PIK3R1

UniProt ID: P27986
Organism: Homo sapiens
Review Status: COMPLETE
πŸ“ Provide Detailed Feedback

Gene Description

PIK3R1 encodes p85Ξ± (and, via alternative promoters/splicing, the shorter p55Ξ± and p50Ξ±), the regulatory subunit of class IA phosphatidylinositol 3-kinase (PI3K). p85Ξ± is a non-catalytic, multidomain adaptor/regulatory protein (SH3, proline-rich, BCR/RhoGAP-homology (BH), and nSH2–iSH2–cSH2 modules). Its inter-SH2 coiled coil binds and stabilizes the otherwise-unstable p110 catalytic subunit (PIK3CA/PIK3CB/PIK3CD), forming an obligate heterodimer, while its SH2 domains restrain basal lipid-kinase activity. Upon growth-factor/insulin stimulation, the two SH2 domains dock tyrosine-phosphorylated YXXM motifs on activated receptor tyrosine kinases and on adaptor/scaffold proteins such as IRS1/2, thereby relieving autoinhibition and recruiting the heterodimer to the cytoplasmic face of receptor-bearing membranes; there the associated p110 phosphorylates PI(4,5)P2 to PI(3,4,5)P3, initiating AKT-centered signaling that controls growth, survival, proliferation, metabolism (including insulin-stimulated glucose uptake), cytoskeletal remodeling and vesicular trafficking. p85Ξ± is chiefly cytosolic at rest and is recruited to membranes on activation; it also has p110-independent roles, including modulation of the ER-stress/unfolded protein response through interaction with XBP1. Human loss-of-function or dominant variants cause SHORT syndrome, agammaglobulinemia and immunodeficiency with lymphoproliferation, and somatic variants occur in cancers and vascular malformations.

