MAPT

UniProt ID: P10636
Organism: Homo sapiens
Review Status: COMPLETE
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Gene Description

MAPT encodes microtubule-associated protein tau, a neuronally enriched, intrinsically disordered microtubule-associated protein. Tau binds tubulin and microtubules through repeat domains, promotes microtubule assembly and stability, and contributes to axonal microtubule organization, axonal transport, neurite/axon development, and neuronal polarity. Tau also associates with neural plasma-membrane and axonal cortex components, and disease-associated phosphorylation or acetylation can alter microtubule and membrane binding. In Alzheimer disease and other tauopathies, modified tau mislocalizes and self-assembles into paired-helical-filament/neurofibrillary-tangle pathology with downstream synaptic, mitochondrial, glial, secretion, and stress-response consequences.

Existing Annotations Review

GO Term Evidence Action Reason
GO:0000226 microtubule cytoskeleton organization
IBA
GO_REF:0000033
ACCEPT
Summary: microtubule cytoskeleton organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0000226 (microtubule cytoskeleton organization), the IBA annotation with qualifier involved_in from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0004857 enzyme inhibitor activity
IBA
GO_REF:0000033
KEEP AS NON CORE
Summary: enzyme inhibitor activity is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0004857 (enzyme inhibitor activity), the IBA annotation with qualifier enables from GO_REF:0000033 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0008017 microtubule binding
IBA
GO_REF:0000033
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the IBA annotation with qualifier enables from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0015630 microtubule cytoskeleton
IBA
GO_REF:0000033
ACCEPT
Summary: microtubule cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015630 (microtubule cytoskeleton), the IBA annotation with qualifier is_active_in from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
IBA
GO_REF:0000033
ACCEPT
Summary: axon is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030424 (axon), the IBA annotation with qualifier is_active_in from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0060632 regulation of microtubule-based movement
IBA
GO_REF:0000033
ACCEPT
Summary: regulation of microtubule-based movement is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0060632 (regulation of microtubule-based movement), the IBA annotation with qualifier involved_in from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0140778 microtubule stabilizing activity
IBA
GO_REF:0000033
ACCEPT
Summary: microtubule stabilizing activity is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0140778 (microtubule stabilizing activity), the IBA annotation with qualifier enables from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031175 neuron projection development
IBA
GO_REF:0000033
ACCEPT
Summary: neuron projection development is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031175 (neuron projection development), the IBA annotation with qualifier involved_in from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005576 extracellular region
IEA
GO_REF:0000044
KEEP AS NON CORE
Summary: extracellular region is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005576 (extracellular region), the IEA annotation with qualifier located_in from GO_REF:0000044 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005829 cytosol
IEA
GO_REF:0000044
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the IEA annotation with qualifier located_in from GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005856 cytoskeleton
IEA
GO_REF:0000044
ACCEPT
Summary: cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005856 (cytoskeleton), the IEA annotation with qualifier located_in from GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005886 plasma membrane
IEA
GO_REF:0000044
ACCEPT
Summary: plasma membrane is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005886 (plasma membrane), the IEA annotation with qualifier located_in from GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0008017 microtubule binding
IEA
GO_REF:0000002
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the IEA annotation with qualifier enables from GO_REF:0000002 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0015631 tubulin binding
IEA
GO_REF:0000002
ACCEPT
Summary: tubulin binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015631 (tubulin binding), the IEA annotation with qualifier enables from GO_REF:0000002 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
IEA
GO_REF:0000044
ACCEPT
Summary: axon is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030424 (axon), the IEA annotation with qualifier located_in from GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0030425 dendrite
IEA
GO_REF:0000044
KEEP AS NON CORE
Summary: dendrite is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030425 (dendrite), the IEA annotation with qualifier located_in from GO_REF:0000044 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1904115 axon cytoplasm
IEA
GO_REF:0000108
ACCEPT
Summary: axon cytoplasm is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1904115 (axon cytoplasm), the IEA annotation with qualifier located_in from GO_REF:0000108 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005515 protein binding
IPI
PMID:16275660
Identification of VCP/p97, carboxyl terminus of Hsp70-intera...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:16275660 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:16446437
Abeta and tau form soluble complexes that may promote self a...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:16446437 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:17317006
14-3-3zeta facilitates GSK3beta-catalyzed tau phosphorylatio...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:17317006 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:18292230
Akt and CHIP coregulate tau degradation through coordinated ...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:18292230 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:19138662
Effect of phosphorylation on interaction of human tau protei...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:19138662 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:19527721
Excitatory amino acid transporter 2 associates with phosphor...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:19527721 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:19647741
Phosphorylation of more than one site is required for tight ...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:19647741 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:21692989
Tyrosine phosphorylation of tau regulates its interactions w...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:21692989 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:24028865
Impact of the cellular prion protein on amyloid-Ξ² and 3PO-ta...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:24028865 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:25156556
AlphaScreen HTS and live-cell bioluminescence resonance ener...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:25156556 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:26402096
Identification of Novel Tau Interactions with Endoplasmic Re...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:26402096 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:28650319
Somatostatin binds to the human amyloid Ξ² peptide and favors...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:28650319 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:29764935
The disorderly conduct of Hsc70 and its interaction with the...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:29764935 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:30668577
Role of tau N-terminal motif in the secretion of human tau b...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:30668577 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:31640277
GSKIP-Mediated Anchoring Increases Phosphorylation of Tau by...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:31640277 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:32814053
Interactome Mapping Provides a Network of Neurodegenerative ...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:32814053 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:33961781
Dual proteome-scale networks reveal cell-specific remodeling...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:33961781 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:34757590
Targeting Tau Mitigates Mitochondrial Fragmentation and Oxid...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:34757590 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:7566652
ApoE3 binding to tau tandem repeat I is abolished by tau ser...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:7566652 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0042802 identical protein binding
IPI
PMID:16446437
Abeta and tau form soluble complexes that may promote self a...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IPI annotation with qualifier enables from PMID:16446437 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0042802 identical protein binding
IPI
PMID:30158706
Structures of filaments from Pick's disease reveal a novel t...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IPI annotation with qualifier enables from PMID:30158706 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0042802 identical protein binding
IPI
PMID:30894745
Novel tau filament fold in chronic traumatic encephalopathy ...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IPI annotation with qualifier enables from PMID:30894745 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005886 plasma membrane
IDA
GO_REF:0000052
ACCEPT
Summary: plasma membrane is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005886 (plasma membrane), the IDA annotation with qualifier located_in from GO_REF:0000052 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005576 extracellular region
EXP
PMID:32272059
A Translocation Pathway for Vesicle-Mediated Unconventional ...
KEEP AS NON CORE
Summary: extracellular region is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005576 (extracellular region), the EXP annotation with qualifier located_in from PMID:32272059 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0004857 enzyme inhibitor activity
IDA
PMID:19457097
Tau--an inhibitor of deacetylase HDAC6 function.
KEEP AS NON CORE
Summary: enzyme inhibitor activity is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0004857 (enzyme inhibitor activity), the IDA annotation with qualifier enables from PMID:19457097 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0060632 regulation of microtubule-based movement
IGI
PMID:19457097
Tau--an inhibitor of deacetylase HDAC6 function.
ACCEPT
Summary: regulation of microtubule-based movement is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0060632 (regulation of microtubule-based movement), the IGI annotation with qualifier involved_in from PMID:19457097 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0019901 protein kinase binding
IPI
PMID:25556849
A novel DYRK1A (dual specificity tyrosine phosphorylation-re...
KEEP AS NON CORE
Summary: protein kinase binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019901 (protein kinase binding), the IPI annotation with qualifier enables from PMID:25556849 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9836404
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from Reactome:R-HSA-9836404 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0003779 actin binding
TAS
PMID:22920254
Tau promotes neurodegeneration via DRP1 mislocalization in v...
KEEP AS NON CORE
Summary: actin binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003779 (actin binding), the TAS annotation with qualifier enables from PMID:22920254 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0032930 positive regulation of superoxide anion generation
IMP
PMID:22920254
Tau promotes neurodegeneration via DRP1 mislocalization in v...
KEEP AS NON CORE
Summary: positive regulation of superoxide anion generation is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0032930 (positive regulation of superoxide anion generation), the IMP annotation with qualifier involved_in from PMID:22920254 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0090258 negative regulation of mitochondrial fission
IMP
PMID:22920254
Tau promotes neurodegeneration via DRP1 mislocalization in v...
KEEP AS NON CORE
Summary: negative regulation of mitochondrial fission is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0090258 (negative regulation of mitochondrial fission), the IMP annotation with qualifier involved_in from PMID:22920254 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0019901 protein kinase binding
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: protein kinase binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019901 (protein kinase binding), the ISS annotation with qualifier enables from GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0051087 protein-folding chaperone binding
IPI
PMID:25051234
Intracellular clusterin interacts with brain isoforms of the...
KEEP AS NON CORE
Summary: protein-folding chaperone binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0051087 (protein-folding chaperone binding), the IPI annotation with qualifier enables from PMID:25051234 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0019899 enzyme binding
ISS
PMID:23362255
Isomerase Pin1 stimulates dephosphorylation of tau protein a...
MARK AS OVER ANNOTATED
Summary: enzyme binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019899 (enzyme binding), the ISS annotation with qualifier enables from PMID:23362255 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0042802 identical protein binding
IPI
PMID:27044754
FRMD4A-cytohesin signaling modulates the cellular release of...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IPI annotation with qualifier enables from PMID:27044754 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0042802 identical protein binding
IDA
PMID:15654759
Residual structure in the repeat domain of tau: echoes of mi...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IDA annotation with qualifier enables from PMID:15654759 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0097435 supramolecular fiber organization
IDA
PMID:15654759
Residual structure in the repeat domain of tau: echoes of mi...
ACCEPT
Summary: supramolecular fiber organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0097435 (supramolecular fiber organization), the IDA annotation with qualifier involved_in from PMID:15654759 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005515 protein binding
IPI
PMID:22367970
Abnormal interaction between the mitochondrial fission prote...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:22367970 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0019901 protein kinase binding
IPI
PMID:16923168
Tau-tubulin kinase 1 (TTBK1), a neuron-specific tau kinase c...
KEEP AS NON CORE
Summary: protein kinase binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019901 (protein kinase binding), the IPI annotation with qualifier enables from PMID:16923168 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005737 cytoplasm
IMP
PMID:27044754
FRMD4A-cytohesin signaling modulates the cellular release of...
KEEP AS NON CORE
Summary: cytoplasm is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005737 (cytoplasm), the IMP annotation with qualifier located_in from PMID:27044754 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0051258 protein polymerization
IMP
PMID:27044754
FRMD4A-cytohesin signaling modulates the cellular release of...
ACCEPT
Summary: protein polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0051258 (protein polymerization), the IMP annotation with qualifier involved_in from PMID:27044754 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005515 protein binding
IPI
PMID:26014385
LRRK2 Promotes Tau Accumulation, Aggregation and Release.
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:26014385 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005737 cytoplasm
IMP
PMID:26014385
LRRK2 Promotes Tau Accumulation, Aggregation and Release.
KEEP AS NON CORE
Summary: cytoplasm is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005737 (cytoplasm), the IMP annotation with qualifier located_in from PMID:26014385 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0042802 identical protein binding
IMP
PMID:26014385
LRRK2 Promotes Tau Accumulation, Aggregation and Release.
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IMP annotation with qualifier enables from PMID:26014385 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005829 cytosol
TAS
Reactome:R-NUL-9619534
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from Reactome:R-NUL-9619534 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005829 cytosol
TAS
Reactome:R-NUL-9619539
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from Reactome:R-NUL-9619539 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005515 protein binding
IPI
PMID:24251416
Phosphorylation of human Tau protein by microtubule affinity...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:24251416 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0019901 protein kinase binding
IPI
PMID:24251416
Phosphorylation of human Tau protein by microtubule affinity...
KEEP AS NON CORE
Summary: protein kinase binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019901 (protein kinase binding), the IPI annotation with qualifier enables from PMID:24251416 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0015630 microtubule cytoskeleton
NAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: microtubule cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015630 (microtubule cytoskeleton), the NAS annotation with qualifier located_in from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0015630 microtubule cytoskeleton
IDA
PMID:28377597
Dyrk1A overexpression leads to increase of 3R-tau expression...
ACCEPT
Summary: microtubule cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015630 (microtubule cytoskeleton), the IDA annotation with qualifier located_in from PMID:28377597 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0015630 microtubule cytoskeleton
NAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: microtubule cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015630 (microtubule cytoskeleton), the NAS annotation with qualifier located_in from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005515 protein binding
IPI
PMID:23666762
Role of individual MARK isoforms in phosphorylation of tau a...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:23666762 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005737 cytoplasm
IDA
PMID:23666762
Role of individual MARK isoforms in phosphorylation of tau a...
KEEP AS NON CORE
Summary: cytoplasm is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005737 (cytoplasm), the IDA annotation with qualifier located_in from PMID:23666762 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0030425 dendrite
IDA
PMID:23666762
Role of individual MARK isoforms in phosphorylation of tau a...
KEEP AS NON CORE
Summary: dendrite is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030425 (dendrite), the IDA annotation with qualifier located_in from PMID:23666762 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0044297 cell body
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: cell body is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0044297 (cell body), the ISS annotation with qualifier located_in from GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0045121 membrane raft
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: membrane raft is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0045121 (membrane raft), the ISS annotation with qualifier located_in from GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0097386 glial cell projection
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: glial cell projection is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0097386 (glial cell projection), the ISS annotation with qualifier located_in from GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005515 protein binding
IPI
PMID:28755476
Regulation of the interaction between the neuronal BIN1 isof...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:28755476 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005737 cytoplasm
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: cytoplasm is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005737 (cytoplasm), the ISS annotation with qualifier located_in from GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0008017 microtubule binding
TAS
PMID:26609151
Defects in Synaptic Plasticity, Reduced NMDA-Receptor Transp...
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the TAS annotation with qualifier enables from PMID:26609151 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0044304 main axon
ISS
GO_REF:0000024
ACCEPT
Summary: main axon is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0044304 (main axon), the ISS annotation with qualifier located_in from GO_REF:0000024 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0061564 axon development
TAS
PMID:26609151
Defects in Synaptic Plasticity, Reduced NMDA-Receptor Transp...
ACCEPT
Summary: axon development is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0061564 (axon development), the TAS annotation with qualifier involved_in from PMID:26609151 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0051879 Hsp90 protein binding
IPI
PMID:17517623
Roles of heat-shock protein 90 in maintaining and facilitati...
KEEP AS NON CORE
Summary: Hsp90 protein binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0051879 (Hsp90 protein binding), the IPI annotation with qualifier enables from PMID:17517623 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005515 protein binding
IPI
PMID:25051234
Intracellular clusterin interacts with brain isoforms of the...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:25051234 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0046785 microtubule polymerization
TAS
PMID:26996940
Regulation of Microtubule Assembly by Tau and not by Pin1.
ACCEPT
Summary: microtubule polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0046785 (microtubule polymerization), the TAS annotation with qualifier involved_in from PMID:26996940 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0019901 protein kinase binding
ISS
PMID:17906291
DYRK1A-mediated hyperphosphorylation of Tau. A functional li...
KEEP AS NON CORE
Summary: protein kinase binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019901 (protein kinase binding), the ISS annotation with qualifier enables from PMID:17906291 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0003677 DNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003677 (DNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0003680 minor groove of adenine-thymine-rich DNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: minor groove of adenine-thymine-rich DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003680 (minor groove of adenine-thymine-rich DNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0003690 double-stranded DNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: double-stranded DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003690 (double-stranded DNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0003697 single-stranded DNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: single-stranded DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003697 (single-stranded DNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0003723 RNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: RNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003723 (RNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0003779 actin binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: actin binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003779 (actin binding), the TAS annotation with qualifier enables from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005576 extracellular region
NAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: extracellular region is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005576 (extracellular region), the NAS annotation with qualifier located_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005634 nucleus
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: nucleus is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005634 (nucleus), the TAS annotation with qualifier located_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005739 mitochondrion
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: mitochondrion is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005739 (mitochondrion), the TAS annotation with qualifier located_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005886 plasma membrane
TAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: plasma membrane is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005886 (plasma membrane), the TAS annotation with qualifier located_in from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0007267 cell-cell signaling
NAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: cell-cell signaling is marked over-annotated for MAPT because this biological-process term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0007267 (cell-cell signaling), the NAS annotation with qualifier involved_in from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0008017 microtubule binding
NAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the NAS annotation with qualifier enables from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0016072 rRNA metabolic process
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: rRNA metabolic process is marked over-annotated for MAPT because this biological-process term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0016072 (rRNA metabolic process), the TAS annotation with qualifier involved_in from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0021954 central nervous system neuron development
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: central nervous system neuron development is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0021954 (central nervous system neuron development), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
TAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: axon is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030424 (axon), the TAS annotation with qualifier located_in from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0030674 protein-macromolecule adaptor activity
TAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: protein-macromolecule adaptor activity is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030674 (protein-macromolecule adaptor activity), the TAS annotation with qualifier enables from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031113 regulation of microtubule polymerization
TAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: regulation of microtubule polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031113 (regulation of microtubule polymerization), the TAS annotation with qualifier involved_in from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0033044 regulation of chromosome organization
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: regulation of chromosome organization is marked over-annotated for MAPT because this biological-process term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0033044 (regulation of chromosome organization), the TAS annotation with qualifier involved_in from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0034063 stress granule assembly
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: stress granule assembly is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0034063 (stress granule assembly), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0034614 cellular response to reactive oxygen species
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: cellular response to reactive oxygen species is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0034614 (cellular response to reactive oxygen species), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0035091 phosphatidylinositol binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: phosphatidylinositol binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0035091 (phosphatidylinositol binding), the TAS annotation with qualifier enables from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0036477 somatodendritic compartment
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: somatodendritic compartment is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0036477 (somatodendritic compartment), the TAS annotation with qualifier located_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0043197 dendritic spine
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: dendritic spine is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0043197 (dendritic spine), the TAS annotation with qualifier located_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0043565 sequence-specific DNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: sequence-specific DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0043565 (sequence-specific DNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0048167 regulation of synaptic plasticity
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: regulation of synaptic plasticity is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0048167 (regulation of synaptic plasticity), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0070507 regulation of microtubule cytoskeleton organization
TAS
PMID:28386764
Roles of tau protein in health and disease.
ACCEPT
Summary: regulation of microtubule cytoskeleton organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0070507 (regulation of microtubule cytoskeleton organization), the TAS annotation with qualifier involved_in from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0099077 histone-dependent DNA binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
MARK AS OVER ANNOTATED
Summary: histone-dependent DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0099077 (histone-dependent DNA binding), the TAS annotation with qualifier enables from PMID:28386764 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:1900452 regulation of long-term synaptic depression
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: regulation of long-term synaptic depression is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1900452 (regulation of long-term synaptic depression), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1902936 phosphatidylinositol bisphosphate binding
