-kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0005524 ATP binding
IEA
GO_REF:0000043
ACCEPT
Summary: ATP binding is required because IP6K3 uses ATP as the phosphate donor for inositol polyphosphate kinase reactions.
Reason: ATP binding is mechanistically relevant, though less specific than the inositol phosphate kinase activity terms.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Catalytic reactions list ATP as substrate and ADP as product for IP6K3 reactions.
GO:0005737 cytoplasm
IEA
GO_REF:0000044
ACCEPT
Summary: cytoplasm localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0016301 kinase activity
IEA
GO_REF:0000120
MARK AS OVER ANNOTATED
Summary: kinase activity is a broad parent term that does not convey IP6K3 substrate specificity.
Reason: The annotation should be interpreted through the specific inositol hexakisphosphate/inositol pyrophosphate kinase activity terms. Broad kinase, transferase, and nucleotide-binding terms are over-general.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
The curated function identifies IP6K3 as an inositol hexakisphosphate kinase with specific inositol polyphosphate substrates.
GO:0016740 transferase activity
IEA
GO_REF:0000043
MARK AS OVER ANNOTATED
Summary: transferase activity is a broad parent term that does not convey IP6K3 substrate specificity.
Reason: The annotation should be interpreted through the specific inositol hexakisphosphate/inositol pyrophosphate kinase activity terms. Broad kinase, transferase, and nucleotide-binding terms are over-general.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
The curated function identifies IP6K3 as an inositol hexakisphosphate kinase with specific inositol polyphosphate substrates.
GO:0032958 inositol phosphate biosynthetic process
IEA
GO_REF:0000002
ACCEPT
Summary: inositol phosphate biosynthetic process is supported because IP6K3 biosynthesizes/metabolizes soluble inositol pyrophosphates from inositol phosphate substrates.
Reason: This biological-process term matches the characterized IP6K3 reaction products and Reactome inositol phosphate pathway context.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5).
GO:0043647 inositol phosphate metabolic process
IEA
GO_REF:0000117
ACCEPT
Summary: inositol phosphate metabolic process is supported because IP6K3 biosynthesizes/metabolizes soluble inositol pyrophosphates from inositol phosphate substrates.
Reason: This biological-process term matches the characterized IP6K3 reaction products and Reactome inositol phosphate pathway context.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5).
GO:0046488 phosphatidylinositol metabolic process
IEA
GO_REF:0000117
MODIFY
Summary: phosphatidylinositol metabolic process describes phosphatidylinositol lipid metabolism, but the evidence for IP6K3 is soluble inositol phosphate/pyrophosphate metabolism.
Reason: IP6K3 is not primarily a phosphatidylinositol lipid kinase. The supported biological process is soluble inositol phosphate biosynthesis/metabolism.
Supporting Evidence:
PMID:11502751
We now report the identification, cloning, and characterization of a third InsP(7) forming enzyme designated InsP(6)K3.
file:human/IP6K3/IP6K3-deep-research-manual.md
The available reviewed function text and the PMID:11502751 abstract support inositol phosphate kinase activity rather than direct phosphorylation of protein substrates.
GO:0052836 inositol 5-diphosphate pentakisphosphate 5-kinase activity
IEA
GO_REF:0000117
ACCEPT
Summary: inositol 5-diphosphate pentakisphosphate 5-kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0052839 diphosphoinositol tetrakisphosphate kinase activity
IEA
GO_REF:0000117
ACCEPT
Summary: diphosphoinositol tetrakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0090407 organophosphate biosynthetic process
IEA
GO_REF:0000117
MARK AS OVER ANNOTATED
Summary: Organophosphate biosynthetic process is too broad for IP6K3 when inositol phosphate biosynthetic process is already annotated.
Reason: The more informative process is soluble inositol phosphate/pyrophosphate biosynthesis. This broad parent term obscures the pathway specificity.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
IP6K3 converts inositol hexakisphosphate to diphosphoinositol pentakisphosphate.
GO:0005515 protein binding
IPI
PMID:32296183
A reference map of the human binary protein interactome.
MARK AS OVER ANNOTATED
Summary: Generic protein binding from a high-throughput interactome screen does not describe IP6K3 catalytic function.
Reason: GO:0005515 is uninformative here. The core annotation should focus on inositol phosphate kinase activity and inositol phosphate biosynthesis.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