Existing Annotations Review

GO Term Evidence Action Reason
GO:0000209 protein polyubiquitination
IDA
PMID:27708159
Insulin resistance and diabetes caused by genetic or diet-in...
KEEP AS NON CORE
Summary: p85Ξ± undergoes KBTBD2/Cullin-3-mediated polyubiquitination controlling PI3K abundance in adipocytes.
Reason: p85Ξ± is the SUBSTRATE of this polyubiquitination (recognized by the BTB-Kelch protein KBTBD2 of a Cullin-3 E3 ligase), not the enzyme performing it. This is a regulated event governing p85Ξ± levels and insulin signaling rather than a core molecular function of p85Ξ±; retained as non-core with the GOA-supplied qualifier.
Supporting Evidence:
PMID:27708159
KBTBD2 targeted p85Ξ±, the regulatory subunit
PMID:27708159
causing p85Ξ± ubiquitination
GO:0001678 intracellular glucose homeostasis
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Role in intracellular glucose homeostasis via insulin/PI3K signaling.
Reason: p85Ξ± contributes to glucose handling as the regulatory subunit coupling the insulin receptor/IRS to PI3K-AKT; this is a downstream physiological role rather than the core biochemical function. Electronic annotation from mouse ortholog is biologically reasonable.
GO:0001678 intracellular glucose homeostasis
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Role in intracellular glucose homeostasis (ISS from mouse ortholog).
Reason: p85Ξ± contributes to glucose handling as the regulatory subunit coupling the insulin receptor/IRS to PI3K-AKT; this is a downstream physiological role rather than the core biochemical function. Electronic annotation from mouse ortholog is biologically reasonable.
GO:0001784 phosphotyrosine residue binding
IEA
GO_REF:0000117
ACCEPT
Summary: SH2 domains of p85Ξ± bind phosphotyrosine (pYXXM) motifs – a core adaptor activity.
Reason: p85Ξ± has two SH2 domains that dock tyrosine-phosphorylated YXXM motifs on activated receptors and adaptor/scaffold proteins; this is a well-established core molecular function and the basis of its receptor-coupling role.
GO:0001784 phosphotyrosine residue binding
IPI
PMID:20624904
Tarp regulates early Chlamydia-induced host cell survival th...
ACCEPT
Summary: p85Ξ± SH2 phosphotyrosine binding confirmed on an SH2/PTB-domain microarray.
Reason: Supports the core phosphotyrosine-binding activity of p85Ξ±'s SH2 domains, assayed here on a comprehensive SH2/PTB-domain protein microarray.
Supporting Evidence:
PMID:20624904
virtually all human SRC homology 2 (SH2) and phosphotyrosine binding domains
GO:0005068 transmembrane receptor protein tyrosine kinase adaptor activity
ISS
GO_REF:0000024
ACCEPT
Summary: Adaptor that couples activated receptor tyrosine kinases to the p110 catalytic subunit.
Reason: Captures p85Ξ±'s core adaptor role: its SH2 domains bind phospho-RTKs and its iSH2 recruits p110, bridging receptors to PI3K. ISS from the mouse ortholog is consistent with extensive human data.
GO:0005158 insulin receptor binding
IPI
PMID:7537849
Phosphotyrosine-dependent interaction of SHC and insulin rec...
KEEP AS NON CORE
Summary: p85Ξ± SH2 domains bind the tyrosine-phosphorylated insulin receptor.
Reason: p85Ξ± binds phosphorylated INSR (via its SH2 domains at the YTHM/NPEY region; UniProt SUBUNIT), an instance of its general phosphotyrosine-binding/adaptor activity toward a specific receptor. Retained as a specific, non-core facet of the core SH2 function.
GO:0005159 insulin-like growth factor receptor binding
IPI
PMID:7541045
Non-SH2 domains within insulin receptor substrate-1 and SHC ...
KEEP AS NON CORE
Summary: p85Ξ± interacts directly and specifically with the IGF-I receptor.
Reason: Direct SH2-mediated binding of p85Ξ± to IGF1R, a specific instance of its phosphotyrosine-binding adaptor activity toward a receptor tyrosine kinase.
Supporting Evidence:
PMID:7541045
phosphatidylinositol 3-kinase interact directly and specifically with the IGFIR.
GO:0005168 neurotrophin TRKA receptor binding
IPI
PMID:15488758
TrkA alternative splicing: a regulated tumor-promoting switc...
KEEP AS NON CORE
Summary: p85Ξ± links to NTRK1/TrkA signaling (PI3K/Akt) in neuroblastoma.
Reason: Specific receptor-binding facet of p85Ξ±'s SH2 adaptor activity toward TrkA; TrkA/TrkAIII signals through PI3K/Akt. Retained as non-core, receptor-specific.
GO:0005515 protein binding
IPI
PMID:10500481
A fluorescent indicator for tyrosine phosphorylation-based i...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:10572067
Dominance of ErbB-1 heterodimers in lung epithelial cells ov...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:10660596
Tyrosine dephosphorylation and deactivation of insulin recep...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:10752619
Alternative modes of binding of proteins with tandem SH2 dom...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:10811803
Association of Grb2, Gads, and phospholipase C-gamma 1 with ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:10820259
GRID: a novel Grb-2-related adapter protein that interacts w...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:10978177
Identification of major tyrosine phosphorylation sites in th...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:11123912
NMR structure of the N-SH2 of the p85 subunit of phosphoinos...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:11403293
Split luciferase as an optical probe for detecting protein-p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:11416002
Tyr(612) and Tyr(632) in human insulin receptor substrate-1 ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:11710529
Tyrosine phosphorylation-dependent yeast two-hybrid system f...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:11796522
PLC-gamma1 enzyme activity is required for insulin-induced D...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:11896612
Use of signal specific receptor tyrosine kinase oncoproteins...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:1314164
Phosphorylation sites in the PDGF receptor with different sp...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:1330535
Interaction of the p85 subunit of PI 3-kinase and its N-term...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:1380456
Phosphatidylinositol 3'-kinase is activated by association w...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:1382595
Inhibition of SH2 domain/phosphoprotein association by a non...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:14632132
Locating a protein-protein interaction in living cells via s...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16043515
The p85 regulatory subunit of phosphoinositide 3-kinase down...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16230374
Insulin receptor substrate is a mediator of phosphoinositide...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16273093
A quantitative protein interaction network for the ErbB rece...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16273093
A quantitative protein interaction network for the ErbB rece...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16273093
A quantitative protein interaction network for the ErbB rece...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16638574
Reduced phosphatase activity of SHP-2 in LEOPARD syndrome: c...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16843263
HER2 kinase domain mutation results in constitutive phosphor...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16858407
Toll-like receptor 3 associates with c-Src tyrosine kinase o...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16917505
Direct binding of p85 to sst2 somatostatin receptor reveals ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16982329
Constitutive c-jun N-terminal kinase activity in acute myelo...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16982329
Constitutive c-jun N-terminal kinase activity in acute myelo...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16982329
Constitutive c-jun N-terminal kinase activity in acute myelo...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:16982329
Constitutive c-jun N-terminal kinase activity in acute myelo...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17474147
Systematic identification of SH3 domain-mediated human prote...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:17500595
Huntingtin interacting proteins are genetic modifiers of neu...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:18059340
Interaction with PI3-kinase contributes to the cytotoxic act...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:18079394
The structure of a human p110alpha/p85alpha complex elucidat...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:18412956
Myeloproliferative disorder FOP-FGFR1 fusion kinase recruits...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:18641334
ICOS ligation recruits the p50alpha PI3K regulatory subunit ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:18641334
ICOS ligation recruits the p50alpha PI3K regulatory subunit ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19380743
Charting the molecular network of the drug target Bcr-Abl.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19380743
Charting the molecular network of the drug target Bcr-Abl.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19574958
Mal connects TLR2 to PI3Kinase activation and phagocyte pola...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19574958
Mal connects TLR2 to PI3Kinase activation and phagocyte pola...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19805105
A frequent kinase domain mutation that changes the interacti...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19807924
Identification of SH3 domain interaction partners of human F...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19864249
High content screening for inhibitors of protein interaction...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:19903481
Involvement of Src tyrosine kinase in Escherichia coli invas...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20007781
Specific apoptosis induction by the dual PI3K/mTor inhibitor...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20018188
C-mip interacts with the p85 subunit of PI3 kinase and exert...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20018188
C-mip interacts with the p85 subunit of PI3 kinase and exert...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20227043
An activated ErbB3/NRG1 autocrine loop supports in vivo prol...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20541701