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: phosphatidylinositol bisphosphate binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1902936 (phosphatidylinositol bisphosphate binding), the TAS annotation with qualifier enables from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1990090 cellular response to nerve growth factor stimulus
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: cellular response to nerve growth factor stimulus is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1990090 (cellular response to nerve growth factor stimulus), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1990416 cellular response to brain-derived neurotrophic factor stimulus
TAS
PMID:28386764
Roles of tau protein in health and disease.
KEEP AS NON CORE
Summary: cellular response to brain-derived neurotrophic factor stimulus is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1990416 (cellular response to brain-derived neurotrophic factor stimulus), the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0010288 response to lead ion
ISS
GO_REF:0000024
MARK AS OVER ANNOTATED
Summary: response to lead ion is marked over-annotated for MAPT because this biological-process term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0010288 (response to lead ion), the ISS annotation with qualifier involved_in from GO_REF:0000024 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0050848 regulation of calcium-mediated signaling
IDA
PMID:18272392
Extracellular tau promotes intracellular calcium increase th...
KEEP AS NON CORE
Summary: regulation of calcium-mediated signaling is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0050848 (regulation of calcium-mediated signaling), the IDA annotation with qualifier involved_in from PMID:18272392 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005515 protein binding
IPI
PMID:3111527
Comparison of S100b protein with calmodulin: interactions wi...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:3111527 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0031113 regulation of microtubule polymerization
TAS
PMID:10640627
Regulation of phosphorylation of neuronal microtubule-associ...
ACCEPT
Summary: regulation of microtubule polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031113 (regulation of microtubule polymerization), the TAS annotation with qualifier involved_in from PMID:10640627 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0000226 microtubule cytoskeleton organization
NAS
PMID:28377597
Dyrk1A overexpression leads to increase of 3R-tau expression...
ACCEPT
Summary: microtubule cytoskeleton organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0000226 (microtubule cytoskeleton organization), the NAS annotation with qualifier involved_in from PMID:28377597 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0008017 microtubule binding
IDA
PMID:28377597
Dyrk1A overexpression leads to increase of 3R-tau expression...
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the IDA annotation with qualifier enables from PMID:28377597 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0098930 axonal transport
NAS
PMID:28377597
Dyrk1A overexpression leads to increase of 3R-tau expression...
ACCEPT
Summary: axonal transport is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0098930 (axonal transport), the NAS annotation with qualifier involved_in from PMID:28377597 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005874 microtubule
ISS
GO_REF:0000024
ACCEPT
Summary: microtubule is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005874 (microtubule), the ISS annotation with qualifier colocalizes_with from GO_REF:0000024 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0010629 negative regulation of gene expression
IMP
PMID:23164821
Clusterin regulates Ξ²-amyloid toxicity via Dickkopf-1-driven...
MARK AS OVER ANNOTATED
Summary: negative regulation of gene expression is marked over-annotated for MAPT because this biological-process term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0010629 (negative regulation of gene expression), the IMP annotation with qualifier involved_in from PMID:23164821 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:20655099
Dendritic function of tau mediates amyloid-beta toxicity in ...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:20655099 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
IMP
PMID:20655099
Dendritic function of tau mediates amyloid-beta toxicity in ...
KEEP AS NON CORE
Summary: memory is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0007613 (memory), the IMP annotation with qualifier involved_in from PMID:20655099 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0016020 membrane
IMP
PMID:20655099
Dendritic function of tau mediates amyloid-beta toxicity in ...
KEEP AS NON CORE
Summary: membrane is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0016020 (membrane), the IMP annotation with qualifier colocalizes_with from PMID:20655099 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0036477 somatodendritic compartment
IMP
PMID:20655099
Dendritic function of tau mediates amyloid-beta toxicity in ...
KEEP AS NON CORE
Summary: somatodendritic compartment is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0036477 (somatodendritic compartment), the IMP annotation with qualifier located_in from PMID:20655099 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0043025 neuronal cell body
IMP
PMID:20655099
Dendritic function of tau mediates amyloid-beta toxicity in ...
KEEP AS NON CORE
Summary: neuronal cell body is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0043025 (neuronal cell body), the IMP annotation with qualifier located_in from PMID:20655099 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0050808 synapse organization
IMP
PMID:17270732
Synapse loss and microglial activation precede tangles in a ...
KEEP AS NON CORE
Summary: synapse organization is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0050808 (synapse organization), the IMP annotation with qualifier involved_in from PMID:17270732 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1902474 positive regulation of protein localization to synapse
IMP
PMID:20655099
Dendritic function of tau mediates amyloid-beta toxicity in ...
KEEP AS NON CORE
Summary: positive regulation of protein localization to synapse is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1902474 (positive regulation of protein localization to synapse), the IMP annotation with qualifier involved_in from PMID:20655099 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0003677 DNA binding
ISS
GO_REF:0000024
MARK AS OVER ANNOTATED
Summary: DNA binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003677 (DNA binding), the ISS annotation with qualifier enables from GO_REF:0000024 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005634 nucleus
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: nucleus is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005634 (nucleus), the ISS annotation with qualifier located_in from GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005829 cytosol
ISS
GO_REF:0000024
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the ISS annotation with qualifier located_in from GO_REF:0000024 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0006974 DNA damage response
IMP
PMID:21131359
Nuclear tau, a key player in neuronal DNA protection.
KEEP AS NON CORE
Summary: DNA damage response is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0006974 (DNA damage response), the IMP annotation with qualifier involved_in from PMID:21131359 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0010917 negative regulation of mitochondrial membrane potential
IMP
PMID:22920254
Tau promotes neurodegeneration via DRP1 mislocalization in v...
KEEP AS NON CORE
Summary: negative regulation of mitochondrial membrane potential is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0010917 (negative regulation of mitochondrial membrane potential), the IMP annotation with qualifier involved_in from PMID:22920254 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0090140 regulation of mitochondrial fission
IC
PMID:22367970
Abnormal interaction between the mitochondrial fission prote...
KEEP AS NON CORE
Summary: regulation of mitochondrial fission is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0090140 (regulation of mitochondrial fission), the IC annotation with qualifier involved_in from PMID:22367970 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1900034 regulation of cellular response to heat
IMP
PMID:21131359
Nuclear tau, a key player in neuronal DNA protection.
KEEP AS NON CORE
Summary: regulation of cellular response to heat is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1900034 (regulation of cellular response to heat), the IMP annotation with qualifier involved_in from PMID:21131359 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:1903748 negative regulation of protein localization to mitochondrion
IMP
PMID:22920254
Tau promotes neurodegeneration via DRP1 mislocalization in v...
KEEP AS NON CORE
Summary: negative regulation of protein localization to mitochondrion is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1903748 (negative regulation of protein localization to mitochondrion), the IMP annotation with qualifier involved_in from PMID:22920254 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0048312 intracellular distribution of mitochondria
IMP
PMID:21854751
Tau accumulation causes mitochondrial distribution deficits ...
KEEP AS NON CORE
Summary: intracellular distribution of mitochondria is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0048312 (intracellular distribution of mitochondria), the IMP annotation with qualifier involved_in from PMID:21854751 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0001774 microglial cell activation
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: microglial cell activation is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0001774 (microglial cell activation), the TAS annotation with qualifier involved_in from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0003779 actin binding
TAS
PMID:27306859
Tau physiology and pathomechanisms in frontotemporal lobar d...
KEEP AS NON CORE
Summary: actin binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0003779 (actin binding), the TAS annotation with qualifier enables from PMID:27306859 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0008017 microtubule binding
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the TAS annotation with qualifier enables from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0019896 axonal transport of mitochondrion
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: axonal transport of mitochondrion is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019896 (axonal transport of mitochondrion), the TAS annotation with qualifier involved_in from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
NAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: axon is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030424 (axon), the NAS annotation with qualifier located_in from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031122 cytoplasmic microtubule organization
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: cytoplasmic microtubule organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031122 (cytoplasmic microtubule organization), the TAS annotation with qualifier involved_in from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031175 neuron projection development
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: neuron projection development is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031175 (neuron projection development), the TAS annotation with qualifier involved_in from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0034452 dynactin binding
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: dynactin binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0034452 (dynactin binding), the TAS annotation with qualifier enables from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0034605 cellular response to heat
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: cellular response to heat is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0034605 (cellular response to heat), the TAS annotation with qualifier involved_in from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0048143 astrocyte activation
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: astrocyte activation is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0048143 (astrocyte activation), the TAS annotation with qualifier involved_in from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0051721 protein phosphatase 2A binding
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: protein phosphatase 2A binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0051721 (protein phosphatase 2A binding), the TAS annotation with qualifier enables from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0072386 plus-end-directed organelle transport along microtubule
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
KEEP AS NON CORE
Summary: plus-end-directed organelle transport along microtubule is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0072386 (plus-end-directed organelle transport along microtubule), the TAS annotation with qualifier involved_in from PMID:26363795 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0098930 axonal transport
TAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: axonal transport is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0098930 (axonal transport), the TAS annotation with qualifier involved_in from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:1902988 neurofibrillary tangle assembly
NAS
PMID:26363795
The Role of MAPT in Neurodegenerative Diseases: Genetics, Me...
ACCEPT
Summary: neurofibrillary tangle assembly is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1902988 (neurofibrillary tangle assembly), the NAS annotation with qualifier involved_in from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005737 cytoplasm
IDA
PMID:27356871
Acetylated tau destabilizes the cytoskeleton in the axon ini...
KEEP AS NON CORE
Summary: cytoplasm is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005737 (cytoplasm), the IDA annotation with qualifier located_in from PMID:27356871 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0015630 microtubule cytoskeleton
IDA
PMID:27356871
Acetylated tau destabilizes the cytoskeleton in the axon ini...
ACCEPT
Summary: microtubule cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015630 (microtubule cytoskeleton), the IDA annotation with qualifier located_in from PMID:27356871 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031110 regulation of microtubule polymerization or depolymerization
IMP
PMID:27356871
Acetylated tau destabilizes the cytoskeleton in the axon ini...
ACCEPT
Summary: regulation of microtubule polymerization or depolymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031110 (regulation of microtubule polymerization or depolymerization), the IMP annotation with qualifier involved_in from PMID:27356871 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:1903829 positive regulation of protein localization
IMP
PMID:27356871
Acetylated tau destabilizes the cytoskeleton in the axon ini...
MARK AS OVER ANNOTATED
Summary: positive regulation of protein localization is marked over-annotated for MAPT because this biological-process term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1903829 (positive regulation of protein localization), the IMP annotation with qualifier involved_in from PMID:27356871 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005829 cytosol
IDA
PMID:10747907
Interaction of tau with the neural membrane cortex is regula...
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the IDA annotation with qualifier located_in from PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005886 plasma membrane
IDA
PMID:10747907
Interaction of tau with the neural membrane cortex is regula...
ACCEPT
Summary: plasma membrane is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005886 (plasma membrane), the IDA annotation with qualifier located_in from PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0008017 microtubule binding
IMP
PMID:10747907
Interaction of tau with the neural membrane cortex is regula...
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the IMP annotation with qualifier enables from PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0015630 microtubule cytoskeleton
IDA
PMID:15147841
Surface-decoration of microtubules by human tau.
ACCEPT
Summary: microtubule cytoskeleton is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0015630 (microtubule cytoskeleton), the IDA annotation with qualifier located_in from PMID:15147841 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0030673 axolemma
IDA
PMID:10747907
Interaction of tau with the neural membrane cortex is regula...
ACCEPT
Summary: axolemma is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030673 (axolemma), the IDA annotation with qualifier located_in from PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0042802 identical protein binding
IMP
PMID:14769047
Tau paired helical filaments from Alzheimer's disease brain ...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IMP annotation with qualifier enables from PMID:14769047 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0042802 identical protein binding
IMP
PMID:7929085
Structural studies of tau protein and Alzheimer paired helic...
ACCEPT
Summary: identical protein binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0042802 (identical protein binding), the IMP annotation with qualifier enables from PMID:7929085 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0070507 regulation of microtubule cytoskeleton organization
IMP
PMID:15147841
Surface-decoration of microtubules by human tau.
ACCEPT
Summary: regulation of microtubule cytoskeleton organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0070507 (regulation of microtubule cytoskeleton organization), the IMP annotation with qualifier involved_in from PMID:15147841 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0097418 neurofibrillary tangle
IDA
PMID:2484340
Multiple isoforms of human microtubule-associated protein ta...
ACCEPT
Summary: neurofibrillary tangle is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0097418 (neurofibrillary tangle), the IDA annotation with qualifier located_in from PMID:2484340 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0097435 supramolecular fiber organization
IMP
PMID:14769047
Tau paired helical filaments from Alzheimer's disease brain ...
ACCEPT
Summary: supramolecular fiber organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0097435 (supramolecular fiber organization), the IMP annotation with qualifier involved_in from PMID:14769047 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0099609 microtubule lateral binding
IMP
PMID:15147841
Surface-decoration of microtubules by human tau.
ACCEPT
Summary: microtubule lateral binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0099609 (microtubule lateral binding), the IMP annotation with qualifier enables from PMID:15147841 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:1990000 amyloid fibril formation
IMP
PMID:7929085
Structural studies of tau protein and Alzheimer paired helic...
ACCEPT
Summary: amyloid fibril formation is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:1990000 (amyloid fibril formation), the IMP annotation with qualifier involved_in from PMID:7929085 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0030425 dendrite
IDA
PMID:25620700
A genome-wide gene-expression analysis and database in trans...
KEEP AS NON CORE
Summary: dendrite is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030425 (dendrite), the IDA annotation with qualifier located_in from PMID:25620700 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0044297 cell body
IDA
PMID:25620700
A genome-wide gene-expression analysis and database in trans...
KEEP AS NON CORE
Summary: cell body is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0044297 (cell body), the IDA annotation with qualifier located_in from PMID:25620700 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0097418 neurofibrillary tangle
NAS
PMID:25620700
A genome-wide gene-expression analysis and database in trans...
ACCEPT
Summary: neurofibrillary tangle is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0097418 (neurofibrillary tangle), the NAS annotation with qualifier located_in from PMID:25620700 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0036464 cytoplasmic ribonucleoprotein granule
IDA
PMID:15121898
The composition of Staufen-containing RNA granules from huma...
KEEP AS NON CORE
Summary: cytoplasmic ribonucleoprotein granule is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0036464 (cytoplasmic ribonucleoprotein granule), the IDA annotation with qualifier located_in from PMID:15121898 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0005515 protein binding
IPI
PMID:23589030
Clathrin adaptor CALM/PICALM is associated with neurofibrill...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:23589030 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:23399914
Increased expression of BIN1 mediates Alzheimer genetic risk...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:23399914 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0031116 positive regulation of microtubule polymerization
IDA
PMID:7706316
Microtubule-associated protein/microtubule affinity-regulati...
ACCEPT
Summary: positive regulation of microtubule polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031116 (positive regulation of microtubule polymerization), the IDA annotation with qualifier involved_in from PMID:7706316 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005829 cytosol
TAS
Reactome:R-HSA-201629
ACCEPT
Summary: cytosol is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from Reactome:R-HSA-201629 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0034185 apolipoprotein binding
IPI
PMID:7566652
ApoE3 binding to tau tandem repeat I is abolished by tau ser...
KEEP AS NON CORE
Summary: apolipoprotein binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0034185 (apolipoprotein binding), the IPI annotation with qualifier enables from PMID:7566652 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0010506 regulation of autophagy
IGI
PMID:19074461
Tau deletion exacerbates the phenotype of Niemann-Pick type ...
KEEP AS NON CORE
Summary: regulation of autophagy is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0010506 (regulation of autophagy), the IGI annotation with qualifier acts_upstream_of_or_within from PMID:19074461 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0071813 lipoprotein particle binding
IPI
PMID:7972031
Isoform-specific interactions of apolipoprotein E with micro...
KEEP AS NON CORE
Summary: lipoprotein particle binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0071813 (lipoprotein particle binding), the IPI annotation with qualifier enables from PMID:7972031 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0034399 nuclear periphery
IDA
PMID:19157893
Fluorescence microscopy and 3D image reconstruction of cytok...
KEEP AS NON CORE
Summary: nuclear periphery is retained as a non-core cellular-component/localization annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0034399 (nuclear periphery), the IDA annotation with qualifier located_in from PMID:19157893 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0019899 enzyme binding
IPI
PMID:20133804
A role for FKBP52 in Tau protein function.
MARK AS OVER ANNOTATED
Summary: enzyme binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019899 (enzyme binding), the IPI annotation with qualifier enables from PMID:20133804 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:10987820
Tau promoter confers neuronal specificity and binds Sp1 and ...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:10987820 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005515 protein binding
IPI
PMID:9689133
Presenilin 1 associates with glycogen synthase kinase-3beta ...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:9689133 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0005886 plasma membrane
IDA
PMID:8522593
Interaction of tau with the neural plasma membrane mediated ...
ACCEPT
Summary: plasma membrane is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005886 (plasma membrane), the IDA annotation with qualifier located_in from PMID:8522593 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0008017 microtubule binding
IDA
PMID:1918161
Tau protein binds to microtubules through a flexible array o...
ACCEPT
Summary: microtubule binding is retained as a core molecular-function annotation for MAPT; it captures activity or binding specificity within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0008017 (microtubule binding), the IDA annotation with qualifier enables from PMID:1918161 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0017124 SH3 domain binding
IPI
PMID:9763511
Tau interacts with src-family non-receptor tyrosine kinases.
KEEP AS NON CORE
Summary: SH3 domain binding is retained as a non-core molecular-function annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0017124 (SH3 domain binding), the IPI annotation with qualifier enables from PMID:9763511 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0019899 enzyme binding
IPI
PMID:12888622
Caspase cleavage of tau: linking amyloid and neurofibrillary...
MARK AS OVER ANNOTATED
Summary: enzyme binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019899 (enzyme binding), the IPI annotation with qualifier enables from PMID:12888622 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0019899 enzyme binding
IPI
PMID:9736630
Tau cleavage and dephosphorylation in cerebellar granule neu...
MARK AS OVER ANNOTATED
Summary: enzyme binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0019899 (enzyme binding), the IPI annotation with qualifier enables from PMID:9736630 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.
GO:0048699 generation of neurons
NAS
PMID:8522593
Interaction of tau with the neural plasma membrane mediated ...
KEEP AS NON CORE
Summary: generation of neurons is retained as a non-core biological-process annotation for MAPT; it records a supported context, interaction, localization, or pathway branch that is secondary to tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0048699 (generation of neurons), the NAS annotation with qualifier involved_in from PMID:8522593 supports retaining the annotation, but the term describes a context-specific outcome or peripheral branch rather than the principal MAPT function: tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. Keeping it as non-core prevents broad pathway participation from being promoted to core function.
GO:0000226 microtubule cytoskeleton organization
IDA
PMID:1057175
A protein factor essential for microtubule assembly.
ACCEPT
Summary: microtubule cytoskeleton organization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0000226 (microtubule cytoskeleton organization), the IDA annotation with qualifier involved_in from PMID:1057175 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
IDA
PMID:8642405
Tau is enriched on dynamic microtubules in the distal region...
ACCEPT
Summary: axon is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030424 (axon), the IDA annotation with qualifier located_in from PMID:8642405 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0030426 growth cone
IDA
PMID:8642405
Tau is enriched on dynamic microtubules in the distal region...
ACCEPT
Summary: growth cone is retained as a core cellular-component/localization annotation for MAPT; it captures site of action or component context within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0030426 (growth cone), the IDA annotation with qualifier located_in from PMID:8642405 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031113 regulation of microtubule polymerization
NAS
PMID:1057175
A protein factor essential for microtubule assembly.
ACCEPT
Summary: regulation of microtubule polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031113 (regulation of microtubule polymerization), the NAS annotation with qualifier involved_in from PMID:1057175 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0031116 positive regulation of microtubule polymerization
IDA
PMID:1421571
Modulation of the dynamic instability of tubulin assembly by...
ACCEPT
Summary: positive regulation of microtubule polymerization is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0031116 (positive regulation of microtubule polymerization), the IDA annotation with qualifier involved_in from PMID:1421571 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0045298 tubulin complex
IDA
PMID:8642405
Tau is enriched on dynamic microtubules in the distal region...
MODIFY
Summary: tubulin complex is directionally related to MAPT biology but is not the best curation target; tubulin binding better captures the specific supported function or process.
Reason: For GO:0045298 (tubulin complex), the IDA annotation with qualifier part_of from PMID:8642405 supports a relationship to MAPT, but the current term is less precise than tubulin binding for the evidence and for the synthesized core/non-core role of MAPT. The MODIFY action preserves the biological intent while pointing curators to the more informative GO term.
Proposed replacements: tubulin binding
GO:0045773 positive regulation of axon extension
IDA
PMID:1389180
Suppression of MAP2 in cultured cerebellar macroneurons inhi...
ACCEPT
Summary: positive regulation of axon extension is retained as a core biological-process annotation for MAPT; it captures process participation within the synthesized core biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0045773 (positive regulation of axon extension), the IDA annotation with qualifier involved_in from PMID:1389180 is consistent with MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. This action keeps the term because it provides a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation annotation rather than only a downstream phenotype or generic interaction label.
GO:0005515 protein binding
IPI
PMID:15953362
Sequestosome 1/p62 shuttles polyubiquitinated tau for protea...
MARK AS OVER ANNOTATED
Summary: protein binding is marked over-annotated for MAPT because this molecular-function term is too generic, interaction-map-like, or weakly informative relative to the gene-specific biology: tau microtubule binding, microtubule assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent regulation of tau behavior.
Reason: For GO:0005515 (protein binding), the IPI annotation with qualifier enables from PMID:15953362 may reflect a real assay result or interaction, but this GO term does not identify the specific microtubule, tubulin-binding, localization, or tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule assembly and stability, axonal localization, and tau phosphorylation-state biology. More informative gene-specific annotations are present, so this is marked over-annotated rather than accepted as a core function.