The curated function describes conversion of inositol phosphate substrates, while listed interactions are high-throughput IntAct records.
GO:0005634 nucleus
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Nuclear localization is not the main experimentally supported localization for human IP6K3; the original characterization describes IP6K3 as predominantly cytoplasmic.
Reason: Per PR #848 review feedback: a detectable nuclear pool is experimentally supported (PMID:27577108, AcGFP-IP6K3 in both cytoplasm and nucleus in C2C12 cells). The predominant/core localization remains cytoplasm/cytosol, so nucleus is retained as a valid non-core localization (changed from MARK_AS_OVER_ANNOTATED).
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0043647 inositol phosphate metabolic process
TAS
Reactome:R-HSA-1855167
ACCEPT
Summary: inositol phosphate metabolic process is supported because IP6K3 biosynthesizes/metabolizes soluble inositol pyrophosphates from inositol phosphate substrates.
Reason: This biological-process term matches the characterized IP6K3 reaction products and Reactome inositol phosphate pathway context.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5).
GO:0000828 inositol hexakisphosphate kinase activity
TAS
Reactome:R-HSA-1855227
ACCEPT
Summary: inositol hexakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0000829 diphosphoinositol pentakisphosphate kinase activity
TAS
Reactome:R-HSA-1855194
ACCEPT
Summary: diphosphoinositol pentakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0052836 inositol 5-diphosphate pentakisphosphate 5-kinase activity
TAS
Reactome:R-HSA-1855158
ACCEPT
Summary: inositol 5-diphosphate pentakisphosphate 5-kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0052839 diphosphoinositol tetrakisphosphate kinase activity
TAS
Reactome:R-HSA-1855193
ACCEPT
Summary: diphosphoinositol tetrakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0000827 inositol-1,3,4,5,6-pentakisphosphate kinase activity
TAS
Reactome:R-HSA-1855223
ACCEPT
Summary: inositol-1,3,4,5,6-pentakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
GO:0000828 inositol hexakisphosphate kinase activity
IDA
PMID:11502751
Identification and characterization of a novel inositol hexa...
ACCEPT
Summary: inositol hexakisphosphate kinase activity fits the characterized IP6K3 catalytic activity toward soluble inositol polyphosphate and inositol pyrophosphate substrates.
Reason: This is the specific molecular-function space for IP6K3. The PMID:11502751 characterization and UniProt catalytic activities support conversion of InsP6 to PP-InsP5/InsP7 and related inositol polyphosphate kinase reactions.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5). Converts 1,3,4,5,6-pentakisphosphate (InsP5) to PP-InsP4.
file:human/IP6K3/IP6K3-deep-research-falcon.md
IP6K3 (inositol hexakisphosphate kinase 3; EC 2.7.4.21)** is a human inositol phosphate kinase that catalyzes pyrophosphate formation on highly phosphorylated inositols, most prominently converting **InsP6 (IP6) to 5-InsP7 (5-IP7)** and participating in downstream generation of **InsP8**.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1855158
ACCEPT
Summary: cytosol localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1855193
ACCEPT
Summary: cytosol localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1855194
ACCEPT
Summary: cytosol localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1855223
ACCEPT
Summary: cytosol localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1855227
ACCEPT
Summary: cytosol localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
GO:0000831 inositol hexakisphosphate 6-kinase activity
IDA
PMID:11502751
Identification and characterization of a novel inositol hexa...
MARK AS OVER ANNOTATED
Summary: Per PR #848 review feedback: the falcon deep research and Reactome both establish IP6K3's physiological activity as a 5-kinase (InsP6 β†’ 5-PP-InsP5 / 5-InsP7), captured by the separately-ACCEPTed GO:0000832 (inositol hexakisphosphate 5-kinase activity). The 6-position assignment in this older IDA annotation (PMID:11502751, 2001) is not the established physiological positional specificity for IP6K3, so this annotation is downgraded ACCEPT β†’ MARK_AS_OVER_ANNOTATED.
Reason: IP6K3's experimentally and physiologically established activity is 5-kinase (5-PP-InsP5), not 6-kinase; all Reactome IP6K3 reactions and the falcon evidence describe 5-position pyrophosphorylation. The correct MF is GO:0000832 (5-kinase), already ACCEPTed.
Supporting Evidence:
file:human/IP6K3/IP6K3-deep-research-falcon.md
Reported IP6K 5-kinase activity includes pyrophosphorylation of **InsP5 β†’ 5-PP-InsP4**, **InsP6 β†’ 5-PP-InsP5 (5-InsP7)**, and **1-PP-InsP5 β†’ InsP8**; thus IP6K3 shares family substrate scope for highly phosphorylated inositols, with best-established physiological product **5-InsP7**.