NSAID sulindac and its analog bind RXRalpha and inhibit RXRa...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20598684
Abi1/Hssh3bp1 pY213 links Abl kinase signaling to p85 regula...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20711237
LAPTM4B: a novel cancer-associated gene motivates multidrug ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20713702
Cancer-derived mutations in the regulatory subunit p85alpha ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20713702
Cancer-derived mutations in the regulatory subunit p85alpha ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:20936779
A human MAP kinase interactome.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21075308
Recombinant human erythropoietin antagonizes trastuzumab tre...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21075308
Recombinant human erythropoietin antagonizes trastuzumab tre...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21241768
Phosphoinositide 3-kinase as a novel functional target for t...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21241768
Phosphoinositide 3-kinase as a novel functional target for t...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21278786
PI3K inhibition results in enhanced HER signaling and acquir...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21706016
Selected reaction monitoring mass spectrometry reveals the d...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21827948
Dynamics of the phosphoinositide 3-kinase p110Ξ΄ interaction ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21946561
NYAP: a phosphoprotein family that links PI3K to WAVE1 signa...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21946561
NYAP: a phosphoprotein family that links PI3K to WAVE1 signa...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:21954290
Tyrosine phosphorylation of the GΞ±-interacting protein GIV p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:22020336
p37Ξ΄ is a new isoform of PI3K p110Ξ΄ that increases cell prol...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:22402981
Integrin/Fak/Src-mediated regulation of cell survival and an...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:22439932
NKX2-1/TITF1/TTF-1-Induced ROR1 is required to sustain EGFR ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:22810585
Viral immune modulators perturb the human molecular network ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23065768
Activation of rapid oestrogen signalling in aggressive human...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23246379
FLT3 signals via the adapter protein Grb10 and overexpressio...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23275563
Development and application of a DNA microarray-based yeast ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23397142
Analysis of protein-protein interactions in cross-talk pathw...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23397142
Analysis of protein-protein interactions in cross-talk pathw...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23604317
FBXL2- and PTPL1-mediated degradation of p110-free p85Ξ² regu...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23617393
Insulin receptor substrate-2 is expressed in kidney epitheli...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23643389
Gain of interaction with IRS1 by p110Ξ±-helical domain mutant...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23643389
Gain of interaction with IRS1 by p110Ξ±-helical domain mutant...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23676467
FAM83B-mediated activation of PI3K/AKT and MAPK signaling co...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:23853584
The interactomes of influenza virus NS1 and NS2 proteins ide...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24165795
Dominant-activating germline mutations in the gene encoding ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24189400
Perturbation of the mutated EGFR interactome identifies vuln...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24498420
JMJD6 regulates ERΞ± methylation on arginine.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24728074
Enhanced prediction of Src homology 2 (SH2) domain binding p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24728074
Enhanced prediction of Src homology 2 (SH2) domain binding p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24728074
Enhanced prediction of Src homology 2 (SH2) domain binding p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:24728074
Enhanced prediction of Src homology 2 (SH2) domain binding p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25241761
Using an in situ proximity ligation assay to systematically ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25253337
Hijacking Dlg1 for oncogenic phosphatidylinositol 3-kinase a...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25284480
Naturally occurring neomorphic PIK3R1 mutations activate the...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25416956
A proteome-scale map of the human interactome network.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:25416956
A proteome-scale map of the human interactome network.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:26210919
Phosphorylation of serine 523 on 5-lipoxygenase in human B l...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:26210919
Phosphorylation of serine 523 on 5-lipoxygenase in human B l...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:26496610
A human interactome in three quantitative dimensions organiz...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:26496610
A human interactome in three quantitative dimensions organiz...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:26496610
A human interactome in three quantitative dimensions organiz...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:26496610
A human interactome in three quantitative dimensions organiz...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:27135603
A TRAF-like motif of the inducible costimulator ICOS control...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:27478041
Salt-Inducible Kinase 2 Couples Ovarian Cancer Cell Metaboli...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:28169297
Comparative influenza protein interactomes identify the role...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:31031754
Case Study: Mechanism for Increased Follicular Helper T Cell...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:31820037
Elucidation of protein interactions necessary for the mainte...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:32606397
PI3K activation is enhanced by FOXM1D binding to p110 and p8...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:32707033
Kinase Interaction Network Expands Functional and Disease Ro...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:33961781
Dual proteome-scale networks reveal cell-specific remodeling...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:33961781
Dual proteome-scale networks reveal cell-specific remodeling...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:33961781
Dual proteome-scale networks reveal cell-specific remodeling...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:34591612
A protein interaction landscape of breast cancer.
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:34591642
A protein network map of head and neck cancer reveals PIK3CA...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:34606829
Mapping the Phospho-dependent ALK Interactome to Identify No...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35271311
OpenCell: Endogenous tagging for the cartography of human ce...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35271311
OpenCell: Endogenous tagging for the cartography of human ce...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35271311
OpenCell: Endogenous tagging for the cartography of human ce...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35271311
OpenCell: Endogenous tagging for the cartography of human ce...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35384245
Physical and functional interactome atlas of human receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:35512704
Systematic discovery of mutation-directed neo-protein-protei...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:39572596
Multi-layered proteomics identifies insulin-induced upregula...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7536927
Measurement of the binding of tyrosyl phosphopeptides to SH2...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7537096
Formation of signal transfer complexes between stem cell and...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7568038
p56Lck and p59Fyn regulate CD28 binding to phosphatidylinosi...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7584133
Selective CD28pYMNM mutations implicate phosphatidylinositol...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7589433
Interaction of p85 subunit of PI 3-kinase with insulin and I...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7642582
Localization of the insulin-like growth factor I receptor bi...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7657594
Interaction of the Flt-1 tyrosine kinase receptor with the p...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7680095
SH2 domains exhibit high-affinity binding to tyrosine-phosph...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7680644
A photoaffinity scan maps regions of the p85 SH2 domain invo...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7680644
A photoaffinity scan maps regions of the p85 SH2 domain invo...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7692233
Two signaling molecules share a phosphotyrosine-containing b...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7782332
Growth hormone, interferon-gamma, and leukemia inhibitory fa...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7797556
Src phosphorylation of the epidermal growth factor receptor ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7807015
CTLA-4 binding to the lipid kinase phosphatidylinositol 3-ki...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:7807015
CTLA-4 binding to the lipid kinase phosphatidylinositol 3-ki...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8146197
T-cell antigen CD28 interacts with the lipid kinase phosphat...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8183372