Core Functions

Neuronal microtubule-associated protein activity: tau binds tubulin and microtubules, stabilizes microtubules, promotes microtubule assembly, supports axonal microtubule organization, axonal transport, neurite/axon development, and links axonal microtubules to neural membrane-cortex components.

Supporting Evidence:
  • file:human/MAPT/MAPT-uniprot.txt
    Promotes microtubule assembly and stability
  • file:human/MAPT/MAPT-uniprot.txt
    The C-terminus binds axonal microtubules
  • file:human/MAPT/MAPT-uniprot.txt
    N-terminus binds neural plasma membrane components
  • PMID:15147841
    Tau is a neuronal, microtubule-associated protein
  • PMID:15147841
    bind exclusively to the outside of microtubules

Disease-relevant tau self-association and pathological filament/tangle assembly, in which modified tau proteins polymerize into supramolecular fibers, amyloid-like paired helical filaments, and Alzheimer neurofibrillary tangles.

Supporting Evidence:

References

Gene Ontology annotation through association of InterPro records with GO terms
Manual transfer of experimentally-verified manual GO annotation data to orthologs by curator judgment of sequence similarity
Annotation inferences using phylogenetic trees
Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular Location vocabulary mapping, accompanied by conservative changes to GO terms applied by UniProt
Gene Ontology annotation based on curation of immunofluorescence data
Automatic assignment of GO terms using logical inference, based on on inter-ontology links
A protein factor essential for microtubule assembly.
Regulation of phosphorylation of neuronal microtubule-associated proteins MAP1b and MAP2 by protein phosphatase-2A and -2B in rat brain.
Interaction of tau with the neural membrane cortex is regulated by phosphorylation at sites that are modified in paired helical filaments.
Tau promoter confers neuronal specificity and binds Sp1 and AP-2.
Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in Alzheimer's disease.
Suppression of MAP2 in cultured cerebellar macroneurons inhibits minor neurite formation.
Modulation of the dynamic instability of tubulin assembly by the microtubule-associated protein tau.
Tau paired helical filaments from Alzheimer's disease brain and assembled in vitro are based on beta-structure in the core domain.
The composition of Staufen-containing RNA granules from human cells indicates their role in the regulated transport and translation of messenger RNAs.
Surface-decoration of microtubules by human tau.
Residual structure in the repeat domain of tau: echoes of microtubule binding and paired helical filament formation.
Sequestosome 1/p62 shuttles polyubiquitinated tau for proteasomal degradation.
Identification of VCP/p97, carboxyl terminus of Hsp70-interacting protein (CHIP), and amphiphysin II interaction partners using membrane-based human proteome arrays.
Abeta and tau form soluble complexes that may promote self aggregation of both into the insoluble forms observed in Alzheimer's disease.
Tau-tubulin kinase 1 (TTBK1), a neuron-specific tau kinase candidate, is involved in tau phosphorylation and aggregation.
Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model.
14-3-3zeta facilitates GSK3beta-catalyzed tau phosphorylation in HEK-293 cells by a mechanism that requires phosphorylation of GSK3beta on Ser9.
Roles of heat-shock protein 90 in maintaining and facilitating the neurodegenerative phenotype in tauopathies.
DYRK1A-mediated hyperphosphorylation of Tau. A functional link between Down syndrome and Alzheimer disease.
Extracellular tau promotes intracellular calcium increase through M1 and M3 muscarinic receptors in neuronal cells.
Akt and CHIP coregulate tau degradation through coordinated interactions.
Tau deletion exacerbates the phenotype of Niemann-Pick type C mice and implicates autophagy in pathogenesis.
Effect of phosphorylation on interaction of human tau protein with 14-3-3zeta.
Fluorescence microscopy and 3D image reconstruction of cytokine initiated disruption of the Parkinson disease associated proteins alpha-synuclein, tau and ubiquitin in cultured glial cells.
Tau protein binds to microtubules through a flexible array of distributed weak sites.
Tau--an inhibitor of deacetylase HDAC6 function.
Excitatory amino acid transporter 2 associates with phosphorylated tau and is localized in neurofibrillary tangles of tauopathic brains.
Phosphorylation of more than one site is required for tight interaction of human tau protein with 14-3-3zeta.
A role for FKBP52 in Tau protein function.
Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models.
Nuclear tau, a key player in neuronal DNA protection.
Tyrosine phosphorylation of tau regulates its interactions with Fyn SH2 domains, but not SH3 domains, altering the cellular localization of tau.
Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brain.
Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage.
Tau promotes neurodegeneration via DRP1 mislocalization in vivo.
Clusterin regulates Ξ²-amyloid toxicity via Dickkopf-1-driven induction of the wnt-PCP-JNK pathway.
Isomerase Pin1 stimulates dephosphorylation of tau protein at cyclin-dependent kinase (Cdk5)-dependent Alzheimer phosphorylation sites.
Increased expression of BIN1 mediates Alzheimer genetic risk by modulating tau pathology.
Clathrin adaptor CALM/PICALM is associated with neurofibrillary tangles and is cleaved in Alzheimer's brains.
Role of individual MARK isoforms in phosphorylation of tau at Ser²⁢² in Alzheimer's disease.
Impact of the cellular prion protein on amyloid-Ξ² and 3PO-tau processing.
Phosphorylation of human Tau protein by microtubule affinity-regulating kinase 2.
Multiple isoforms of human microtubule-associated protein tau: sequences and localization in neurofibrillary tangles of Alzheimer's disease.
Intracellular clusterin interacts with brain isoforms of the bridging integrator 1 and with the microtubule-associated protein Tau in Alzheimer's disease.
AlphaScreen HTS and live-cell bioluminescence resonance energy transfer (BRET) assays for identification of Tau-Fyn SH3 interaction inhibitors for Alzheimer disease.
A novel DYRK1A (dual specificity tyrosine phosphorylation-regulated kinase 1A) inhibitor for the treatment of Alzheimer's disease: effect on Tau and amyloid pathologies in vitro.
A genome-wide gene-expression analysis and database in transgenic mice during development of amyloid or tau pathology.
LRRK2 Promotes Tau Accumulation, Aggregation and Release.
The Role of MAPT in Neurodegenerative Diseases: Genetics, Mechanisms and Therapy.
Identification of Novel Tau Interactions with Endoplasmic Reticulum Proteins in Alzheimer's Disease Brain.
Defects in Synaptic Plasticity, Reduced NMDA-Receptor Transport, and Instability of Postsynaptic Density Proteins in Mice Lacking Microtubule-Associated Protein 1A.
Regulation of Microtubule Assembly by Tau and not by Pin1.
FRMD4A-cytohesin signaling modulates the cellular release of tau.
Tau physiology and pathomechanisms in frontotemporal lobar degeneration.
Acetylated tau destabilizes the cytoskeleton in the axon initial segment and is mislocalized to the somatodendritic compartment.
Dyrk1A overexpression leads to increase of 3R-tau expression and cognitive deficits in Ts65Dn Down syndrome mice.
Roles of tau protein in health and disease.
Somatostatin binds to the human amyloid Ξ² peptide and favors the formation of distinct oligomers.
Regulation of the interaction between the neuronal BIN1 isoform 1 and Tau proteins - role of the SH3 domain.
The disorderly conduct of Hsc70 and its interaction with the Alzheimer's-related Tau protein.
Structures of filaments from Pick's disease reveal a novel tau protein fold.
Role of tau N-terminal motif in the secretion of human tau by End Binding proteins.
Novel tau filament fold in chronic traumatic encephalopathy encloses hydrophobic molecules.
Comparison of S100b protein with calmodulin: interactions with melittin and microtubule-associated tau proteins and inhibition of phosphorylation of tau proteins by protein kinase C.
GSKIP-Mediated Anchoring Increases Phosphorylation of Tau by PKA but Not by GSK3beta via cAMP/PKA/GSKIP/GSK3/Tau Axis Signaling in Cerebrospinal Fluid and iPS Cells in Alzheimer Disease.
A Translocation Pathway for Vesicle-Mediated Unconventional Protein Secretion.
Interactome Mapping Provides a Network of Neurodegenerative Disease Proteins and Uncovers Widespread Protein Aggregation in Affected Brains.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
Targeting Tau Mitigates Mitochondrial Fragmentation and Oxidative Stress in Amyotrophic Lateral Sclerosis.
ApoE3 binding to tau tandem repeat I is abolished by tau serine262 phosphorylation.
Microtubule-associated protein/microtubule affinity-regulating kinase (p110mark). A novel protein kinase that regulates tau-microtubule interactions and dynamic instability by phosphorylation at the Alzheimer-specific site serine 262.
Structural studies of tau protein and Alzheimer paired helical filaments show no evidence for beta-structure.
Isoform-specific interactions of apolipoprotein E with microtubule-associated protein tau: implications for Alzheimer disease.
Interaction of tau with the neural plasma membrane mediated by tau's amino-terminal projection domain.
Tau is enriched on dynamic microtubules in the distal region of growing axons.
Presenilin 1 associates with glycogen synthase kinase-3beta and its substrate tau.
Tau cleavage and dephosphorylation in cerebellar granule neurons undergoing apoptosis.
Tau interacts with src-family non-receptor tyrosine kinases.
Reactome:R-HSA-201629
Caspase-mediated cleavage of Tau
Reactome:R-HSA-9836404
p-PKR dimer phosphorylates MAPT
Reactome:R-NUL-9619534
AMPK phosphorylates Mapt
Reactome:R-NUL-9619539
AMPK-negatively regulates binding of MAPT to microtubules
file:human/MAPT/MAPT-uniprot.txt
UniProt text export for MAPT (P10636)
file:human/MAPT/MAPT-notes.md
Manual MAPT review notes

Suggested Questions for Experts

Q: Which tau annotations should be treated as normal neuronal microtubule/axon biology versus pathological tauopathy gain-of-function or mislocalization?

