cGAS-STING Cytosolic DNA Sensing Project

SCOPING BIOLOGY_DOMAINFLAGSHIP

Species: human

cGAS-STING Cytosolic DNA Sensing Project

Bottom line: cGAS senses DNA in the cytosol and makes the second messenger
2'3'-cGAMP, which activates the ER adaptor STING1; STING1 then recruits TBK1 to
phosphorylate IRF3 and switch on type I interferon. Scoped, not yet started as
a project: this page lists ten candidate human genes and the pathway
architecture, but no project-specific review work has been done. Three
candidates already have complete reviews from other projects (STING1, TBK1 and
IFI16, 529 annotations between them). The other seven, including the sensor
CGAS itself, have no gene folder. The next step is just fetch-gene human <GENE> for CGAS, IRF3, TREX1, ENPP1, SAMHD1, IRF7 and IFNB1, then review.

We scoped this because the pathway links innate immunity to autoimmune disease
(AGS, SAVI, lupus), cancer immunotherapy and senescence, and many of its
regulators were described after 2020, so existing GO annotation is likely to lag
the literature.

Overview

The cGAS-STING pathway is a critical innate immune signaling system that detects cytosolic DNA (from pathogens, damaged mitochondria, or genomic instability) and triggers type I interferon responses. Many regulators and disease connections have been discovered 2020+.

Model Species

Primary: Homo sapiens (human)
- Cancer immunotherapy relevance
- Autoimmune disease connections

Core Pathway Architecture

1. DNA Sensing

2. Second Messenger

3. Signal Transduction

4. Negative Regulators

5. Downstream Effectors

Candidate Genes (~15)

Gene UniProt Function
CGAS Q8N884 DNA sensor
STING1 Q86WV6 Adaptor protein
TBK1 Q9UHD2 Kinase
IRF3 Q14653 Transcription factor
TREX1 Q9NSU2 Negative regulator
ENPP1 P22413 cGAMP hydrolase
IFI16 Q16666 DNA sensor
SAMHD1 Q9Y3Z3 dNTPase
IRF7 Q92985 Transcription factor
IFNB1 P01574 Type I interferon

Key Recent Discoveries (2020+)

Disease Relevance

Project Status

Slides