Binding of phosphatidylinositol-3-OH kinase to CD28 is requi...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8226808
Identification of Trk binding sites for SHC and phosphatidyl...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8276809
Direct activation of the phosphatidylinositol 3'-kinase by t...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8382612
Specific phosphopeptide binding regulates a conformational c...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8382612
Specific phosphopeptide binding regulates a conformational c...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8564419
Structure-activity studies of phosphorylated peptide inhibit...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8603569
Interaction of the molecular weight 85K regulatory subunit o...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8670861
Structure of a specific peptide complex of the carboxy-termi...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:8961927
Structural and thermodynamic characterization of the interac...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9153411
Dual specificity of Src homology 2 domains for phosphotyrosi...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9160881
Yeast two-hybrid in vivo association of the Src kinase Lyn w...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9178760
Intracellular signaling of the Ufo/Axl receptor tyrosine kin...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9223670
The phosphatidylinositol 3' kinase pathway is required for t...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9398332
Interaction of the cytoplasmic tail of CTLA-4 (CD152) with a...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9417079
Growth factor receptor-bound protein 2 SH2/SH3 domain bindin...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9593725
Association of the insulin receptor with phospholipase C-gam...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9658397
Determination of Gab1 (Grb2-associated binder-1) interaction...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9813138
Resting lymphocyte kinase (Rlk/Txk) phosphorylates the YVKM ...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005515 protein binding
IPI
PMID:9890970
Fyn associates with Cbl and phosphorylates tyrosine 731 in C...
REMOVE
Summary: Interaction annotation captured only as the generic term 'protein binding' (GO:0005515), which conveys no molecular function.
Reason: Per curation policy GO:0005515 is uninformative and is removed. The physical interaction may be genuine, but the generic term adds no functional content and specific interactions are not invented from binding data alone. p85Ξ±'s informative molecular functions are captured by dedicated terms: PI3K regulator activity (GO:0046935), phosphotyrosine residue binding (GO:0001784), RTK adaptor activity (GO:0005068) and class IA PI3K complex membership (GO:0005943).
GO:0005634 nucleus
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Reported nuclear pool of p85Ξ±.
Reason: A nuclear/nucleocytoplasmic pool of p85Ξ± has been reported (e.g. in ER-stress/XBP1 signaling), but the resting protein is predominantly cytoplasmic; retained as non-core. Electronic annotation from mouse.
GO:0005634 nucleus
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Reported nuclear pool of p85Ξ± (ISS from mouse ortholog).
Reason: A nuclear/nucleocytoplasmic pool of p85Ξ± has been reported (e.g. in ER-stress/XBP1 signaling), but the resting protein is predominantly cytoplasmic; retained as non-core. Electronic annotation from mouse.
GO:0005737 cytoplasm
IDA
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
ACCEPT
Summary: Cytoplasmic localization of p85Ξ±.
Reason: Consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm); p85Ξ±/PI3K is a soluble cytoplasmic protein before membrane recruitment.
GO:0005737 cytoplasm
IEA
GO_REF:0000120
ACCEPT
Summary: Cytoplasmic localization of p85Ξ± (electronic).
Reason: Consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm); p85Ξ±/PI3K is a soluble cytoplasmic protein before membrane recruitment.
GO:0005801 cis-Golgi network
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: cis-Golgi network localization (electronic, mouse ortholog).
Reason: Low-specificity electronic annotation from the mouse ortholog; not a well-characterized functional site for p85Ξ±. Retained as non-core.
GO:0005829 cytosol
IEA
GO_REF:0000120
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-109699
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-114542
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1226012
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1226014
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1250189
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1250346
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1250353
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1250370
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1306965
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1306979
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1433514
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1562641
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1676048
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1676109
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-177927
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-177931
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-177939
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1839078
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1839080
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1839091
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1839102
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1839107
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-1839114
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-186780
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-186800
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-198266
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-198315
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-201510
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-201515
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-202203
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-202365
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2029271
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2029273
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-204798
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-205262
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2316434
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2400009
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2424480
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2424482
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2730842
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-2730870
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-388830
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-388832
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-389158
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-416358
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-437118
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-437162
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-443402
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-508247
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5218819
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5357479
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5637765
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5637801
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654591
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654592
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654594
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654596
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654612
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654614
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654620
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654622
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654637
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654640
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654641
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654643
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654659
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654662
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654667
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654669
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654690
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654692
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654697
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654701
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654705
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654709
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654714
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5654717
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655235
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655240
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655245
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655248
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655252
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655263
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655285
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655289
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655290
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655315
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655320
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-5655323
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-6790041
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-74737
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-879917
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-8851954
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-8852019
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-8854905
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9012657
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9013145
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9014294
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9018766
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9021627
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9021660