Q: Which kinase-, phosphatase-, chaperone-, lipid-, and APOE-interaction annotations are reproducible endogenous tau functions rather than context-specific or high-throughput interactions?

Q: For Alzheimer-relevant pathway modules, should extracellular tau secretion/spread, synaptic toxicity, mitochondrial dysfunction, and glial activation be curated as MAPT core functions or as downstream disease-context consequences?

Suggested Experiments

Experiment: Use endogenous MAPT isoform and phosphosite editing in human iPSC-derived neurons, followed by live imaging of microtubule dynamics, axonal transport, membrane-cortex association, and somatodendritic tau accumulation under amyloid and kinase-activation stressors.

Hypothesis: Tau phosphorylation state determines the balance between axonal microtubule stabilization, neural membrane-cortex association, and somatodendritic mislocalization.

Type: endogenous phosphosite/isoform perturbation assay

Experiment: Compare wild-type tau with aggregation-defective and microtubule-binding-defective MAPT variants in neuronal cultures, measuring tau filament formation, neurofibrillary-tangle markers, synapse localization/function, mitochondrial transport, and neuron viability.

Hypothesis: Pathological tau self-assembly is separable from normal microtubule-stabilizing activity and drives downstream synaptic and mitochondrial phenotypes.

Type: structure-function tauopathy model

πŸ“š Additional Documentation

Notes

(MAPT-notes.md)

MAPT notes

Automated deep research was attempted with just deep-research-falcon human MAPT --fallback perplexity-lite, but the run timed out before producing a deep-research file. This review therefore uses cached GOA publications, the UniProt record, and the PANTHER family fetch.

MAPT encodes microtubule-associated protein tau. The UniProt record summarizes the core tau function as microtubule assembly/stability and neuronal polarity, and notes the microtubule/plasma-membrane linker model: "Promotes microtubule assembly and stability", "involved in the establishment and maintenance of neuronal polarity", "The C-terminus binds axonal microtubules", and "N-terminus binds neural plasma membrane components" [file:human/MAPT/MAPT-uniprot.txt "Promotes microtubule assembly and stability"; file:human/MAPT/MAPT-uniprot.txt "involved in the establishment and maintenance of neuronal polarity"; file:human/MAPT/MAPT-uniprot.txt "The C-terminus binds axonal microtubules"; file:human/MAPT/MAPT-uniprot.txt "N-terminus binds neural plasma membrane components"]. PANTHER places MAPT in PTHR11501:SF14, microtubule-associated protein tau.

Primary microtubule-binding evidence supports the central GO annotations. Santarella et al. describe tau as a neuronal microtubule-associated protein that stabilizes microtubules and promotes neurite outgrowth; their structural study reports that tau constructs "bind exclusively to the outside of microtubules" and that "even minute concentrations of tau have a strong" stabilizing effect [PMID:15147841 "Tau is a neuronal, microtubule-associated protein"; PMID:15147841 "stabilizes microtubules"; PMID:15147841 "promotes neurite outgrowth"; PMID:15147841 "bind exclusively to the outside of microtubules"; PMID:15147841 "even minute concentrations of tau have a strong"]. Phosphorylation of tau in the repeat domain can detach tau from microtubules: Drewes et al. report that phosphorylation "largely abolishes tau's binding to microtubules" and makes microtubules "dynamically unstable" [PMID:7706316 "largely abolishes tau's binding to microtubules"; PMID:7706316 "dynamically unstable"].

Tau also has a supported axonal/membrane association. The neural membrane cortex paper describes "The axonal microtubule-associated phosphoprotein tau", reports that tau membrane association "requires an intact membrane cortex", and links endogenous phosphoisoforms to "enrichment in the axonal compartment" [PMID:10747907 "The axonal microtubule-associated phosphoprotein tau"; PMID:10747907 "requires an intact membrane cortex"; PMID:10747907 "enrichment in the axonal compartment"]. This supports axon, cytosol/cytoplasm, cytoskeleton, plasma membrane/axolemma, and somatodendritic/dendrite annotations, but membrane and dendritic terms should not eclipse the microtubule/axon core.

Tau pathological self-assembly is disease-relevant and should be represented separately from physiological microtubule stabilization. Goedert et al. report that "multiple tau protein isoforms are incorporated into" the "neurofibrillary tangles of Alzheimer's disease" [PMID:2484340 "multiple tau protein isoforms are incorporated into"; PMID:2484340 "neurofibrillary tangles of Alzheimer's disease"]. A MAPT neurodegeneration review states that MAPT is "tightly implicated in keeping the function of microtubules" and "axonal transport" and that hyperphosphorylated tau participates in "neurofibrillary tangles (NFTs)" [PMID:26363795 "tightly implicated in keeping the function of microtubules"; PMID:26363795 "axonal transport"; PMID:26363795 "neurofibrillary tangles (NFTs)"]. The broader tau-function review similarly states that "Tau is well established as a microtubule-associated protein in neurons" and that pathological tau assembly is accompanied by "synaptic dysfunction and neural cell death" [PMID:28386764 "Tau is well established as a microtubule-associated protein in neurons"; PMID:28386764 "synaptic dysfunction and neural cell death"].

For curation, accept the core microtubule binding/stabilizing, microtubule organization/polymerization, axonal localization, and tau self-assembly/tangle annotations. Keep synaptic, mitochondrial, extracellular secretion, nucleic-acid/nuclear, stress-response, glial activation, and disease-model phenotypes as non-core when evidence is credible but downstream or context-specific. Mark generic protein binding and very broad or poorly informative review-derived labels as over-annotated, especially when more informative partner-specific or microtubule-specific terms are already present.

2026-06-20 second-pass audit

The second-pass audit confirmed the existing MAPT review and manual reference metadata. No annotation action changes were needed: MAPT remains curated around tau microtubule binding, microtubule stabilization/polymerization, axonal localization, and pathological tau self-assembly, with synaptic, mitochondrial, extracellular, nuclear/nucleic-acid, stress-response, glial, and disease-model phenotypes kept non-core or over-annotated according to the first-pass rationale.

