PN placement: two ALP rows — ALP|Pre-initiation autophagy signaling|mTORC1 pathway, upstream|Nutrient sensing|V0 lysosomal v-ATPase proton pump component and ALP|Lysosomal catabolism|Regulation of lysosomal environment|Lysosomal acidification|V0 lysosomal v-ATPase proton pump component ; PN-node mapping: identical to ATP6V0A2 — V0 subtype leaves mapped/ok_for_propagation → GO:0046610, GO:0033179; Lysosomal-acidification type → GO:0007042; mTORC1/nutrient-sensing ancestors no_mapping; Pre-initiation class context_only/too_broad (GO:0010506).
Consistency: Consistent. Notes, deep-research-manual, review, PN row and PN-node mapping all treat ATP6V0D1 as the d1 V0-sector subunit acidifying endolysosomal compartments. The review carries the mTORC1/Ragulator role as directly-evidenced (PMID:22053050, IDA) but KEEP_AS_NON_CORE, matching the PN ancestor no_mapping. No contradictions.
PN story / NEW pressure: Unlike ATP6V0A2, ATP6V0D1 GOA already captures the lysosomal-specific terms: GO:0046611 lysosomal V-ATPase complex (IDA, PMID:22053050, ACCEPT) and GO:0007035 vacuolar acidification (IBA, ACCEPT) plus GO:1902600 proton transport. The two PN projected GO terms GO:0007042/GO:0046610 (both verified real via OLS) are therefore "more_specific" but the gene already has equivalent-or-adjacent coverage — review adds NO NEW terms (proposed_new_terms: []). No NEW pressure; the story is already captured. PN nutrient-sensing context is correctly represented only as non-core mTORC1 signaling, not promoted.
Mapping strategy: No change. The node mapping is shared with ATP6V0A2 and stands; ATP6V0D1 does not push the node broader. Conservative no_mapping ancestors correct.