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9027275
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-912627
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-914182
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9606887
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9632412
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9658253
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9664646
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9664664
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9664933
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9664940
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9665407
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9665415
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9670431
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9670433
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9672162
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9672172
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9672177
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9672178
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9698170
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9698174
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9703434
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9706340
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9706345
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9712078
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9712083
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9712084
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9842649
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005829 cytosol
TAS
Reactome:R-HSA-9845032
ACCEPT
Summary: Cytosolic localization of p85Ξ± / the class IA PI3K holoenzyme (Reactome TAS).
Reason: The p85α–p110 complex is largely soluble/cytosolic before stimulation, consistent with UniProt SUBCELLULAR LOCATION (Cytoplasm). Duplicate Reactome pathway-level rows are retained; duplicates across evidence are acceptable.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-1250189
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-1250462
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-1306957
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-1306965
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-2045911
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-2076220
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-2316434
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-2394007
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-2400009
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005886 plasma membrane
TAS
Reactome:R-HSA-9606887
ACCEPT
Summary: Plasma membrane localization of the PI3K complex (Reactome TAS).
Reason: Upon receptor activation the p85α–p110 heterodimer is recruited to the cytoplasmic face of the plasma membrane, where p110 accesses PI(4,5)P2; this is where the complex carries out its signaling function. Duplicate Reactome rows retained.
GO:0005911 cell-cell junction
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Cell-cell junction localization (electronic, mouse ortholog).
Reason: Peripheral/low-specificity electronic localization from the mouse ortholog; not central to p85Ξ± function. Retained as non-core.
GO:0005942 phosphatidylinositol 3-kinase complex
ISS
GO_REF:0000024
ACCEPT
Summary: Part of the phosphatidylinositol 3-kinase complex.
Reason: p85Ξ± is an obligate subunit of class IA PI3K; this parent complex term is correct. Complex membership is represented in core_functions via in_complex (class IA PI3K complex).
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
IBA
GO_REF:0000033
ACCEPT
Summary: Subunit of the class IA PI3K complex (phylogenetic/IBD inference).
Reason: Core complex membership: p85Ξ± is the regulatory subunit of the class IA PI3K heterodimer. The IBA reflects a well-supported ancestral assignment across the p85 family (PANTHER:PTN008302071), with the target itself among the descendant evidences.
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
IEA
GO_REF:0000117
ACCEPT
Summary: Subunit of the class IA PI3K complex (electronic).
Reason: Correct complex membership; consistent with all direct structural evidence.
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
IPI
PMID:19805105
A frequent kinase domain mutation that changes the interacti...
ACCEPT
Summary: p85Ξ± forms the class IA PI3K complex with p110 (structural/biochemical).
Reason: Direct evidence: p85Ξ± (nSH2/iSH2) assembles with the p110Ξ± catalytic subunit into the class IA PI3K complex; the nSH2 forms a scaffold for the whole enzyme.
Supporting Evidence:
PMID:19805105
domain of p85alpha is shown to form a scaffold for the entire enzyme complex
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
IPI
PMID:20713702
Cancer-derived mutations in the regulatory subunit p85alpha ...
ACCEPT
Summary: p85Ξ± binds p110Ξ±/p110Ξ² within the class IA PI3K complex (cancer-mutant study).
Reason: Confirms p85α–p110 complex formation; even oncogenic p85Ξ± mutants retain binding to the catalytic subunits while weakening inhibition and preserving stabilization.
Supporting Evidence:
PMID:20713702
The mutant proteins are still able to bind to the catalytic
PMID:20713702
preserving the stabilizing interaction between p85Ξ± iSH2 and the adapter-binding domain of p110Ξ±.
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
IPI
PMID:28108251
Discovery of 7-(3-(piperazin-1-yl)phenyl)pyrrolo[2,1-f][1,2,...
ACCEPT
Summary: p85Ξ± in the class IA PI3K (p110Ξ΄) complex.
Reason: Direct-interaction evidence for class IA PI3K complex membership (p110Ξ΄ context).
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
ISS
GO_REF:0000024
ACCEPT
Summary: Class IA PI3K complex membership (ISS).
Reason: Correct complex membership by sequence similarity to an ortholog; consistent with all evidence.
GO:0005943 phosphatidylinositol 3-kinase complex, class IA
NAS
P27986-3
PMID:20379207
The emerging mechanisms of isoform-specific PI3K signalling.
ACCEPT
Summary: p50Ξ± (isoform 3) as a class IA PI3K regulatory subunit.
Reason: Isoform-specific (P27986-3 / p50Ξ±) membership of the class IA PI3K complex; the shorter regulatory isoforms also heterodimerize with p110. Retained.
GO:0006955 immune response
NAS
PMID:27616589
PI3KΞ΄ and primary immunodeficiencies.
KEEP AS NON CORE
Summary: Involvement in immune response (narrative).
Reason: Broad immune-response role reflecting p85Ξ±'s function in lymphocyte PI3K signaling (loss causes agammaglobulinemia/immunodeficiency). Pleiotropic/downstream; retained as non-core.
GO:0007165 signal transduction
IEA
GO_REF:0000002
KEEP AS NON CORE
Summary: Generic signal transduction (from InterPro SH2 domain).
Reason: Very general BP inferred from the SH2 domain (InterPro:IPR000198). Correct but uninformative given the specific pathway terms present (PI3K/AKT, insulin receptor signaling); retained as non-core.
GO:0008286 insulin receptor signaling pathway
IBA
GO_REF:0000033
ACCEPT
Summary: Insulin receptor signaling pathway (phylogenetic inference).
Reason: Core signaling role: p85Ξ± couples the tyrosine-phosphorylated insulin receptor/IRS to PI3K activation. Well-supported IBA across the p85 family and orthologs.
GO:0008286 insulin receptor signaling pathway
IEA
GO_REF:0000107
ACCEPT
Summary: Insulin receptor signaling pathway (electronic).
Reason: Consistent with the IBA and extensive experimental literature on insulin/IRS/PI3K signaling.
GO:0010592 positive regulation of lamellipodium assembly
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Positive regulation of lamellipodium assembly (electronic, mouse).
Reason: Cytoskeletal role linked to p85Ξ±'s BH domain / Rho-family GTPase interactions and PI3K-driven actin remodeling. Pleiotropic; retained as non-core.
GO:0010592 positive regulation of lamellipodium assembly
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Positive regulation of lamellipodium assembly (ISS, mouse).
Reason: Cytoskeletal role linked to p85Ξ±'s BH domain / Rho-family GTPase interactions and PI3K-driven actin remodeling. Pleiotropic; retained as non-core.
GO:0016020 membrane
HDA
PMID:19946888
Defining the membrane proteome of NK cells.
KEEP AS NON CORE
Summary: Membrane association (high-throughput).
Reason: Generic membrane localization; functionally p85Ξ± is recruited to receptor-bearing membranes, captured more specifically by plasma membrane. Retained as non-core.
GO:0016020 membrane
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Membrane localization (electronic, mouse).
Reason: Generic membrane localization; functionally p85Ξ± is recruited to receptor-bearing membranes, captured more specifically by plasma membrane. Retained as non-core.
GO:0019207 kinase regulator activity
IEA
GO_REF:0000117
MODIFY
Summary: Kinase regulator activity – generalizes to PI3K regulator activity.
Reason: The specific and evidence-backed activity is regulation of the PI3-kinase (p110) catalytic subunit; the generic 'kinase regulator activity' should be replaced by the specific PI3K regulator term.
GO:0019209 kinase activator activity
IEA
GO_REF:0000107
MODIFY
Summary: Kinase activator activity – specifically PI3K activation.
Reason: p85Ξ± enables receptor-driven activation of PI3K (while also restraining basal activity); the generic 'kinase activator activity' should be the PI3K-specific activator term.
GO:0019221 cytokine-mediated signaling pathway
IGI
PMID:7782332
Growth hormone, interferon-gamma, and leukemia inhibitory fa...
KEEP AS NON CORE
Summary: Cytokine-mediated signaling (growth hormone) via IRS/PI3K.
Reason: GH/cytokine receptors signal through JAK2β†’IRS-1 tyrosine phosphorylation, recruiting p85Ξ±/PI3K. Downstream pathway role; retained as non-core.
Supporting Evidence:
PMID:7782332
binding of IRS-1 to the 85-kDa regulatory subunit of PI 3'-kinase.
GO:0019903 protein phosphatase binding
IEA
GO_REF:0000117
KEEP AS NON CORE
Summary: Protein phosphatase binding (electronic).
Reason: p85Ξ± interacts with protein phosphatases (e.g. PTPRJ/CD148 which dephosphorylates it, and PTEN). A specific binding facet; retained as non-core.
GO:0019903 protein phosphatase binding
IPI
PMID:14699157
Human homolog of disc-large is required for adherens junctio...
KEEP AS NON CORE
Summary: Protein phosphatase binding (direct interaction).
Reason: Direct-interaction support for p85Ξ± binding a protein phosphatase; a specific binding facet, retained as non-core rather than a core evolved function.