πŸ“„ View Raw YAML

id: P10636
gene_symbol: MAPT
product_type: PROTEIN
status: COMPLETE
taxon:
  id: NCBITaxon:9606
  label: Homo sapiens
description: 'MAPT encodes microtubule-associated protein tau, a neuronally enriched,
  intrinsically disordered microtubule-associated protein. Tau binds tubulin and microtubules
  through repeat domains, promotes microtubule assembly and stability, and contributes
  to axonal microtubule organization, axonal transport, neurite/axon development,
  and neuronal polarity. Tau also associates with neural plasma-membrane and axonal
  cortex components, and disease-associated phosphorylation or acetylation can alter
  microtubule and membrane binding. In Alzheimer disease and other tauopathies, modified
  tau mislocalizes and self-assembles into paired-helical-filament/neurofibrillary-tangle
  pathology with downstream synaptic, mitochondrial, glial, secretion, and stress-response
  consequences.'
alternative_products:
- name: PNS-tau
  id: P10636-1
- name: Fetal-tau (0N3R {ECO:0000303|PubMed:9789048})
  id: P10636-2
  sequence_note: VSP_003176, VSP_003177, VSP_003179,
- name: Tau-A
  id: P10636-3
  sequence_note: VSP_003175, VSP_003176, VSP_003177,
- name: Tau-B (1N3R {ECO:0000303|PubMed:9789048})
  id: P10636-4
  sequence_note: VSP_003177, VSP_003179, VSP_003180,
- name: Tau-C (Tau-3, 2N3R {ECO:0000303|PubMed:9789048})
  id: P10636-5
  sequence_note: VSP_003179, VSP_003180, VSP_003181
- name: Tau-D (0N4R {ECO:0000303|PubMed:9789048})
  id: P10636-6
  sequence_note: VSP_003176, VSP_003177, VSP_003179,
- name: Tau-E (1N4R {ECO:0000303|PubMed:9789048})
  id: P10636-7
  sequence_note: VSP_003177, VSP_003179, VSP_003180
- name: Tau-F (Tau-4, 2N4R {ECO:0000303|PubMed:9789048})
  id: P10636-8
  sequence_note: VSP_003179, VSP_003180
- name: Tau-G
  id: P10636-9
  sequence_note: VSP_026780
existing_annotations:
- term:
    id: GO:0000226
    label: microtubule cytoskeleton organization
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: involved_in
  review:
    summary: >-
      microtubule cytoskeleton organization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0000226 (microtubule cytoskeleton organization), the IBA annotation with
      qualifier involved_in from GO_REF:0000033 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0004857
    label: enzyme inhibitor activity
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: enables
  review:
    summary: >-
      enzyme inhibitor activity is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0004857 (enzyme inhibitor activity), the IBA annotation with qualifier
      enables from GO_REF:0000033 supports retaining the annotation, but the term
      describes a context-specific outcome or peripheral branch rather than the principal
      MAPT function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the IBA annotation with qualifier enables
      from GO_REF:0000033 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015630
    label: microtubule cytoskeleton
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: is_active_in
  review:
    summary: >-
      microtubule cytoskeleton is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015630 (microtubule cytoskeleton), the IBA annotation with qualifier
      is_active_in from GO_REF:0000033 is consistent with MAPT's core role in tau
      binding to tubulin and microtubules, regulation of microtubule assembly and
      stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030424
    label: axon
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: is_active_in
  review:
    summary: >-
      axon is retained as a core cellular-component/localization annotation for MAPT;
      it captures site of action or component context within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030424 (axon), the IBA annotation with qualifier is_active_in from GO_REF:0000033
      is consistent with MAPT's core role in tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. This action keeps the term because it provides
      a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation
      annotation rather than only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0060632
    label: regulation of microtubule-based movement
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule-based movement is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0060632 (regulation of microtubule-based movement), the IBA annotation
      with qualifier involved_in from GO_REF:0000033 is consistent with MAPT's core
      role in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0140778
    label: microtubule stabilizing activity
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: enables
  review:
    summary: >-
      microtubule stabilizing activity is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0140778 (microtubule stabilizing activity), the IBA annotation with qualifier
      enables from GO_REF:0000033 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031175
    label: neuron projection development
  evidence_type: IBA
  original_reference_id: GO_REF:0000033
  qualifier: involved_in
  review:
    summary: >-
      neuron projection development is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031175 (neuron projection development), the IBA annotation with qualifier
      involved_in from GO_REF:0000033 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005576
    label: extracellular region
  evidence_type: IEA
  original_reference_id: GO_REF:0000044
  qualifier: located_in
  review:
    summary: >-
      extracellular region is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005576 (extracellular region), the IEA annotation with qualifier located_in
      from GO_REF:0000044 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: IEA
  original_reference_id: GO_REF:0000044
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the IEA annotation with qualifier located_in from
      GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005856
    label: cytoskeleton
  evidence_type: IEA
  original_reference_id: GO_REF:0000044
  qualifier: located_in
  review:
    summary: >-
      cytoskeleton is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005856 (cytoskeleton), the IEA annotation with qualifier located_in
      from GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005886
    label: plasma membrane
  evidence_type: IEA
  original_reference_id: GO_REF:0000044
  qualifier: located_in
  review:
    summary: >-
      plasma membrane is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005886 (plasma membrane), the IEA annotation with qualifier located_in
      from GO_REF:0000044 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: IEA
  original_reference_id: GO_REF:0000002
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the IEA annotation with qualifier enables
      from GO_REF:0000002 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015631
    label: tubulin binding
  evidence_type: IEA
  original_reference_id: GO_REF:0000002
  qualifier: enables
  review:
    summary: >-
      tubulin binding is retained as a core molecular-function annotation for MAPT;
      it captures activity or binding specificity within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015631 (tubulin binding), the IEA annotation with qualifier enables
      from GO_REF:0000002 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030424
    label: axon
  evidence_type: IEA
  original_reference_id: GO_REF:0000044
  qualifier: located_in
  review:
    summary: >-
      axon is retained as a core cellular-component/localization annotation for MAPT;
      it captures site of action or component context within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030424 (axon), the IEA annotation with qualifier located_in from GO_REF:0000044
      is consistent with MAPT's core role in tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. This action keeps the term because it provides
      a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation
      annotation rather than only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030425
    label: dendrite
  evidence_type: IEA
  original_reference_id: GO_REF:0000044
  qualifier: located_in
  review:
    summary: >-
      dendrite is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0030425 (dendrite), the IEA annotation with qualifier located_in from
      GO_REF:0000044 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1904115
    label: axon cytoplasm
  evidence_type: IEA
  original_reference_id: GO_REF:0000108
  qualifier: located_in
  review:
    summary: >-
      axon cytoplasm is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:1904115 (axon cytoplasm), the IEA annotation with qualifier located_in
      from GO_REF:0000108 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:16275660
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:16275660 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:16446437
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:16446437 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:17317006
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:17317006 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:18292230
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:18292230 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:19138662
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:19138662 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:19527721
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:19527721 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:19647741
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:19647741 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:21692989
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:21692989 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:24028865
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:24028865 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:25156556
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:25156556 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:26402096
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:26402096 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:28650319
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:28650319 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:29764935
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:29764935 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:30668577
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:30668577 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:31640277
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:31640277 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:32814053
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:32814053 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:33961781
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:33961781 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:34757590
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:34757590 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:7566652
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:7566652 may reflect a real assay result or interaction, but this GO
      term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IPI
  original_reference_id: PMID:16446437
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IPI annotation with qualifier
      enables from PMID:16446437 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IPI
  original_reference_id: PMID:30158706
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IPI annotation with qualifier
      enables from PMID:30158706 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IPI
  original_reference_id: PMID:30894745
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IPI annotation with qualifier
      enables from PMID:30894745 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005886
    label: plasma membrane
  evidence_type: IDA
  original_reference_id: GO_REF:0000052
  qualifier: located_in
  review:
    summary: >-
      plasma membrane is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005886 (plasma membrane), the IDA annotation with qualifier located_in
      from GO_REF:0000052 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005576
    label: extracellular region
  evidence_type: EXP
  original_reference_id: PMID:32272059
  qualifier: located_in
  review:
    summary: >-
      extracellular region is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005576 (extracellular region), the EXP annotation with qualifier located_in
      from PMID:32272059 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0004857
    label: enzyme inhibitor activity
  evidence_type: IDA
  original_reference_id: PMID:19457097
  qualifier: enables
  review:
    summary: >-
      enzyme inhibitor activity is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0004857 (enzyme inhibitor activity), the IDA annotation with qualifier
      enables from PMID:19457097 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0060632
    label: regulation of microtubule-based movement
  evidence_type: IGI
  original_reference_id: PMID:19457097
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule-based movement is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0060632 (regulation of microtubule-based movement), the IGI annotation
      with qualifier involved_in from PMID:19457097 is consistent with MAPT's core
      role in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019901
    label: protein kinase binding
  evidence_type: IPI
  original_reference_id: PMID:25556849
  qualifier: enables
  review:
    summary: >-
      protein kinase binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0019901 (protein kinase binding), the IPI annotation with qualifier enables
      from PMID:25556849 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: TAS
  original_reference_id: Reactome:R-HSA-9836404
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from
      Reactome:R-HSA-9836404 is consistent with MAPT's core role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0003779
    label: actin binding
  evidence_type: TAS
  original_reference_id: PMID:22920254
  qualifier: enables
  review:
    summary: >-
      actin binding is retained as a non-core molecular-function annotation for MAPT;
      it records a supported context, interaction, localization, or pathway branch
      that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0003779 (actin binding), the TAS annotation with qualifier enables from
      PMID:22920254 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0032930
    label: positive regulation of superoxide anion generation
  evidence_type: IMP
  original_reference_id: PMID:22920254
  qualifier: involved_in
  review:
    summary: >-
      positive regulation of superoxide anion generation is retained as a non-core
      biological-process annotation for MAPT; it records a supported context, interaction,
      localization, or pathway branch that is secondary to tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0032930 (positive regulation of superoxide anion generation), the IMP
      annotation with qualifier involved_in from PMID:22920254 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0090258
    label: negative regulation of mitochondrial fission
  evidence_type: IMP
  original_reference_id: PMID:22920254
  qualifier: involved_in
  review:
    summary: >-
      negative regulation of mitochondrial fission is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0090258 (negative regulation of mitochondrial fission), the IMP annotation
      with qualifier involved_in from PMID:22920254 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019901
    label: protein kinase binding
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: enables
  review:
    summary: >-
      protein kinase binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0019901 (protein kinase binding), the ISS annotation with qualifier enables
      from GO_REF:0000024 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0051087
    label: protein-folding chaperone binding
  evidence_type: IPI
  original_reference_id: PMID:25051234
  qualifier: enables
  review:
    summary: >-
      protein-folding chaperone binding is retained as a non-core molecular-function
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0051087 (protein-folding chaperone binding), the IPI annotation with
      qualifier enables from PMID:25051234 supports retaining the annotation, but
      the term describes a context-specific outcome or peripheral branch rather than
      the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019899
    label: enzyme binding
  evidence_type: ISS
  original_reference_id: PMID:23362255
  qualifier: enables
  review:
    summary: >-
      enzyme binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0019899 (enzyme binding), the ISS annotation with qualifier enables from
      PMID:23362255 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IPI
  original_reference_id: PMID:27044754
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IPI annotation with qualifier
      enables from PMID:27044754 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IDA
  original_reference_id: PMID:15654759
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IDA annotation with qualifier
      enables from PMID:15654759 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0097435
    label: supramolecular fiber organization
  evidence_type: IDA
  original_reference_id: PMID:15654759
  qualifier: involved_in
  review:
    summary: >-
      supramolecular fiber organization is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0097435 (supramolecular fiber organization), the IDA annotation with
      qualifier involved_in from PMID:15654759 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:22367970
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:22367970 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0019901
    label: protein kinase binding
  evidence_type: IPI
  original_reference_id: PMID:16923168
  qualifier: enables
  review:
    summary: >-
      protein kinase binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0019901 (protein kinase binding), the IPI annotation with qualifier enables
      from PMID:16923168 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005737
    label: cytoplasm
  evidence_type: IMP
  original_reference_id: PMID:27044754
  qualifier: located_in
  review:
    summary: >-
      cytoplasm is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005737 (cytoplasm), the IMP annotation with qualifier located_in from
      PMID:27044754 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0051258
    label: protein polymerization
  evidence_type: IMP
  original_reference_id: PMID:27044754
  qualifier: involved_in
  review:
    summary: >-
      protein polymerization is retained as a core biological-process annotation for
      MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0051258 (protein polymerization), the IMP annotation with qualifier involved_in
      from PMID:27044754 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:26014385
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:26014385 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005737
    label: cytoplasm
  evidence_type: IMP
  original_reference_id: PMID:26014385
  qualifier: located_in
  review:
    summary: >-
      cytoplasm is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005737 (cytoplasm), the IMP annotation with qualifier located_in from
      PMID:26014385 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IMP
  original_reference_id: PMID:26014385
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IMP annotation with qualifier
      enables from PMID:26014385 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: TAS
  original_reference_id: Reactome:R-NUL-9619534
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from
      Reactome:R-NUL-9619534 is consistent with MAPT's core role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: TAS
  original_reference_id: Reactome:R-NUL-9619539
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from
      Reactome:R-NUL-9619539 is consistent with MAPT's core role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:24251416
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:24251416 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0019901
    label: protein kinase binding
  evidence_type: IPI
  original_reference_id: PMID:24251416
  qualifier: enables
  review:
    summary: >-
      protein kinase binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0019901 (protein kinase binding), the IPI annotation with qualifier enables
      from PMID:24251416 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015630
    label: microtubule cytoskeleton
  evidence_type: NAS
  original_reference_id: PMID:26363795
  qualifier: located_in
  review:
    summary: >-
      microtubule cytoskeleton is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015630 (microtubule cytoskeleton), the NAS annotation with qualifier
      located_in from PMID:26363795 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015630
    label: microtubule cytoskeleton
  evidence_type: IDA
  original_reference_id: PMID:28377597
  qualifier: located_in
  review:
    summary: >-
      microtubule cytoskeleton is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015630 (microtubule cytoskeleton), the IDA annotation with qualifier
      located_in from PMID:28377597 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015630
    label: microtubule cytoskeleton
  evidence_type: NAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      microtubule cytoskeleton is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015630 (microtubule cytoskeleton), the NAS annotation with qualifier
      located_in from PMID:28386764 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:23666762
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:23666762 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005737
    label: cytoplasm
  evidence_type: IDA
  original_reference_id: PMID:23666762
  qualifier: located_in
  review:
    summary: >-
      cytoplasm is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005737 (cytoplasm), the IDA annotation with qualifier located_in from
      PMID:23666762 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030425
    label: dendrite
  evidence_type: IDA
  original_reference_id: PMID:23666762
  qualifier: located_in
  review:
    summary: >-
      dendrite is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0030425 (dendrite), the IDA annotation with qualifier located_in from
      PMID:23666762 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0044297
    label: cell body
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      cell body is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0044297 (cell body), the ISS annotation with qualifier located_in from
      GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0045121
    label: membrane raft
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      membrane raft is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0045121 (membrane raft), the ISS annotation with qualifier located_in
      from GO_REF:0000024 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0097386
    label: glial cell projection
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      glial cell projection is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0097386 (glial cell projection), the ISS annotation with qualifier located_in
      from GO_REF:0000024 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:28755476
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:28755476 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005737
    label: cytoplasm
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      cytoplasm is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005737 (cytoplasm), the ISS annotation with qualifier located_in from
      GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: TAS
  original_reference_id: PMID:26609151
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the TAS annotation with qualifier enables
      from PMID:26609151 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0044304
    label: main axon
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      main axon is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0044304 (main axon), the ISS annotation with qualifier located_in from
      GO_REF:0000024 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0061564
    label: axon development
  evidence_type: TAS
  original_reference_id: PMID:26609151
  qualifier: involved_in
  review:
    summary: >-
      axon development is retained as a core biological-process annotation for MAPT;
      it captures process participation within the synthesized core biology: tau microtubule
      binding, microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0061564 (axon development), the TAS annotation with qualifier involved_in
      from PMID:26609151 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0051879
    label: Hsp90 protein binding
  evidence_type: IPI
  original_reference_id: PMID:17517623
  qualifier: enables
  review:
    summary: >-
      Hsp90 protein binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0051879 (Hsp90 protein binding), the IPI annotation with qualifier enables
      from PMID:17517623 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:25051234
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:25051234 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0046785
    label: microtubule polymerization
  evidence_type: TAS
  original_reference_id: PMID:26996940
  qualifier: involved_in