GO:0032958 inositol phosphate biosynthetic process
IDA
PMID:11502751
Identification and characterization of a novel inositol hexa...
ACCEPT
Summary: inositol phosphate biosynthetic process is supported because IP6K3 biosynthesizes/metabolizes soluble inositol pyrophosphates from inositol phosphate substrates.
Reason: This biological-process term matches the characterized IP6K3 reaction products and Reactome inositol phosphate pathway context.
Supporting Evidence:
file:human/IP6K3/IP6K3-uniprot.txt
Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5).
file:human/IP6K3/IP6K3-deep-research-falcon.md
In mammals, **IP6Ks** (IP6K1/2/3) are central enzymes that produce **5-InsP7 (5-IP7)** from **InsP6 (IP6)** and can contribute to production of **InsP8** from **1-IP7**.
GO:0006468 protein phosphorylation
IDA
PMID:11502751
Identification and characterization of a novel inositol hexa...
REMOVE
Summary: IP6K3 is an inositol polyphosphate kinase, not a protein kinase. The characterized substrates are InsP6, InsP5, and related inositol pyrophosphates.
Reason: The evidence supports phosphorylation of soluble inositol phosphate substrates, not protein phosphorylation. This row should not remain as a protein phosphorylation BP annotation.
Supporting Evidence:
PMID:11502751
We now report the identification, cloning, and characterization of a third InsP(7) forming enzyme designated InsP(6)K3.
GO:0046488 phosphatidylinositol metabolic process
IDA
PMID:11502751
Identification and characterization of a novel inositol hexa...
MODIFY
Summary: phosphatidylinositol metabolic process describes phosphatidylinositol lipid metabolism, but the evidence for IP6K3 is soluble inositol phosphate/pyrophosphate metabolism.
Reason: IP6K3 is not primarily a phosphatidylinositol lipid kinase. The supported biological process is soluble inositol phosphate biosynthesis/metabolism.
Supporting Evidence:
PMID:11502751
We now report the identification, cloning, and characterization of a third InsP(7) forming enzyme designated InsP(6)K3.
GO:0005737 cytoplasm
IDA
PMID:11502751
Identification and characterization of a novel inositol hexa...
ACCEPT
Summary: cytoplasm localization is supported by the original characterization, which reported IP6K3 as predominantly cytoplasmic.
Reason: Cytoplasmic/cytosolic localization is the experimentally supported cellular component for human IP6K3.
Supporting Evidence:
PMID:11502751
Intracellular disposition discriminates the three enzymes with InsP(6)K2 being exclusively nuclear, InsP(6)K3 predominating in the cytoplasm, and InsP(6)K1 displaying comparable nuclear and cytosolic densities.
file:human/IP6K3/IP6K3-deep-research-falcon.md
In mouse cerebellum, immunostaining supports localization within the **molecular layer/Purkinje cell compartment**, consistent with Purkinje-cell phenotypes observed in knockout animals.
GO:0030674 protein-macromolecule adaptor activity
IPI
PMID:26245967
Inositol Hexakisphosphate Kinase-3 Regulates the Morphology ...
NEW
Summary: Beyond its catalytic kinase activity, IP6K3 has a distinct, experimentally demonstrated noncatalytic scaffolding/adaptor molecular function in cerebellar Purkinje cells: it physiologically binds Ξ±/Ξ²-adducin and Ξ±2/Ξ²2-spectrin and promotes their mutual interaction. Critically, this activity is independent of catalysis (the kinase-dead K217A mutant retains it), establishing it as a bona fide molecular function rather than a downstream phenotype. Added as a NEW adaptor-activity annotation per PR #848 review feedback. Provenance corrected per PR #848 re-review: the primary evidence is the direct physical interaction reported in PMID:26245967, so the annotation now uses evidence_type IPI with original_reference_id PMID:26245967 (the falcon deep research file is retained as corroborating support).
Reason: A specific protein-macromolecule adaptor activity captures IP6K3's kinase-independent scaffolding role (linking spectrin and adducin) far better than generic protein binding; it is the primary reason IP6K3 is biologically distinctive among the IP6K family.
Supporting Evidence:
PMID:26245967
IP6K3 physiologically binds to the cytoskeletal proteins adducin and spectrin, whose mutual interactions are perturbed in IP6K3-null mutants.
file:human/IP6K3/IP6K3-deep-research-falcon.md
IP6K3 co-immunoprecipitates with **Ξ±/Ξ²-adducin** and **Ξ±2/Ξ²2-spectrin**, and these interactions were reported as **specific to IP6K3** (not observed similarly for IP6K1/2 in that study). Importantly, IP6K3's promotion of spectrin–adducin interaction was reported to be **independent of kinase activity**, consistent with a **noncatalytic scaffolding role** (kinase-dead mutant K217A still promotes binding).