GO:0030183 B cell differentiation
NAS
PMID:20200404
The catalytic PI3K isoforms p110gamma and p110delta contribu...
KEEP AS NON CORE
Summary: B cell differentiation.
Reason: p85Ξ± is required for B-cell development (biallelic loss causes agammaglobulinemia, AGM7). A physiological/developmental consequence of its PI3K-signaling role; retained as non-core.
GO:0030217 T cell differentiation
NAS
PMID:17371229
The PI3K p110delta controls T-cell development, differentiat...
KEEP AS NON CORE
Summary: T cell differentiation.
Reason: p85Ξ±/PI3K signaling contributes to T-cell development and function (CD28/ICOS costimulation). Developmental/pleiotropic; retained as non-core.
GO:0032869 cellular response to insulin stimulus
IDA
PMID:27708159
Insulin resistance and diabetes caused by genetic or diet-in...
ACCEPT
Summary: Cellular response to insulin stimulus.
Reason: Core physiological context: p85Ξ± mediates the cellular insulin response by coupling INSR/IRS to PI3K. Supported by the KBTBD2 study linking p85Ξ± abundance to insulin signaling.
Supporting Evidence:
PMID:27708159
KBTBD2 targeted p85Ξ±, the regulatory subunit
GO:0032869 cellular response to insulin stimulus
IEA
GO_REF:0000120
ACCEPT
Summary: Cellular response to insulin stimulus (electronic).
Reason: Consistent with p85Ξ±'s central role in insulin/IRS/PI3K signaling.
GO:0032869 cellular response to insulin stimulus
ISS
GO_REF:0000024
ACCEPT
Summary: Cellular response to insulin stimulus (ISS).
Reason: Consistent with p85Ξ±'s central role in insulin/IRS/PI3K signaling.
GO:0033120 positive regulation of RNA splicing
IMP
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Positive regulation of RNA splicing via the IRE1/XBP1 UPR arm.
Reason: Through its ER-stress-dependent interaction with XBP1 and modulation of IRE1Ξ±, p85Ξ± influences the UPR (which includes XBP1 mRNA splicing). A specialized, context-specific role; retained as non-core.
Supporting Evidence:
PMID:20348923
transcriptional mediator of the unfolded protein response (UPR), in an
GO:0034143 regulation of toll-like receptor 4 signaling pathway
IDA
PMID:19289601
TLR4/MyD88/PI3K interactions regulate TLR4 signaling.
KEEP AS NON CORE
Summary: Regulation of TLR4 signaling via MyD88/PI3K.
Reason: p85Ξ± co-immunoprecipitates with MyD88 (YXXM motif) and modulates TLR4/PI3K signaling. A specific immune-signaling role; retained as non-core.
Supporting Evidence:
PMID:19289601
MyD88 and p85 were shown previously to
GO:0034446 substrate adhesion-dependent cell spreading
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Substrate adhesion-dependent cell spreading (ISS).
Reason: PI3K/p85Ξ± signaling contributes to adhesion-dependent spreading and cytoskeletal dynamics; pleiotropic. Retained as non-core.
GO:0034976 response to endoplasmic reticulum stress
IDA
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Response to endoplasmic reticulum stress (XBP1/UPR).
Reason: p85Ξ± modulates the UPR by interacting with XBP1 in an ER-stress-dependent manner; loss reduces nuclear XBP1 and UPR target-gene induction. A specialized role; retained as non-core.
Supporting Evidence:
PMID:20348923
ER stress-dependent accumulation of nuclear XBP-1, decreased induction of UPR
GO:0034976 response to endoplasmic reticulum stress
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Response to ER stress (electronic).
Reason: p85Ξ± modulates the UPR by interacting with XBP1 in an ER-stress-dependent manner; loss reduces nuclear XBP1 and UPR target-gene induction. A specialized role; retained as non-core.
GO:0034976 response to endoplasmic reticulum stress
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Response to ER stress (ISS).
Reason: p85Ξ± modulates the UPR by interacting with XBP1 in an ER-stress-dependent manner; loss reduces nuclear XBP1 and UPR target-gene induction. A specialized role; retained as non-core.
GO:0035014 phosphatidylinositol 3-kinase regulator activity
IDA
PMID:27708159
Insulin resistance and diabetes caused by genetic or diet-in...
ACCEPT
Summary: Phosphatidylinositol 3-kinase regulator activity – core function.
Reason: Core molecular function: p85Ξ± is the regulatory subunit of PI3K, stabilizing p110 and restraining/enabling its activity. Directly supported by the KBTBD2 study.
Supporting Evidence:
PMID:27708159
KBTBD2 targeted p85Ξ±, the regulatory subunit
PMID:27708159
causing p85Ξ± ubiquitination
GO:0035014 phosphatidylinositol 3-kinase regulator activity
ISS
GO_REF:0000024
ACCEPT
Summary: Phosphatidylinositol 3-kinase regulator activity (ISS).
Reason: Core regulator activity, supported by sequence similarity to an ortholog and abundant human data.
GO:0035655 interleukin-18-mediated signaling pathway
IMP
PMID:21321938
Interleukin-18/WNT1-inducible signaling pathway protein-1 si...
KEEP AS NON CORE
Summary: IL-18-mediated signaling via PI3K/Akt.
Reason: IL-18 signaling engages PI3K/Akt (with WISP1) to drive vascular smooth-muscle proliferation; p85Ξ± acts as the PI3K regulatory subunit in this pathway. Specific/context; retained as non-core.
Supporting Evidence:
PMID:21321938
phosphatidylinositol 3-kinase/Akt-dependent IKK/NF-ΞΊB
GO:0036312 phosphatidylinositol 3-kinase regulatory subunit binding
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Phosphatidylinositol 3-kinase regulatory subunit binding (electronic).
Reason: p85Ξ± can associate with regulatory subunits (homo/hetero-association, e.g. with PIK3R2); a specific binding facet. Retained as non-core.
GO:0042267 natural killer cell mediated cytotoxicity
IDA
PMID:16582911
NKG2D-mediated signaling requires a DAP10-bound Grb2-Vav1 in...
KEEP AS NON CORE
Summary: Natural killer cell mediated cytotoxicity via DAP10/PI3K.
Reason: The p85 subunit binds DAP10 and, together with Grb2-Vav1, is required for full NK-cell cytotoxicity downstream of NKG2D. A specific immune role; retained as non-core.
Supporting Evidence:
PMID:16582911
For full calcium release and cytotoxicity to occur, both Grb2-Vav1 and p85 had
GO:0042307 positive regulation of protein import into nucleus
IDA
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Positive regulation of protein import into nucleus (XBP1).
Reason: p85Ξ± promotes nuclear accumulation/import of XBP1 during the UPR. Specialized role; retained as non-core.
Supporting Evidence:
PMID:20348923
ER stress-dependent accumulation of nuclear XBP-1, decreased induction of UPR
GO:0042307 positive regulation of protein import into nucleus
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Positive regulation of protein import into nucleus (electronic).
Reason: Electronic annotation consistent with the XBP1 nuclear-accumulation role.
GO:0043066 negative regulation of apoptotic process
IEA
GO_REF:0000117
KEEP AS NON CORE
Summary: Negative regulation of apoptotic process (electronic).
Reason: Consistent with p85Ξ±/PI3K-AKT pro-survival signaling and the UPR study (p85Ξ± loss increases apoptosis). Pleiotropic; retained as non-core.
GO:0043066 negative regulation of apoptotic process
IMP
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Negative regulation of apoptotic process (XBP1/UPR, IMP).
Reason: Cells lacking p85Ξ± show increased apoptosis under ER stress, indicating an anti-apoptotic role via the UPR. Retained as non-core.
Supporting Evidence:
PMID:20348923
target genes and increased rates of apoptosis.
GO:0043125 ErbB-3 class receptor binding
IDA
PMID:10572067
Dominance of ErbB-1 heterodimers in lung epithelial cells ov...
KEEP AS NON CORE
Summary: ErbB-3 (ERBB3) class receptor binding.
Reason: p85Ξ± SH2 domains dock the multiple phospho-YXXM motifs of ERBB3, a specific instance of its phosphotyrosine-binding adaptor activity. Retained as non-core.
GO:0043491 phosphatidylinositol 3-kinase/protein kinase B signal transduction
IDA
PMID:7782332
Growth hormone, interferon-gamma, and leukemia inhibitory fa...
ACCEPT
Summary: PI3K/AKT signal transduction – downstream pathway p85Ξ± enables.
Reason: Core downstream pathway: by recruiting/regulating p110, p85Ξ± enables PIP3 production and AKT activation. Growth hormone/IRS-1 engagement of p85Ξ± exemplifies pathway entry.
Supporting Evidence:
PMID:7782332
binding of IRS-1 to the 85-kDa regulatory subunit of PI 3'-kinase.
GO:0043491 phosphatidylinositol 3-kinase/protein kinase B signal transduction
IEA
GO_REF:0000117
ACCEPT
Summary: PI3K/AKT signal transduction (electronic).
Reason: Core downstream pathway of the class IA PI3K complex; consistent with all evidence.
GO:0043491 phosphatidylinositol 3-kinase/protein kinase B signal transduction
IMP
PMID:21321938
Interleukin-18/WNT1-inducible signaling pathway protein-1 si...
ACCEPT
Summary: PI3K/AKT signal transduction (IMP, IL-18 context).
Reason: Functional (IMP) support that p85Ξ±/PI3K drives Akt signaling in an IL-18-stimulated context.
Supporting Evidence:
PMID:21321938
phosphatidylinositol 3-kinase/Akt-dependent IKK/NF-ΞΊB
GO:0043491 phosphatidylinositol 3-kinase/protein kinase B signal transduction
ISS
GO_REF:0000024
ACCEPT
Summary: PI3K/AKT signal transduction (ISS).
Reason: Core downstream pathway; consistent with the IBA and human experimental data.
GO:0043548 phosphatidylinositol 3-kinase binding
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Phosphatidylinositol 3-kinase (p110) binding.
Reason: Direct binding of p85Ξ± (iSH2) to the p110 catalytic subunit underlies the heterodimer; the functional consequence (regulator activity, complex membership) is captured by GO:0046935 and GO:0005943. Retained as a non-core binding facet.
GO:0043559 insulin binding
IDA
PMID:8440175
Ligand-binding properties of the two isoforms of the human i...
REMOVE
Summary: 'Insulin binding' – not a demonstrated activity of p85Ξ±.
Reason: GO:0043559 (binding the insulin hormone) is implausible for p85Ξ±, which has no known capacity to bind insulin itself. The cited paper characterizes ligand-binding kinetics of the two insulin-RECEPTOR isoforms and does not assay p85Ξ±. p85Ξ±'s insulin-axis role is SH2-mediated binding to tyrosine-phosphorylated INSR and IRS proteins (GO:0005158, GO:0043560), not binding the hormone.
GO:0043560 insulin receptor substrate binding
IEA
GO_REF:0000107
ACCEPT
Summary: Insulin receptor substrate (IRS) binding.
Reason: Core adaptor facet: p85Ξ± SH2 domains bind tyrosine-phosphorylated IRS proteins (IRS1/2/4), the principal route by which the insulin/IGF axis engages PI3K.
GO:0045944 positive regulation of transcription by RNA polymerase II
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Positive regulation of transcription by RNA Pol II (electronic).