  review:
    summary: >-
      microtubule polymerization is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0046785 (microtubule polymerization), the TAS annotation with qualifier
      involved_in from PMID:26996940 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019901
    label: protein kinase binding
  evidence_type: ISS
  original_reference_id: PMID:17906291
  qualifier: enables
  review:
    summary: >-
      protein kinase binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0019901 (protein kinase binding), the ISS annotation with qualifier enables
      from PMID:17906291 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0003677
    label: DNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      DNA binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0003677 (DNA binding), the TAS annotation with qualifier enables from
      PMID:28386764 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0003680
    label: minor groove of adenine-thymine-rich DNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      minor groove of adenine-thymine-rich DNA binding is marked over-annotated for
      MAPT because this molecular-function term is too generic, interaction-map-like,
      or weakly informative relative to the gene-specific biology: tau microtubule
      binding, microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0003680 (minor groove of adenine-thymine-rich DNA binding), the TAS annotation
      with qualifier enables from PMID:28386764 may reflect a real assay result or
      interaction, but this GO term does not identify the specific microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation that explains MAPT's role in
      tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. More
      informative gene-specific annotations are present, so this is marked over-annotated
      rather than accepted as a core function.
- term:
    id: GO:0003690
    label: double-stranded DNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      double-stranded DNA binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0003690 (double-stranded DNA binding), the TAS annotation with qualifier
      enables from PMID:28386764 may reflect a real assay result or interaction, but
      this GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:0003697
    label: single-stranded DNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      single-stranded DNA binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0003697 (single-stranded DNA binding), the TAS annotation with qualifier
      enables from PMID:28386764 may reflect a real assay result or interaction, but
      this GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:0003723
    label: RNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      RNA binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0003723 (RNA binding), the TAS annotation with qualifier enables from
      PMID:28386764 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0003779
    label: actin binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      actin binding is retained as a non-core molecular-function annotation for MAPT;
      it records a supported context, interaction, localization, or pathway branch
      that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0003779 (actin binding), the TAS annotation with qualifier enables from
      PMID:28386764 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005576
    label: extracellular region
  evidence_type: NAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      extracellular region is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005576 (extracellular region), the NAS annotation with qualifier located_in
      from PMID:28386764 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005634
    label: nucleus
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      nucleus is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005634 (nucleus), the TAS annotation with qualifier located_in from
      PMID:28386764 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005739
    label: mitochondrion
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      mitochondrion is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005739 (mitochondrion), the TAS annotation with qualifier located_in
      from PMID:28386764 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005886
    label: plasma membrane
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      plasma membrane is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005886 (plasma membrane), the TAS annotation with qualifier located_in
      from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0007267
    label: cell-cell signaling
  evidence_type: NAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      cell-cell signaling is marked over-annotated for MAPT because this biological-process
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0007267 (cell-cell signaling), the NAS annotation with qualifier involved_in
      from PMID:28386764 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: NAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the NAS annotation with qualifier enables
      from PMID:28386764 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0016072
    label: rRNA metabolic process
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      rRNA metabolic process is marked over-annotated for MAPT because this biological-process
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0016072 (rRNA metabolic process), the TAS annotation with qualifier involved_in
      from PMID:28386764 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:0021954
    label: central nervous system neuron development
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      central nervous system neuron development is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0021954 (central nervous system neuron development), the TAS annotation
      with qualifier involved_in from PMID:28386764 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030424
    label: axon
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      axon is retained as a core cellular-component/localization annotation for MAPT;
      it captures site of action or component context within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030424 (axon), the TAS annotation with qualifier located_in from PMID:28386764
      is consistent with MAPT's core role in tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. This action keeps the term because it provides
      a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation
      annotation rather than only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030674
    label: protein-macromolecule adaptor activity
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      protein-macromolecule adaptor activity is retained as a core molecular-function
      annotation for MAPT; it captures activity or binding specificity within the
      synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030674 (protein-macromolecule adaptor activity), the TAS annotation
      with qualifier enables from PMID:28386764 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031113
    label: regulation of microtubule polymerization
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule polymerization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031113 (regulation of microtubule polymerization), the TAS annotation
      with qualifier involved_in from PMID:28386764 is consistent with MAPT's core
      role in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0033044
    label: regulation of chromosome organization
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      regulation of chromosome organization is marked over-annotated for MAPT because
      this biological-process term is too generic, interaction-map-like, or weakly
      informative relative to the gene-specific biology: tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0033044 (regulation of chromosome organization), the TAS annotation with
      qualifier involved_in from PMID:28386764 may reflect a real assay result or
      interaction, but this GO term does not identify the specific microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation that explains MAPT's role in
      tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. More
      informative gene-specific annotations are present, so this is marked over-annotated
      rather than accepted as a core function.
- term:
    id: GO:0034063
    label: stress granule assembly
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      stress granule assembly is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0034063 (stress granule assembly), the TAS annotation with qualifier
      involved_in from PMID:28386764 supports retaining the annotation, but the term
      describes a context-specific outcome or peripheral branch rather than the principal
      MAPT function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0034614
    label: cellular response to reactive oxygen species
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      cellular response to reactive oxygen species is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0034614 (cellular response to reactive oxygen species), the TAS annotation
      with qualifier involved_in from PMID:28386764 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0035091
    label: phosphatidylinositol binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      phosphatidylinositol binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0035091 (phosphatidylinositol binding), the TAS annotation with qualifier
      enables from PMID:28386764 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0036477
    label: somatodendritic compartment
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      somatodendritic compartment is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0036477 (somatodendritic compartment), the TAS annotation with qualifier
      located_in from PMID:28386764 supports retaining the annotation, but the term
      describes a context-specific outcome or peripheral branch rather than the principal
      MAPT function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0043197
    label: dendritic spine
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: located_in
  review:
    summary: >-
      dendritic spine is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0043197 (dendritic spine), the TAS annotation with qualifier located_in
      from PMID:28386764 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0043565
    label: sequence-specific DNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      sequence-specific DNA binding is marked over-annotated for MAPT because this
      molecular-function term is too generic, interaction-map-like, or weakly informative
      relative to the gene-specific biology: tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0043565 (sequence-specific DNA binding), the TAS annotation with qualifier
      enables from PMID:28386764 may reflect a real assay result or interaction, but
      this GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:0048167
    label: regulation of synaptic plasticity
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      regulation of synaptic plasticity is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0048167 (regulation of synaptic plasticity), the TAS annotation with
      qualifier involved_in from PMID:28386764 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0070507
    label: regulation of microtubule cytoskeleton organization
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule cytoskeleton organization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0070507 (regulation of microtubule cytoskeleton organization), the TAS
      annotation with qualifier involved_in from PMID:28386764 is consistent with
      MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      This action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0099077
    label: histone-dependent DNA binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      histone-dependent DNA binding is marked over-annotated for MAPT because this
      molecular-function term is too generic, interaction-map-like, or weakly informative
      relative to the gene-specific biology: tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0099077 (histone-dependent DNA binding), the TAS annotation with qualifier
      enables from PMID:28386764 may reflect a real assay result or interaction, but
      this GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:1900452
    label: regulation of long-term synaptic depression
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      regulation of long-term synaptic depression is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1900452 (regulation of long-term synaptic depression), the TAS annotation
      with qualifier involved_in from PMID:28386764 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1902936
    label: phosphatidylinositol bisphosphate binding
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: enables
  review:
    summary: >-
      phosphatidylinositol bisphosphate binding is retained as a non-core molecular-function
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1902936 (phosphatidylinositol bisphosphate binding), the TAS annotation
      with qualifier enables from PMID:28386764 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1990090
    label: cellular response to nerve growth factor stimulus
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      cellular response to nerve growth factor stimulus is retained as a non-core
      biological-process annotation for MAPT; it records a supported context, interaction,
      localization, or pathway branch that is secondary to tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1990090 (cellular response to nerve growth factor stimulus), the TAS
      annotation with qualifier involved_in from PMID:28386764 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1990416
    label: cellular response to brain-derived neurotrophic factor stimulus
  evidence_type: TAS
  original_reference_id: PMID:28386764
  qualifier: involved_in
  review:
    summary: >-
      cellular response to brain-derived neurotrophic factor stimulus is retained
      as a non-core biological-process annotation for MAPT; it records a supported
      context, interaction, localization, or pathway branch that is secondary to tau
      microtubule binding, microtubule assembly/stability, axonal and neuronal projection
      localization, and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1990416 (cellular response to brain-derived neurotrophic factor stimulus),
      the TAS annotation with qualifier involved_in from PMID:28386764 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0010288
    label: response to lead ion
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: involved_in
  review:
    summary: >-
      response to lead ion is marked over-annotated for MAPT because this biological-process
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0010288 (response to lead ion), the ISS annotation with qualifier involved_in
      from GO_REF:0000024 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
- term:
    id: GO:0050848
    label: regulation of calcium-mediated signaling
  evidence_type: IDA
  original_reference_id: PMID:18272392
  qualifier: involved_in
  review:
    summary: >-
      regulation of calcium-mediated signaling is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0050848 (regulation of calcium-mediated signaling), the IDA annotation
      with qualifier involved_in from PMID:18272392 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:3111527
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:3111527 may reflect a real assay result or interaction, but this GO
      term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0031113
    label: regulation of microtubule polymerization
  evidence_type: TAS
  original_reference_id: PMID:10640627
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule polymerization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031113 (regulation of microtubule polymerization), the TAS annotation
      with qualifier involved_in from PMID:10640627 is consistent with MAPT's core
      role in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0000226
    label: microtubule cytoskeleton organization
  evidence_type: NAS
  original_reference_id: PMID:28377597
  qualifier: involved_in
  review:
    summary: >-
      microtubule cytoskeleton organization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0000226 (microtubule cytoskeleton organization), the NAS annotation with
      qualifier involved_in from PMID:28377597 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: IDA
  original_reference_id: PMID:28377597
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the IDA annotation with qualifier enables
      from PMID:28377597 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0098930
    label: axonal transport
  evidence_type: NAS
  original_reference_id: PMID:28377597
  qualifier: involved_in
  review:
    summary: >-
      axonal transport is retained as a core biological-process annotation for MAPT;
      it captures process participation within the synthesized core biology: tau microtubule
      binding, microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0098930 (axonal transport), the NAS annotation with qualifier involved_in
      from PMID:28377597 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005874
    label: microtubule
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: colocalizes_with
  review:
    summary: >-
      microtubule is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005874 (microtubule), the ISS annotation with qualifier colocalizes_with
      from GO_REF:0000024 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0010629
    label: negative regulation of gene expression
  evidence_type: IMP
  original_reference_id: PMID:23164821
  qualifier: involved_in
  review:
    summary: >-
      negative regulation of gene expression is marked over-annotated for MAPT because
      this biological-process term is too generic, interaction-map-like, or weakly
      informative relative to the gene-specific biology: tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0010629 (negative regulation of gene expression), the IMP annotation
      with qualifier involved_in from PMID:23164821 may reflect a real assay result
      or interaction, but this GO term does not identify the specific microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation that explains
      MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      More informative gene-specific annotations are present, so this is marked over-annotated
      rather than accepted as a core function.
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:20655099
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:20655099 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0007613
    label: memory
  evidence_type: IMP
  original_reference_id: PMID:20655099
  qualifier: involved_in
  review:
    summary: >-
      memory is retained as a non-core biological-process annotation for MAPT; it
      records a supported context, interaction, localization, or pathway branch that
      is secondary to tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0007613 (memory), the IMP annotation with qualifier involved_in from
      PMID:20655099 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0016020
    label: membrane
  evidence_type: IMP
  original_reference_id: PMID:20655099
  qualifier: colocalizes_with
  review:
    summary: >-
      membrane is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0016020 (membrane), the IMP annotation with qualifier colocalizes_with
      from PMID:20655099 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0036477
    label: somatodendritic compartment
  evidence_type: IMP
  original_reference_id: PMID:20655099
  qualifier: located_in
  review:
    summary: >-
      somatodendritic compartment is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0036477 (somatodendritic compartment), the IMP annotation with qualifier
      located_in from PMID:20655099 supports retaining the annotation, but the term
      describes a context-specific outcome or peripheral branch rather than the principal
      MAPT function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0043025
    label: neuronal cell body
  evidence_type: IMP
  original_reference_id: PMID:20655099
  qualifier: located_in
  review:
    summary: >-
      neuronal cell body is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0043025 (neuronal cell body), the IMP annotation with qualifier located_in
      from PMID:20655099 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0050808
    label: synapse organization
  evidence_type: IMP
  original_reference_id: PMID:17270732
  qualifier: involved_in
  review:
    summary: >-
      synapse organization is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0050808 (synapse organization), the IMP annotation with qualifier involved_in
      from PMID:17270732 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1902474
    label: positive regulation of protein localization to synapse
  evidence_type: IMP
  original_reference_id: PMID:20655099
  qualifier: involved_in
  review:
    summary: >-
      positive regulation of protein localization to synapse is retained as a non-core
      biological-process annotation for MAPT; it records a supported context, interaction,
      localization, or pathway branch that is secondary to tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1902474 (positive regulation of protein localization to synapse), the
      IMP annotation with qualifier involved_in from PMID:20655099 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0003677
    label: DNA binding
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: enables
  review:
    summary: >-
      DNA binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0003677 (DNA binding), the ISS annotation with qualifier enables from
      GO_REF:0000024 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0005634
    label: nucleus
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      nucleus is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005634 (nucleus), the ISS annotation with qualifier located_in from
      GO_REF:0000024 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: ISS
  original_reference_id: GO_REF:0000024
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the ISS annotation with qualifier located_in from
      GO_REF:0000024 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0006974
    label: DNA damage response
  evidence_type: IMP
  original_reference_id: PMID:21131359
  qualifier: involved_in
  review:
    summary: >-
      DNA damage response is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0006974 (DNA damage response), the IMP annotation with qualifier involved_in
      from PMID:21131359 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0010917
    label: negative regulation of mitochondrial membrane potential
  evidence_type: IMP
  original_reference_id: PMID:22920254
  qualifier: involved_in
  review:
    summary: >-
      negative regulation of mitochondrial membrane potential is retained as a non-core
      biological-process annotation for MAPT; it records a supported context, interaction,
      localization, or pathway branch that is secondary to tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0010917 (negative regulation of mitochondrial membrane potential), the
      IMP annotation with qualifier involved_in from PMID:22920254 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0090140
    label: regulation of mitochondrial fission
  evidence_type: IC
  original_reference_id: PMID:22367970
  qualifier: involved_in
  review:
    summary: >-
      regulation of mitochondrial fission is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0090140 (regulation of mitochondrial fission), the IC annotation with
      qualifier involved_in from PMID:22367970 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1900034
    label: regulation of cellular response to heat
  evidence_type: IMP
  original_reference_id: PMID:21131359
  qualifier: involved_in
  review:
    summary: >-
      regulation of cellular response to heat is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1900034 (regulation of cellular response to heat), the IMP annotation
      with qualifier involved_in from PMID:21131359 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1903748
    label: negative regulation of protein localization to mitochondrion
  evidence_type: IMP
  original_reference_id: PMID:22920254
  qualifier: involved_in
  review:
    summary: >-
      negative regulation of protein localization to mitochondrion is retained as
      a non-core biological-process annotation for MAPT; it records a supported context,
      interaction, localization, or pathway branch that is secondary to tau microtubule
      binding, microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:1903748 (negative regulation of protein localization to mitochondrion),
      the IMP annotation with qualifier involved_in from PMID:22920254 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0048312
    label: intracellular distribution of mitochondria
  evidence_type: IMP
  original_reference_id: PMID:21854751
  qualifier: involved_in
  review:
    summary: >-
      intracellular distribution of mitochondria is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0048312 (intracellular distribution of mitochondria), the IMP annotation