Core Functions

Phosphorylates soluble inositol polyphosphates, especially converting InsP6 to diphosphoinositol pentakisphosphate/InsP7 and supporting inositol pyrophosphate biosynthesis in the cytoplasm.

Supporting Evidence:
  • file:human/IP6K3/IP6K3-uniprot.txt
    Converts inositol hexakisphosphate (InsP6) to diphosphoinositol pentakisphosphate (InsP7/PP-InsP5).
  • file:human/IP6K3/IP6K3-deep-research-manual.md
    Reactome R-HSA-1855227 describes IP6K1 and IP6K3 phosphorylating IP6 to 5-PP-IP5 in the cytosol.

Noncatalytic scaffolding/adaptor function in cerebellar Purkinje cells: IP6K3 binds Ξ±/Ξ²-adducin and Ξ±2/Ξ²2-spectrin and promotes their mutual interaction, organizing the submembranous cytoskeleton required for normal Purkinje dendritic architecture and synapse number. This role is independent of kinase catalysis (kinase-dead K217A retains it), making IP6K3 a determinant of neuronal cytoskeletal organization distinct from its inositol-pyrophosphate kinase activity.

Cellular Locations:
Supporting Evidence:
  • file:human/IP6K3/IP6K3-deep-research-falcon.md
    IP6K3 co-immunoprecipitates with **Ξ±/Ξ²-adducin** and **Ξ±2/Ξ²2-spectrin**, and these interactions were reported as **specific to IP6K3** (not observed similarly for IP6K1/2 in that study). Importantly, IP6K3's promotion of spectrin–adducin interaction was reported to be **independent of kinase activity**, consistent with a **noncatalytic scaffolding role** (kinase-dead mutant K217A still promotes binding).

References

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Suggested Questions for Experts

Q: What fraction of endogenous IP6K3 is nuclear in human neurons compared with the predominant cytoplasmic pool?

Suggested experts: inositol phosphate biochemists, neurocell biologists

Q: Which inositol pyrophosphate products are generated by IP6K3 in brain tissue under physiological conditions?

Suggested experts: metabolomics experts, neurochemists

Suggested Experiments

Experiment: Quantify inositol pyrophosphate species in IP6K3 knockout and rescue neuronal models.

Hypothesis: IP6K3 loss reduces specific InsP7/PP-InsP products in brain-derived cells.

Type: Targeted metabolomics

Experiment: Use validated IP6K3 antibodies or tagged knock-in alleles to measure cytoplasmic versus nuclear localization in neuronal cells.

Hypothesis: Endogenous IP6K3 is predominantly cytoplasmic, consistent with the original characterization.

Type: Endogenous localization

Deep Research

Falcon

(IP6K3-deep-research-falcon.md)

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Manual

(IP6K3-deep-research-manual.md)

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πŸ“„ View Raw YAML

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