Reason: Indirect transcriptional effects via XBP1/UPR and PI3K-AKT signaling; not a direct DNA/transcription function of p85Ξ±. Retained as non-core.
GO:0045944 positive regulation of transcription by RNA polymerase II
IMP
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Positive regulation of transcription by RNA Pol II (XBP1, IMP).
Reason: p85Ξ± promotes XBP1 nuclear accumulation and induction of UPR target genes, an indirect transcriptional effect. Retained as non-core.
Supporting Evidence:
PMID:20348923
ER stress-dependent accumulation of nuclear XBP-1, decreased induction of UPR
GO:0045944 positive regulation of transcription by RNA polymerase II
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Positive regulation of transcription by RNA Pol II (ISS).
Reason: Indirect transcriptional effect via UPR/PI3K signaling; retained as non-core.
GO:0046326 positive regulation of D-glucose import across plasma membrane
ISS
PMID:8052599
1-Phosphatidylinositol 3-kinase activity is required for ins...
KEEP AS NON CORE
Summary: Positive regulation of glucose import (GLUT4) – insulin action.
Reason: PI3K activation downstream of insulin drives GLUT4-mediated glucose uptake; p85Ξ± is the regulatory subunit enabling this. Downstream metabolic role; retained as non-core.
GO:0046854 phosphatidylinositol phosphate biosynthetic process
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Phosphatidylinositol phosphate biosynthetic process (as PI3K subunit).
Reason: p85Ξ± is an obligate subunit of the class IA PI3K complex that synthesizes PI(3,4,5)P3; it contributes the regulatory/scaffolding activity the reaction depends on but does not itself catalyse it. Retained as non-core (the catalytic step is p110's).
GO:0046854 phosphatidylinositol phosphate biosynthetic process
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Phosphatidylinositol phosphate biosynthetic process (as PI3K subunit).
Reason: p85Ξ± is an obligate subunit of the class IA PI3K complex that synthesizes PI(3,4,5)P3; it contributes the regulatory/scaffolding activity the reaction depends on but does not itself catalyse it. Retained as non-core (the catalytic step is p110's).
GO:0046935 1-phosphatidylinositol-3-kinase regulator activity
IBA
GO_REF:0000033
ACCEPT
Summary: 1-phosphatidylinositol-3-kinase regulator activity – core function (IBA).
Reason: The precise core molecular function of p85Ξ±. Well-supported ancestral (IBD/IBA) assignment across the p85 regulatory-subunit family (PANTHER:PTN008302071) with mouse/rat experimental descendants.
Supporting Evidence:
file:human/PIK3R1/PIK3R1-deep-research-falcon.md
stabilizes p110Ξ±/Ξ²/Ξ΄ while suppressing basal lipid-kinase activity
GO:0046982 protein heterodimerization activity
IEA
GO_REF:0000107
ACCEPT
Summary: Protein heterodimerization activity (p85α–p110 heterodimer).
Reason: Class IA PI3K is an obligate p85–p110 heterodimer; p85Ξ±'s iSH2 mediates this heterodimerization. Correct and relevant.
GO:0048009 insulin-like growth factor receptor signaling pathway
IDA
PMID:7782332
Growth hormone, interferon-gamma, and leukemia inhibitory fa...
KEEP AS NON CORE
Summary: IGF-I receptor signaling pathway.
Reason: p85Ξ± couples the IGF1R/IRS module to PI3K; a specific instance of RTK-driven PI3K signaling. Retained as non-core.
Supporting Evidence:
PMID:7782332
binding of IRS-1 to the 85-kDa regulatory subunit of PI 3'-kinase.
GO:0048009 insulin-like growth factor receptor signaling pathway
IEA
GO_REF:0000120
KEEP AS NON CORE
Summary: IGF-I receptor signaling pathway (electronic).
Reason: Consistent with p85Ξ±'s SH2-mediated coupling to IGF1R/IRS. Retained as non-core.
GO:0048009 insulin-like growth factor receptor signaling pathway
IPI
PMID:7541045
Non-SH2 domains within insulin receptor substrate-1 and SHC ...
KEEP AS NON CORE
Summary: IGF-I receptor signaling pathway (direct interaction).
Reason: Direct p85α–IGF1R interaction underlies its role in IGF signaling.
Supporting Evidence:
PMID:7541045
phosphatidylinositol 3-kinase interact directly and specifically with the IGFIR.
GO:0048471 perinuclear region of cytoplasm
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Perinuclear region of cytoplasm (electronic).
Reason: Low-specificity electronic localization; retained as non-core.
GO:0048471 perinuclear region of cytoplasm
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Perinuclear region of cytoplasm (ISS).
Reason: Low-specificity electronic localization; retained as non-core.
GO:0048661 positive regulation of smooth muscle cell proliferation
IMP
PMID:21321938
Interleukin-18/WNT1-inducible signaling pathway protein-1 si...
KEEP AS NON CORE
Summary: Positive regulation of smooth muscle cell proliferation (IL-18/PI3K).
Reason: PI3K/Akt signaling (with p85Ξ± as regulatory subunit) supports vascular smooth-muscle proliferation downstream of IL-18. Context-specific; retained as non-core.
Supporting Evidence:
PMID:21321938
phosphatidylinositol 3-kinase/Akt-dependent IKK/NF-ΞΊB
GO:0050821 protein stabilization
IDA
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Protein stabilization (p110 stabilization / XBP1).
Reason: p85Ξ± stabilizes its binding partners – canonically the otherwise-unstable p110 catalytic subunit, and in the UPR it stabilizes/promotes nuclear XBP1. A facet of its regulator/adaptor role; retained as non-core.
Supporting Evidence:
PMID:20348923
transcriptional mediator of the unfolded protein response (UPR), in an
GO:0051491 positive regulation of filopodium assembly
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Positive regulation of filopodium assembly (electronic).
Reason: Cytoskeletal role via PI3K/Rho-family signaling; pleiotropic. Retained as non-core.
GO:0051491 positive regulation of filopodium assembly
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Positive regulation of filopodium assembly (ISS).
Reason: Cytoskeletal role via PI3K/Rho-family signaling; pleiotropic. Retained as non-core.
GO:0051497 negative regulation of stress fiber assembly
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Negative regulation of stress fiber assembly (electronic).
Reason: Cytoskeletal remodeling via PI3K/Rho-family signaling; pleiotropic. Retained as non-core.
GO:0051497 negative regulation of stress fiber assembly
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Negative regulation of stress fiber assembly (ISS).
Reason: Cytoskeletal remodeling via PI3K/Rho-family signaling; pleiotropic. Retained as non-core.
GO:0052742 phosphatidylinositol kinase activity
ISS
GO_REF:0000024
REMOVE
Summary: 'Phosphatidylinositol kinase activity' wrongly attributes catalysis to p85Ξ±.
Reason: p85Ξ± is the non-catalytic regulatory subunit; lipid (PI) kinase catalytic activity resides in the p110 catalytic subunit, not p85Ξ±. Assigning catalytic phosphatidylinositol kinase activity to the regulatory subunit is incorrect (the deep-research synthesis and structural literature are explicit that p85Ξ± is not an enzyme). The correct p85Ξ± activity is regulator activity (GO:0046935).
GO:0060396 growth hormone receptor signaling pathway
IDA
PMID:7782332
Growth hormone, interferon-gamma, and leukemia inhibitory fa...
KEEP AS NON CORE
Summary: Growth hormone receptor signaling pathway.
Reason: GH receptor signaling recruits p85Ξ±/PI3K via JAK2/IRS-1 tyrosine phosphorylation. Specific pathway instance; retained as non-core.
Supporting Evidence:
PMID:7782332
binding of IRS-1 to the 85-kDa regulatory subunit of PI 3'-kinase.
GO:0061470 T follicular helper cell differentiation
IDA
PMID:30523347
Transmembrane domain-mediated Lck association underlies byst...
KEEP AS NON CORE
Summary: T follicular helper cell differentiation via ICOS/PI3K.
Reason: ICOS costimulation recruits p85Ξ±/PI3K, and this is required for Tfh development. A specific immune/developmental role; retained as non-core.
Supporting Evidence:
PMID:30523347
required for p85 recruitment to ICOS and subsequent PI3K activation
PMID:30523347
fails to support TFH
GO:0120183 positive regulation of focal adhesion disassembly
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Positive regulation of focal adhesion disassembly (electronic).
Reason: Cytoskeletal/adhesion dynamics via PI3K signaling; pleiotropic. Retained as non-core.
GO:0120183 positive regulation of focal adhesion disassembly
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Positive regulation of focal adhesion disassembly (ISS).
Reason: Cytoskeletal/adhesion dynamics via PI3K signaling; pleiotropic. Retained as non-core.
GO:0140767 enzyme-substrate adaptor activity
ISS
PMID:1322797
Phosphatidylinositol 3-kinase: structure and expression of t...
ACCEPT
Summary: Enzyme-substrate adaptor activity (recruits p110 to substrate/membrane).
Reason: Captures p85Ξ±'s adaptor role bridging the p110 enzyme to its lipid substrate/membrane context via SH2-mediated receptor docking. Consistent with the core adaptor/regulator function.
GO:0141038 phosphatidylinositol 3-kinase activator activity
ISS
GO_REF:0000024
KEEP AS NON CORE
Summary: Phosphatidylinositol 3-kinase activator activity.
Reason: Upon phosphotyrosine engagement p85Ξ± relieves autoinhibition and enables p110 activation – the activating facet of its dual regulator role (it also restrains basal activity). Retained as non-core alongside the core regulator activity.
GO:1900103 positive regulation of endoplasmic reticulum unfolded protein response
IMP
PMID:20348923
A regulatory subunit of phosphoinositide 3-kinase increases ...
KEEP AS NON CORE
Summary: Positive regulation of the ER unfolded protein response (XBP1).
Reason: p85Ξ± promotes the UPR via XBP1 nuclear accumulation and IRE1Ξ±/ATF6 activation; loss attenuates the UPR. Specialized role; retained as non-core.
Supporting Evidence:
PMID:20348923
transcriptional mediator of the unfolded protein response (UPR), in an
GO:1903078 positive regulation of protein localization to plasma membrane
ISS
PMID:8052599
1-Phosphatidylinositol 3-kinase activity is required for ins...
KEEP AS NON CORE
Summary: Positive regulation of protein localization to plasma membrane (ISS).
Reason: Consistent with PI3K-driven membrane recruitment of effectors (e.g. GLUT4 trafficking in insulin action). Downstream; retained as non-core.
GO:1990578 perinuclear endoplasmic reticulum membrane
IEA
GO_REF:0000107
KEEP AS NON CORE
Summary: Perinuclear ER membrane localization (electronic).
Reason: Consistent with the ER-stress/XBP1 role; low-specificity electronic annotation. Retained as non-core.