      with qualifier involved_in from PMID:21854751 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0001774
    label: microglial cell activation
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      microglial cell activation is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0001774 (microglial cell activation), the TAS annotation with qualifier
      involved_in from PMID:26363795 supports retaining the annotation, but the term
      describes a context-specific outcome or peripheral branch rather than the principal
      MAPT function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0003779
    label: actin binding
  evidence_type: TAS
  original_reference_id: PMID:27306859
  qualifier: enables
  review:
    summary: >-
      actin binding is retained as a non-core molecular-function annotation for MAPT;
      it records a supported context, interaction, localization, or pathway branch
      that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0003779 (actin binding), the TAS annotation with qualifier enables from
      PMID:27306859 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the TAS annotation with qualifier enables
      from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019896
    label: axonal transport of mitochondrion
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      axonal transport of mitochondrion is retained as a non-core biological-process
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0019896 (axonal transport of mitochondrion), the TAS annotation with
      qualifier involved_in from PMID:26363795 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030424
    label: axon
  evidence_type: NAS
  original_reference_id: PMID:26363795
  qualifier: located_in
  review:
    summary: >-
      axon is retained as a core cellular-component/localization annotation for MAPT;
      it captures site of action or component context within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030424 (axon), the NAS annotation with qualifier located_in from PMID:26363795
      is consistent with MAPT's core role in tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. This action keeps the term because it provides
      a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation
      annotation rather than only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031122
    label: cytoplasmic microtubule organization
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      cytoplasmic microtubule organization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031122 (cytoplasmic microtubule organization), the TAS annotation with
      qualifier involved_in from PMID:26363795 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031175
    label: neuron projection development
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      neuron projection development is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031175 (neuron projection development), the TAS annotation with qualifier
      involved_in from PMID:26363795 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0034452
    label: dynactin binding
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: enables
  review:
    summary: >-
      dynactin binding is retained as a non-core molecular-function annotation for
      MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0034452 (dynactin binding), the TAS annotation with qualifier enables
      from PMID:26363795 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0034605
    label: cellular response to heat
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      cellular response to heat is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0034605 (cellular response to heat), the TAS annotation with qualifier
      involved_in from PMID:26363795 supports retaining the annotation, but the term
      describes a context-specific outcome or peripheral branch rather than the principal
      MAPT function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0048143
    label: astrocyte activation
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      astrocyte activation is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0048143 (astrocyte activation), the TAS annotation with qualifier involved_in
      from PMID:26363795 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0051721
    label: protein phosphatase 2A binding
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: enables
  review:
    summary: >-
      protein phosphatase 2A binding is retained as a non-core molecular-function
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0051721 (protein phosphatase 2A binding), the TAS annotation with qualifier
      enables from PMID:26363795 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0072386
    label: plus-end-directed organelle transport along microtubule
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      plus-end-directed organelle transport along microtubule is retained as a non-core
      biological-process annotation for MAPT; it records a supported context, interaction,
      localization, or pathway branch that is secondary to tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0072386 (plus-end-directed organelle transport along microtubule), the
      TAS annotation with qualifier involved_in from PMID:26363795 supports retaining
      the annotation, but the term describes a context-specific outcome or peripheral
      branch rather than the principal MAPT function: tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. Keeping it as non-core prevents broad pathway
      participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0098930
    label: axonal transport
  evidence_type: TAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      axonal transport is retained as a core biological-process annotation for MAPT;
      it captures process participation within the synthesized core biology: tau microtubule
      binding, microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0098930 (axonal transport), the TAS annotation with qualifier involved_in
      from PMID:26363795 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1902988
    label: neurofibrillary tangle assembly
  evidence_type: NAS
  original_reference_id: PMID:26363795
  qualifier: involved_in
  review:
    summary: >-
      neurofibrillary tangle assembly is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:1902988 (neurofibrillary tangle assembly), the NAS annotation with qualifier
      involved_in from PMID:26363795 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005737
    label: cytoplasm
  evidence_type: IDA
  original_reference_id: PMID:27356871
  qualifier: located_in
  review:
    summary: >-
      cytoplasm is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0005737 (cytoplasm), the IDA annotation with qualifier located_in from
      PMID:27356871 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015630
    label: microtubule cytoskeleton
  evidence_type: IDA
  original_reference_id: PMID:27356871
  qualifier: located_in
  review:
    summary: >-
      microtubule cytoskeleton is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015630 (microtubule cytoskeleton), the IDA annotation with qualifier
      located_in from PMID:27356871 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031110
    label: regulation of microtubule polymerization or depolymerization
  evidence_type: IMP
  original_reference_id: PMID:27356871
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule polymerization or depolymerization is retained as
      a core biological-process annotation for MAPT; it captures process participation
      within the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031110 (regulation of microtubule polymerization or depolymerization),
      the IMP annotation with qualifier involved_in from PMID:27356871 is consistent
      with MAPT's core role in tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. This action keeps the term because it provides a specific, evidence-backed
      microtubule, tubulin-binding, localization, or tau-phosphorylation annotation
      rather than only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1903829
    label: positive regulation of protein localization
  evidence_type: IMP
  original_reference_id: PMID:27356871
  qualifier: involved_in
  review:
    summary: >-
      positive regulation of protein localization is marked over-annotated for MAPT
      because this biological-process term is too generic, interaction-map-like, or
      weakly informative relative to the gene-specific biology: tau microtubule binding,
      microtubule assembly/stability, axonal and neuronal projection localization,
      and phosphorylation-dependent regulation of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:1903829 (positive regulation of protein localization), the IMP annotation
      with qualifier involved_in from PMID:27356871 may reflect a real assay result
      or interaction, but this GO term does not identify the specific microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation that explains
      MAPT's role in tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      More informative gene-specific annotations are present, so this is marked over-annotated
      rather than accepted as a core function.
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: IDA
  original_reference_id: PMID:10747907
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the IDA annotation with qualifier located_in from
      PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005886
    label: plasma membrane
  evidence_type: IDA
  original_reference_id: PMID:10747907
  qualifier: located_in
  review:
    summary: >-
      plasma membrane is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005886 (plasma membrane), the IDA annotation with qualifier located_in
      from PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: IMP
  original_reference_id: PMID:10747907
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the IMP annotation with qualifier enables
      from PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0015630
    label: microtubule cytoskeleton
  evidence_type: IDA
  original_reference_id: PMID:15147841
  qualifier: located_in
  review:
    summary: >-
      microtubule cytoskeleton is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0015630 (microtubule cytoskeleton), the IDA annotation with qualifier
      located_in from PMID:15147841 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030673
    label: axolemma
  evidence_type: IDA
  original_reference_id: PMID:10747907
  qualifier: located_in
  review:
    summary: >-
      axolemma is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030673 (axolemma), the IDA annotation with qualifier located_in from
      PMID:10747907 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IMP
  original_reference_id: PMID:14769047
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IMP annotation with qualifier
      enables from PMID:14769047 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0042802
    label: identical protein binding
  evidence_type: IMP
  original_reference_id: PMID:7929085
  qualifier: enables
  review:
    summary: >-
      identical protein binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0042802 (identical protein binding), the IMP annotation with qualifier
      enables from PMID:7929085 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0070507
    label: regulation of microtubule cytoskeleton organization
  evidence_type: IMP
  original_reference_id: PMID:15147841
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule cytoskeleton organization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0070507 (regulation of microtubule cytoskeleton organization), the IMP
      annotation with qualifier involved_in from PMID:15147841 is consistent with
      MAPT's core role in tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      This action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0097418
    label: neurofibrillary tangle
  evidence_type: IDA
  original_reference_id: PMID:2484340
  qualifier: located_in
  review:
    summary: >-
      neurofibrillary tangle is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0097418 (neurofibrillary tangle), the IDA annotation with qualifier located_in
      from PMID:2484340 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0097435
    label: supramolecular fiber organization
  evidence_type: IMP
  original_reference_id: PMID:14769047
  qualifier: involved_in
  review:
    summary: >-
      supramolecular fiber organization is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0097435 (supramolecular fiber organization), the IMP annotation with
      qualifier involved_in from PMID:14769047 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0099609
    label: microtubule lateral binding
  evidence_type: IMP
  original_reference_id: PMID:15147841
  qualifier: enables
  review:
    summary: >-
      microtubule lateral binding is retained as a core molecular-function annotation
      for MAPT; it captures activity or binding specificity within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0099609 (microtubule lateral binding), the IMP annotation with qualifier
      enables from PMID:15147841 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:1990000
    label: amyloid fibril formation
  evidence_type: IMP
  original_reference_id: PMID:7929085
  qualifier: involved_in
  review:
    summary: >-
      amyloid fibril formation is retained as a core biological-process annotation
      for MAPT; it captures process participation within the synthesized core biology:
      tau microtubule binding, microtubule assembly/stability, axonal and neuronal
      projection localization, and phosphorylation-dependent regulation of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:1990000 (amyloid fibril formation), the IMP annotation with qualifier
      involved_in from PMID:7929085 is consistent with MAPT's core role in tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030425
    label: dendrite
  evidence_type: IDA
  original_reference_id: PMID:25620700
  qualifier: located_in
  review:
    summary: >-
      dendrite is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0030425 (dendrite), the IDA annotation with qualifier located_in from
      PMID:25620700 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0044297
    label: cell body
  evidence_type: IDA
  original_reference_id: PMID:25620700
  qualifier: located_in
  review:
    summary: >-
      cell body is retained as a non-core cellular-component/localization annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0044297 (cell body), the IDA annotation with qualifier located_in from
      PMID:25620700 supports retaining the annotation, but the term describes a context-specific
      outcome or peripheral branch rather than the principal MAPT function: tau binding
      to tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. Keeping it as non-core
      prevents broad pathway participation from being promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0097418
    label: neurofibrillary tangle
  evidence_type: NAS
  original_reference_id: PMID:25620700
  qualifier: located_in
  review:
    summary: >-
      neurofibrillary tangle is retained as a core cellular-component/localization
      annotation for MAPT; it captures site of action or component context within
      the synthesized core biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0097418 (neurofibrillary tangle), the NAS annotation with qualifier located_in
      from PMID:25620700 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0036464
    label: cytoplasmic ribonucleoprotein granule
  evidence_type: IDA
  original_reference_id: PMID:15121898
  qualifier: located_in
  review:
    summary: >-
      cytoplasmic ribonucleoprotein granule is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0036464 (cytoplasmic ribonucleoprotein granule), the IDA annotation with
      qualifier located_in from PMID:15121898 supports retaining the annotation, but
      the term describes a context-specific outcome or peripheral branch rather than
      the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:23589030
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:23589030 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:23399914
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:23399914 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0031116
    label: positive regulation of microtubule polymerization
  evidence_type: IDA
  original_reference_id: PMID:7706316
  qualifier: involved_in
  review:
    summary: >-
      positive regulation of microtubule polymerization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031116 (positive regulation of microtubule polymerization), the IDA
      annotation with qualifier involved_in from PMID:7706316 is consistent with MAPT's
      core role in tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      This action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005829
    label: cytosol
  evidence_type: TAS
  original_reference_id: Reactome:R-HSA-201629
  qualifier: located_in
  review:
    summary: >-
      cytosol is retained as a core cellular-component/localization annotation for
      MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005829 (cytosol), the TAS annotation with qualifier located_in from
      Reactome:R-HSA-201629 is consistent with MAPT's core role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. This action keeps
      the term because it provides a specific, evidence-backed microtubule, tubulin-binding,
      localization, or tau-phosphorylation annotation rather than only a downstream
      phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0034185
    label: apolipoprotein binding
  evidence_type: IPI
  original_reference_id: PMID:7566652
  qualifier: enables
  review:
    summary: >-
      apolipoprotein binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0034185 (apolipoprotein binding), the IPI annotation with qualifier enables
      from PMID:7566652 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0010506
    label: regulation of autophagy
  evidence_type: IGI
  original_reference_id: PMID:19074461
  qualifier: acts_upstream_of_or_within
  review:
    summary: >-
      regulation of autophagy is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0010506 (regulation of autophagy), the IGI annotation with qualifier
      acts_upstream_of_or_within from PMID:19074461 supports retaining the annotation,
      but the term describes a context-specific outcome or peripheral branch rather
      than the principal MAPT function: tau binding to tubulin and microtubules, regulation
      of microtubule assembly and stability, axonal localization, and tau phosphorylation-state
      biology. Keeping it as non-core prevents broad pathway participation from being
      promoted to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0071813
    label: lipoprotein particle binding
  evidence_type: IPI
  original_reference_id: PMID:7972031
  qualifier: enables
  review:
    summary: >-
      lipoprotein particle binding is retained as a non-core molecular-function annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0071813 (lipoprotein particle binding), the IPI annotation with qualifier
      enables from PMID:7972031 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0034399
    label: nuclear periphery
  evidence_type: IDA
  original_reference_id: PMID:19157893
  qualifier: located_in
  review:
    summary: >-
      nuclear periphery is retained as a non-core cellular-component/localization
      annotation for MAPT; it records a supported context, interaction, localization,
      or pathway branch that is secondary to tau microtubule binding, microtubule
      assembly/stability, axonal and neuronal projection localization, and phosphorylation-dependent
      regulation of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0034399 (nuclear periphery), the IDA annotation with qualifier located_in
      from PMID:19157893 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019899
    label: enzyme binding
  evidence_type: IPI
  original_reference_id: PMID:20133804
  qualifier: enables
  review:
    summary: >-
      enzyme binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0019899 (enzyme binding), the IPI annotation with qualifier enables from
      PMID:20133804 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:10987820
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:10987820 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:9689133
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:9689133 may reflect a real assay result or interaction, but this GO
      term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
- term:
    id: GO:0005886
    label: plasma membrane
  evidence_type: IDA
  original_reference_id: PMID:8522593
  qualifier: located_in
  review:
    summary: >-
      plasma membrane is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0005886 (plasma membrane), the IDA annotation with qualifier located_in
      from PMID:8522593 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0008017
    label: microtubule binding
  evidence_type: IDA
  original_reference_id: PMID:1918161
  qualifier: enables
  review:
    summary: >-
      microtubule binding is retained as a core molecular-function annotation for
      MAPT; it captures activity or binding specificity within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0008017 (microtubule binding), the IDA annotation with qualifier enables
      from PMID:1918161 is consistent with MAPT's core role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0017124
    label: SH3 domain binding
  evidence_type: IPI
  original_reference_id: PMID:9763511
  qualifier: enables
  review:
    summary: >-
      SH3 domain binding is retained as a non-core molecular-function annotation for
      MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0017124 (SH3 domain binding), the IPI annotation with qualifier enables
      from PMID:9763511 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0019899
    label: enzyme binding
  evidence_type: IPI
  original_reference_id: PMID:12888622
  qualifier: enables
  review:
    summary: >-
      enzyme binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0019899 (enzyme binding), the IPI annotation with qualifier enables from
      PMID:12888622 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0019899
    label: enzyme binding
  evidence_type: IPI
  original_reference_id: PMID:9736630
  qualifier: enables
  review:
    summary: >-
      enzyme binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0019899 (enzyme binding), the IPI annotation with qualifier enables from
      PMID:9736630 may reflect a real assay result or interaction, but this GO term
      does not identify the specific microtubule, tubulin-binding, localization, or
      tau-phosphorylation annotation that explains MAPT's role in tau binding to tubulin
      and microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. More informative gene-specific annotations
      are present, so this is marked over-annotated rather than accepted as a core
      function.
- term:
    id: GO:0048699
    label: generation of neurons
  evidence_type: NAS
  original_reference_id: PMID:8522593
  qualifier: involved_in
  review:
    summary: >-
      generation of neurons is retained as a non-core biological-process annotation
      for MAPT; it records a supported context, interaction, localization, or pathway
      branch that is secondary to tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: KEEP_AS_NON_CORE
    reason: >-
      For GO:0048699 (generation of neurons), the NAS annotation with qualifier involved_in
      from PMID:8522593 supports retaining the annotation, but the term describes
      a context-specific outcome or peripheral branch rather than the principal MAPT
      function: tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      Keeping it as non-core prevents broad pathway participation from being promoted
      to core function.
    additional_reference_ids:
    - PMID:28386764
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0000226
    label: microtubule cytoskeleton organization
  evidence_type: IDA
  original_reference_id: PMID:1057175
  qualifier: involved_in
  review:
    summary: >-
      microtubule cytoskeleton organization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0000226 (microtubule cytoskeleton organization), the IDA annotation with
      qualifier involved_in from PMID:1057175 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030424
    label: axon
  evidence_type: IDA
  original_reference_id: PMID:8642405
  qualifier: located_in
  review:
    summary: >-
      axon is retained as a core cellular-component/localization annotation for MAPT;
      it captures site of action or component context within the synthesized core
      biology: tau microtubule binding, microtubule assembly/stability, axonal and
      neuronal projection localization, and phosphorylation-dependent regulation of
      tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030424 (axon), the IDA annotation with qualifier located_in from PMID:8642405
      is consistent with MAPT's core role in tau binding to tubulin and microtubules,
      regulation of microtubule assembly and stability, axonal localization, and tau
      phosphorylation-state biology. This action keeps the term because it provides
      a specific, evidence-backed microtubule, tubulin-binding, localization, or tau-phosphorylation
      annotation rather than only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0030426
    label: growth cone
