ER-phagy (Selective ER Autophagy) Project

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ER-phagy (Selective ER Autophagy) Project

Overview

ER-phagy is the selective autophagy of endoplasmic reticulum, mediated by specific receptor proteins that link ER membranes to the autophagy machinery. Most receptors were characterized 2017-2023, making this a relatively new field with recent discoveries.

Model Species

Primary: Homo sapiens (human)
- Most receptors characterized in human cells
- Disease relevance (neurodegeneration, viral infection)

Core Pathway Architecture

ER-phagy Receptors

Proteins that bridge ER to autophagosomes via LC3-interacting regions (LIRs):
- FAM134B (RETREG1) - Reticulon-like, sheet ER-phagy
- RTN3 - Reticulon 3, tubular ER
- SEC62 - Translocon-associated, recovER-phagy
- CCPG1 - Cell cycle progression gene 1
- TEX264 - Three-way junction ER-phagy
- ATL3 - Atlastin-3, tubular ER

Core Autophagy Machinery

ER Stress Connection

Candidate Genes (~12-15)

Gene UniProt Function
FAM134B Q9H6L5 ER-phagy receptor (sheets)
RTN3 O95197 ER-phagy receptor (tubules)
SEC62 Q99442 RecovER-phagy receptor
CCPG1 Q9ULG6 ER-phagy receptor
TEX264 Q9Y6I9 ER-phagy receptor
ATL3 Q6DD88 ER-phagy receptor
MAP1LC3B Q9GZQ8 Autophagosome protein
GABARAP O95166 ATG8 family
ULK1 O75385 Autophagy kinase
ATG9A Q7Z3C6 Membrane trafficking

Key Recent Discoveries (2020+)

Disease Relevance

Project Status