Core Functions

Non-catalytic regulatory subunit of class IA PI3K: binds and stabilizes the p110 catalytic subunit, restrains its basal lipid-kinase activity, and enables its activation as part of the class IA PI3K heterodimer that produces PI(3,4,5)P3.

Supporting Evidence:
  • PMID:27708159
    KBTBD2 targeted p85Ξ±, the regulatory subunit
  • PMID:19805105
    domain of p85alpha is shown to form a scaffold for the entire enzyme complex
  • PMID:20713702
    preserving the stabilizing interaction between p85Ξ± iSH2 and the adapter-binding domain of p110Ξ±.

Phosphotyrosine-binding adaptor: the SH2 domains dock tyrosine-phosphorylated YXXM motifs on activated receptor tyrosine kinases and on insulin-receptor substrates, recruiting and coupling the PI3K heterodimer to activated receptors.

Supporting Evidence:
  • PMID:20624904
    virtually all human SRC homology 2 (SH2) and phosphotyrosine binding domains
  • PMID:7541045
    phosphatidylinositol 3-kinase interact directly and specifically with the IGFIR.

References

Loading supporting content…

Download this section (compressed HTML)

Deep Research

Falcon

(PIK3R1-deep-research-falcon.md)

Loading supporting content…

Download this section (compressed HTML)

πŸ“š Additional Documentation

Notes

(PIK3R1-notes.md)

Loading supporting content…

Download this section (compressed HTML)

πŸ“„ View Raw YAML

Loading supporting content…

Download this section (compressed HTML)