  evidence_type: IDA
  original_reference_id: PMID:8642405
  qualifier: located_in
  review:
    summary: >-
      growth cone is retained as a core cellular-component/localization annotation
      for MAPT; it captures site of action or component context within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0030426 (growth cone), the IDA annotation with qualifier located_in from
      PMID:8642405 is consistent with MAPT's core role in tau binding to tubulin and
      microtubules, regulation of microtubule assembly and stability, axonal localization,
      and tau phosphorylation-state biology. This action keeps the term because it
      provides a specific, evidence-backed microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation rather than only a downstream phenotype or
      generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031113
    label: regulation of microtubule polymerization
  evidence_type: NAS
  original_reference_id: PMID:1057175
  qualifier: involved_in
  review:
    summary: >-
      regulation of microtubule polymerization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031113 (regulation of microtubule polymerization), the NAS annotation
      with qualifier involved_in from PMID:1057175 is consistent with MAPT's core
      role in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0031116
    label: positive regulation of microtubule polymerization
  evidence_type: IDA
  original_reference_id: PMID:1421571
  qualifier: involved_in
  review:
    summary: >-
      positive regulation of microtubule polymerization is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0031116 (positive regulation of microtubule polymerization), the IDA
      annotation with qualifier involved_in from PMID:1421571 is consistent with MAPT's
      core role in tau binding to tubulin and microtubules, regulation of microtubule
      assembly and stability, axonal localization, and tau phosphorylation-state biology.
      This action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0045298
    label: tubulin complex
  evidence_type: IDA
  original_reference_id: PMID:8642405
  qualifier: part_of
  review:
    summary: >-
      tubulin complex is directionally related to MAPT biology but is not the best
      curation target; tubulin binding better captures the specific supported function
      or process.
    action: MODIFY
    reason: >-
      For GO:0045298 (tubulin complex), the IDA annotation with qualifier part_of
      from PMID:8642405 supports a relationship to MAPT, but the current term is less
      precise than tubulin binding for the evidence and for the synthesized core/non-core
      role of MAPT. The MODIFY action preserves the biological intent while pointing
      curators to the more informative GO term.
    proposed_replacement_terms:
    - id: GO:0015631
      label: tubulin binding
    additional_reference_ids:
    - PMID:15147841
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0045773
    label: positive regulation of axon extension
  evidence_type: IDA
  original_reference_id: PMID:1389180
  qualifier: involved_in
  review:
    summary: >-
      positive regulation of axon extension is retained as a core biological-process
      annotation for MAPT; it captures process participation within the synthesized
      core biology: tau microtubule binding, microtubule assembly/stability, axonal
      and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: ACCEPT
    reason: >-
      For GO:0045773 (positive regulation of axon extension), the IDA annotation with
      qualifier involved_in from PMID:1389180 is consistent with MAPT's core role
      in tau binding to tubulin and microtubules, regulation of microtubule assembly
      and stability, axonal localization, and tau phosphorylation-state biology. This
      action keeps the term because it provides a specific, evidence-backed microtubule,
      tubulin-binding, localization, or tau-phosphorylation annotation rather than
      only a downstream phenotype or generic interaction label.
    additional_reference_ids:
    - PMID:15147841
    - PMID:10747907
    - PMID:2484340
    - file:human/MAPT/MAPT-uniprot.txt
- term:
    id: GO:0005515
    label: protein binding
  evidence_type: IPI
  original_reference_id: PMID:15953362
  qualifier: enables
  review:
    summary: >-
      protein binding is marked over-annotated for MAPT because this molecular-function
      term is too generic, interaction-map-like, or weakly informative relative to
      the gene-specific biology: tau microtubule binding, microtubule assembly/stability,
      axonal and neuronal projection localization, and phosphorylation-dependent regulation
      of tau behavior.
    action: MARK_AS_OVER_ANNOTATED
    reason: >-
      For GO:0005515 (protein binding), the IPI annotation with qualifier enables
      from PMID:15953362 may reflect a real assay result or interaction, but this
      GO term does not identify the specific microtubule, tubulin-binding, localization,
      or tau-phosphorylation annotation that explains MAPT's role in tau binding to
      tubulin and microtubules, regulation of microtubule assembly and stability,
      axonal localization, and tau phosphorylation-state biology. More informative
      gene-specific annotations are present, so this is marked over-annotated rather
      than accepted as a core function.
    additional_reference_ids:
    - file:human/MAPT/MAPT-notes.md
references:
- id: GO_REF:0000002
  title: Gene Ontology annotation through association of InterPro records with 
    GO terms
  findings: []
- id: GO_REF:0000024
  title: Manual transfer of experimentally-verified manual GO annotation data to
    orthologs by curator judgment of sequence similarity
  findings: []
- id: GO_REF:0000033
  title: Annotation inferences using phylogenetic trees
  findings: []
- id: GO_REF:0000044
  title: Gene Ontology annotation based on UniProtKB/Swiss-Prot Subcellular 
    Location vocabulary mapping, accompanied by conservative changes to GO terms
    applied by UniProt
  findings: []
- id: GO_REF:0000052
  title: Gene Ontology annotation based on curation of immunofluorescence data
  findings: []
- id: GO_REF:0000108
  title: Automatic assignment of GO terms using logical inference, based on on 
    inter-ontology links
  findings: []
- id: PMID:1057175
  title: A protein factor essential for microtubule assembly.
  findings: []
- id: PMID:10640627
  title: Regulation of phosphorylation of neuronal microtubule-associated 
    proteins MAP1b and MAP2 by protein phosphatase-2A and -2B in rat brain.
  findings: []
- id: PMID:10747907
  title: Interaction of tau with the neural membrane cortex is regulated by 
    phosphorylation at sites that are modified in paired helical filaments.
  findings: []
  reference_review:
    relevance: HIGH
    correctness: VERIFIED
    review_notes: Primary study supporting axonal tau association with the 
      neural membrane cortex and phosphorylation-sensitive membrane association.
- id: PMID:10987820
  title: Tau promoter confers neuronal specificity and binds Sp1 and AP-2.
  findings: []
- id: PMID:12888622
  title: 'Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in
    Alzheimer''s disease.'
  findings: []
- id: PMID:1389180
  title: Suppression of MAP2 in cultured cerebellar macroneurons inhibits minor 
    neurite formation.
  findings: []
- id: PMID:1421571
  title: Modulation of the dynamic instability of tubulin assembly by the 
    microtubule-associated protein tau.
  findings: []
- id: PMID:14769047
  title: Tau paired helical filaments from Alzheimer's disease brain and 
    assembled in vitro are based on beta-structure in the core domain.
  findings: []
- id: PMID:15121898
  title: The composition of Staufen-containing RNA granules from human cells 
    indicates their role in the regulated transport and translation of messenger
    RNAs.
  findings: []
- id: PMID:15147841
  title: Surface-decoration of microtubules by human tau.
  findings: []
  reference_review:
    relevance: HIGH
    correctness: VERIFIED
    review_notes: Primary structural/microtubule study supporting tau 
      microtubule surface binding and microtubule stabilization.
- id: PMID:15654759
  title: 'Residual structure in the repeat domain of tau: echoes of microtubule binding
    and paired helical filament formation.'
  findings: []
- id: PMID:15953362
  title: Sequestosome 1/p62 shuttles polyubiquitinated tau for proteasomal 
    degradation.
  findings: []
- id: PMID:16275660
  title: Identification of VCP/p97, carboxyl terminus of Hsp70-interacting 
    protein (CHIP), and amphiphysin II interaction partners using membrane-based
    human proteome arrays.
  findings: []
- id: PMID:16446437
  title: Abeta and tau form soluble complexes that may promote self aggregation 
    of both into the insoluble forms observed in Alzheimer's disease.
  findings: []
- id: PMID:16923168
  title: Tau-tubulin kinase 1 (TTBK1), a neuron-specific tau kinase candidate, 
    is involved in tau phosphorylation and aggregation.
  findings: []
- id: PMID:17270732
  title: Synapse loss and microglial activation precede tangles in a P301S 
    tauopathy mouse model.
  findings: []
- id: PMID:17317006
  title: 14-3-3zeta facilitates GSK3beta-catalyzed tau phosphorylation in 
    HEK-293 cells by a mechanism that requires phosphorylation of GSK3beta on 
    Ser9.
  findings: []
- id: PMID:17517623
  title: Roles of heat-shock protein 90 in maintaining and facilitating the 
    neurodegenerative phenotype in tauopathies.
  findings: []
- id: PMID:17906291
  title: DYRK1A-mediated hyperphosphorylation of Tau. A functional link between 
    Down syndrome and Alzheimer disease.
  findings: []
- id: PMID:18272392
  title: Extracellular tau promotes intracellular calcium increase through M1 
    and M3 muscarinic receptors in neuronal cells.
  findings: []
- id: PMID:18292230
  title: Akt and CHIP coregulate tau degradation through coordinated 
    interactions.
  findings: []
- id: PMID:19074461
  title: Tau deletion exacerbates the phenotype of Niemann-Pick type C mice and 
    implicates autophagy in pathogenesis.
  findings: []
- id: PMID:19138662
  title: Effect of phosphorylation on interaction of human tau protein with 
    14-3-3zeta.
  findings: []
- id: PMID:19157893
  title: Fluorescence microscopy and 3D image reconstruction of cytokine 
    initiated disruption of the Parkinson disease associated proteins 
    alpha-synuclein, tau and ubiquitin in cultured glial cells.
  findings: []
- id: PMID:1918161
  title: Tau protein binds to microtubules through a flexible array of 
    distributed weak sites.
  findings: []
- id: PMID:19457097
  title: Tau--an inhibitor of deacetylase HDAC6 function.
  findings: []
- id: PMID:19527721
  title: Excitatory amino acid transporter 2 associates with phosphorylated tau 
    and is localized in neurofibrillary tangles of tauopathic brains.
  findings: []
- id: PMID:19647741
  title: Phosphorylation of more than one site is required for tight interaction
    of human tau protein with 14-3-3zeta.
  findings: []
- id: PMID:20133804
  title: A role for FKBP52 in Tau protein function.
  findings: []
- id: PMID:20655099
  title: Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's
    disease mouse models.
  findings: []
  reference_review:
    relevance: MEDIUM
    correctness: VERIFIED
    review_notes: Primary AD mouse-model paper supporting tau-dependent 
      dendritic/Fyn/synaptic and memory phenotypes, kept as non-core downstream 
      disease context.
- id: PMID:21131359
  title: Nuclear tau, a key player in neuronal DNA protection.
  findings: []
- id: PMID:21692989
  title: Tyrosine phosphorylation of tau regulates its interactions with Fyn SH2
    domains, but not SH3 domains, altering the cellular localization of tau.
  findings: []
- id: PMID:21854751
  title: Tau accumulation causes mitochondrial distribution deficits in neurons 
    in a mouse model of tauopathy and in human Alzheimer's disease brain.
  findings: []
- id: PMID:22367970
  title: 'Abnormal interaction between the mitochondrial fission protein Drp1 and
    hyperphosphorylated tau in Alzheimer''s disease neurons: implications for mitochondrial
    dysfunction and neuronal damage.'
  findings: []
- id: PMID:22920254
  title: Tau promotes neurodegeneration via DRP1 mislocalization in vivo.
  findings: []
  reference_review:
    relevance: MEDIUM
    correctness: VERIFIED
    review_notes: Full-text primary tauopathy model paper supporting 
      actin-mediated mitochondrial dynamics phenotypes, kept as non-core 
      downstream toxicity context.
- id: PMID:23164821
  title: Clusterin regulates Ξ²-amyloid toxicity via Dickkopf-1-driven induction 
    of the wnt-PCP-JNK pathway.
  findings: []
- id: PMID:23362255
  title: Isomerase Pin1 stimulates dephosphorylation of tau protein at 
    cyclin-dependent kinase (Cdk5)-dependent Alzheimer phosphorylation sites.
  findings: []
- id: PMID:23399914
  title: Increased expression of BIN1 mediates Alzheimer genetic risk by 
    modulating tau pathology.
  findings: []
- id: PMID:23589030
  title: Clathrin adaptor CALM/PICALM is associated with neurofibrillary tangles
    and is cleaved in Alzheimer's brains.
  findings: []
- id: PMID:23666762
  title: Role of individual MARK isoforms in phosphorylation of tau at Ser²⁢² in
    Alzheimer's disease.
  findings: []
- id: PMID:24028865
  title: Impact of the cellular prion protein on amyloid-Ξ² and 3PO-tau 
    processing.
  findings: []
- id: PMID:24251416
  title: Phosphorylation of human Tau protein by microtubule affinity-regulating
    kinase 2.
  findings: []
- id: PMID:2484340
  title: 'Multiple isoforms of human microtubule-associated protein tau: sequences
    and localization in neurofibrillary tangles of Alzheimer''s disease.'
  findings: []
  reference_review:
    relevance: HIGH
    correctness: VERIFIED
    review_notes: Primary human tau isoform paper supporting incorporation of 
      multiple tau isoforms into Alzheimer neurofibrillary tangles.
- id: PMID:25051234
  title: Intracellular clusterin interacts with brain isoforms of the bridging 
    integrator 1 and with the microtubule-associated protein Tau in Alzheimer's 
    disease.
  findings: []
- id: PMID:25156556
  title: AlphaScreen HTS and live-cell bioluminescence resonance energy transfer
    (BRET) assays for identification of Tau-Fyn SH3 interaction inhibitors for 
    Alzheimer disease.
  findings: []
- id: PMID:25556849
  title: 'A novel DYRK1A (dual specificity tyrosine phosphorylation-regulated kinase
    1A) inhibitor for the treatment of Alzheimer''s disease: effect on Tau and amyloid
    pathologies in vitro.'
  findings: []
- id: PMID:25620700
  title: A genome-wide gene-expression analysis and database in transgenic mice 
    during development of amyloid or tau pathology.
  findings: []
- id: PMID:26014385
  title: LRRK2 Promotes Tau Accumulation, Aggregation and Release.
  findings: []
- id: PMID:26363795
  title: 'The Role of MAPT in Neurodegenerative Diseases: Genetics, Mechanisms and
    Therapy.'
  findings: []
  reference_review:
    relevance: MEDIUM
    correctness: VERIFIED
    review_notes: Review supporting MAPT roles in neurodegenerative disease, 
      microtubules, axonal transport, and neurofibrillary tangles.
- id: PMID:26402096
  title: Identification of Novel Tau Interactions with Endoplasmic Reticulum 
    Proteins in Alzheimer's Disease Brain.
  findings: []
- id: PMID:26609151
  title: Defects in Synaptic Plasticity, Reduced NMDA-Receptor Transport, and 
    Instability of Postsynaptic Density Proteins in Mice Lacking 
    Microtubule-Associated Protein 1A.
  findings: []
- id: PMID:26996940
  title: Regulation of Microtubule Assembly by Tau and not by Pin1.
  findings: []
- id: PMID:27044754
  title: FRMD4A-cytohesin signaling modulates the cellular release of tau.
  findings: []
- id: PMID:27306859
  title: Tau physiology and pathomechanisms in frontotemporal lobar 
    degeneration.
  findings: []
- id: PMID:27356871
  title: Acetylated tau destabilizes the cytoskeleton in the axon initial 
    segment and is mislocalized to the somatodendritic compartment.
  findings: []
- id: PMID:28377597
  title: Dyrk1A overexpression leads to increase of 3R-tau expression and 
    cognitive deficits in Ts65Dn Down syndrome mice.
  findings: []
- id: PMID:28386764
  title: Roles of tau protein in health and disease.
  findings: []
  reference_review:
    relevance: MEDIUM
    correctness: VERIFIED
    review_notes: Full-text review supporting broad tau health/disease biology; 
      useful for secondary pathway context but not sufficient to make all broad 
      review-derived terms core.
- id: PMID:28650319
  title: Somatostatin binds to the human amyloid Ξ² peptide and favors the 
    formation of distinct oligomers.
  findings: []
- id: PMID:28755476
  title: Regulation of the interaction between the neuronal BIN1 isoform 1 and 
    Tau proteins - role of the SH3 domain.
  findings: []
- id: PMID:29764935
  title: The disorderly conduct of Hsc70 and its interaction with the 
    Alzheimer's-related Tau protein.
  findings: []
- id: PMID:30158706
  title: Structures of filaments from Pick's disease reveal a novel tau protein 
    fold.
  findings: []
- id: PMID:30668577
  title: Role of tau N-terminal motif in the secretion of human tau by End 
    Binding proteins.
  findings: []
- id: PMID:30894745
  title: Novel tau filament fold in chronic traumatic encephalopathy encloses 
    hydrophobic molecules.
  findings: []
- id: PMID:3111527
  title: 'Comparison of S100b protein with calmodulin: interactions with melittin
    and microtubule-associated tau proteins and inhibition of phosphorylation of tau
    proteins by protein kinase C.'
  findings: []
- id: PMID:31640277
  title: GSKIP-Mediated Anchoring Increases Phosphorylation of Tau by PKA but 
    Not by GSK3beta via cAMP/PKA/GSKIP/GSK3/Tau Axis Signaling in Cerebrospinal 
    Fluid and iPS Cells in Alzheimer Disease.
  findings: []
- id: PMID:32272059
  title: A Translocation Pathway for Vesicle-Mediated Unconventional Protein 
    Secretion.
  findings: []
  reference_review:
    relevance: LOW
    correctness: VERIFIED
    review_notes: Primary unconventional secretion paper supporting 
      extracellular/secreted tau context through leaderless cargo secretion, not
      the core tau function.
- id: PMID:32814053
  title: Interactome Mapping Provides a Network of Neurodegenerative Disease 
    Proteins and Uncovers Widespread Protein Aggregation in Affected Brains.
  findings: []
- id: PMID:33961781
  title: Dual proteome-scale networks reveal cell-specific remodeling of the 
    human interactome.
  findings: []
- id: PMID:34757590
  title: Targeting Tau Mitigates Mitochondrial Fragmentation and Oxidative 
    Stress in Amyotrophic Lateral Sclerosis.
  findings: []
- id: PMID:7566652
  title: ApoE3 binding to tau tandem repeat I is abolished by tau serine262 
    phosphorylation.
  findings: []
- id: PMID:7706316
  title: Microtubule-associated protein/microtubule affinity-regulating kinase 
    (p110mark). A novel protein kinase that regulates tau-microtubule 
    interactions and dynamic instability by phosphorylation at the 
    Alzheimer-specific site serine 262.
  findings: []
  reference_review:
    relevance: HIGH
    correctness: VERIFIED
    review_notes: Primary study supporting phosphorylation-dependent reduction 
      of tau microtubule binding and microtubule dynamic instability.
- id: PMID:7929085
  title: Structural studies of tau protein and Alzheimer paired helical 
    filaments show no evidence for beta-structure.
  findings: []
- id: PMID:7972031
  title: 'Isoform-specific interactions of apolipoprotein E with microtubule-associated
    protein tau: implications for Alzheimer disease.'
  findings: []
- id: PMID:8522593
  title: Interaction of tau with the neural plasma membrane mediated by tau's 
    amino-terminal projection domain.
  findings: []
- id: PMID:8642405
  title: Tau is enriched on dynamic microtubules in the distal region of growing
    axons.
  findings: []
- id: PMID:9689133
  title: Presenilin 1 associates with glycogen synthase kinase-3beta and its 
    substrate tau.
  findings: []
- id: PMID:9736630
  title: Tau cleavage and dephosphorylation in cerebellar granule neurons 
    undergoing apoptosis.
  findings: []
- id: PMID:9763511
  title: Tau interacts with src-family non-receptor tyrosine kinases.
  findings: []
- id: Reactome:R-HSA-201629
  title: Caspase-mediated cleavage of Tau
  findings: []
- id: Reactome:R-HSA-9836404
  title: p-PKR dimer phosphorylates MAPT
  findings: []
- id: Reactome:R-NUL-9619534
  title: AMPK phosphorylates Mapt
  findings: []
- id: Reactome:R-NUL-9619539
  title: AMPK-negatively regulates binding of MAPT to microtubules
  findings: []
- id: file:human/MAPT/MAPT-uniprot.txt
  title: UniProt text export for MAPT (P10636)
  findings: []
  reference_review:
    relevance: HIGH
    correctness: VERIFIED
    review_notes: Local UniProt record directly summarizes tau microtubule 
      assembly/stability, neuronal polarity, axonal microtubule binding, 
      membrane-component binding, subcellular localization, and tubulin-repeat 
      domain biology.
- id: file:human/MAPT/MAPT-notes.md
  title: Manual MAPT review notes
  findings: []
  reference_review:
    relevance: MEDIUM
    correctness: VERIFIED
    review_notes: Manual notes document the deep-research timeout and summarize 
      the cached UniProt, PANTHER, GOA, and publication evidence used for this 
      review.
core_functions:
- description: 'Neuronal microtubule-associated protein activity: tau binds tubulin
    and microtubules, stabilizes microtubules, promotes microtubule assembly, supports
    axonal microtubule organization, axonal transport, neurite/axon development, and
    links axonal microtubules to neural membrane-cortex components.'
  supported_by:
  - reference_id: file:human/MAPT/MAPT-uniprot.txt
    supporting_text: Promotes microtubule assembly and stability
  - reference_id: file:human/MAPT/MAPT-uniprot.txt
    supporting_text: The C-terminus binds axonal microtubules
  - reference_id: file:human/MAPT/MAPT-uniprot.txt
    supporting_text: N-terminus binds neural plasma membrane components
  - reference_id: PMID:15147841
    supporting_text: Tau is a neuronal, microtubule-associated protein
  - reference_id: PMID:15147841
    supporting_text: bind exclusively to the outside of microtubules
  molecular_function:
    id: GO:0140778
    label: microtubule stabilizing activity
  directly_involved_in:
  - id: GO:0000226
    label: microtubule cytoskeleton organization
  - id: GO:0031116
    label: positive regulation of microtubule polymerization
  - id: GO:0031175
    label: neuron projection development
  - id: GO:0098930
    label: axonal transport
  locations:
  - id: GO:0015630
    label: microtubule cytoskeleton
  - id: GO:0030424
    label: axon
  - id: GO:0005829
    label: cytosol
  - id: GO:0005886
    label: plasma membrane
- description: Disease-relevant tau self-association and pathological 
    filament/tangle assembly, in which modified tau proteins polymerize into 
    supramolecular fibers, amyloid-like paired helical filaments, and Alzheimer 
    neurofibrillary tangles.
  supported_by:
  - reference_id: PMID:2484340
    supporting_text: multiple tau protein isoforms are incorporated into
  - reference_id: PMID:2484340
    supporting_text: neurofibrillary tangles of Alzheimer's disease
  - reference_id: PMID:26363795
    supporting_text: neurofibrillary tangles (NFTs)
  - reference_id: PMID:28386764
    supporting_text: aberrant assembly of tau into insoluble
  molecular_function:
    id: GO:0042802
    label: identical protein binding
  directly_involved_in:
  - id: GO:1990000
    label: amyloid fibril formation
  - id: GO:0097435
    label: supramolecular fiber organization
  - id: GO:1902988
    label: neurofibrillary tangle assembly
  locations:
  - id: GO:0097418
    label: neurofibrillary tangle
proposed_new_terms: []
suggested_questions:
- question: Which tau annotations should be treated as normal neuronal 
    microtubule/axon biology versus pathological tauopathy gain-of-function or 
    mislocalization?
- question: Which kinase-, phosphatase-, chaperone-, lipid-, and 
    APOE-interaction annotations are reproducible endogenous tau functions 
    rather than context-specific or high-throughput interactions?
- question: For Alzheimer-relevant pathway modules, should extracellular tau 
    secretion/spread, synaptic toxicity, mitochondrial dysfunction, and glial 
    activation be curated as MAPT core functions or as downstream 
    disease-context consequences?
suggested_experiments:
- hypothesis: Tau phosphorylation state determines the balance between axonal 
    microtubule stabilization, neural membrane-cortex association, and 
    somatodendritic mislocalization.
  description: Use endogenous MAPT isoform and phosphosite editing in human 
    iPSC-derived neurons, followed by live imaging of microtubule dynamics, 
    axonal transport, membrane-cortex association, and somatodendritic tau 
    accumulation under amyloid and kinase-activation stressors.
  experiment_type: endogenous phosphosite/isoform perturbation assay
- hypothesis: Pathological tau self-assembly is separable from normal 
    microtubule-stabilizing activity and drives downstream synaptic and 
    mitochondrial phenotypes.
  description: Compare wild-type tau with aggregation-defective and 
    microtubule-binding-defective MAPT variants in neuronal cultures, measuring 
    tau filament formation, neurofibrillary-tangle markers, synapse 
    localization/function, mitochondrial transport, and neuron viability.
  experiment_type: structure-function tauopathy model