PN placement: 1 row, ALP — Autophagophore initiation and elongation → Class 3 PI3K complex 1, direct → Class 3 PI3K complex 1 component. PN-node mapping: component leaf=mapped→GO:0034271; ancestors context_only (GO:0035032, GO:0016236). Projected:GO:0034271 (goa_status=already_in_goa_exact).
Consistency: Consistent. The review ACCEPTs GO:0034271 (PI3KC3-C1, IBA) and adds a NEW IMP autophagosome-assembly row, matching the PN "C1 component" placement. The review goes beyond PN in flagging the BECN2-specific, non-shared GPCR/GASP1 endolysosomal-degradation branch (GO:1990172, GO:0008333) — PN's single ALP row does not capture that, but the PN Notes explicitly mention "lysosomal degradation of GPCRs," so the two are aligned in spirit, not contradictory.
PN story / NEW pressure: Already captured (and exceeded). PN asserts only C1 membership = projected GO:0034271, already_in_goa_exact and ACCEPTed. The distinctive GPCR-catabolism role (the real "new" biology) is already minted by the review as core GO:1990172. No additional GO term warranted from the PN placement; PN under-represents BECN2 rather than over-reaching.
Mapping strategy: No change. GO:0034271 propagation is safe (already in GOA exact, supported by PMID:28218432 BECN2-ATG14 coiled-coil). Inclusion of BECN2 does not destabilize the node. The review's open question (curate C1/C2 membership as direct vs Beclin-family inference) is a curator nuance, not a mapping-strategy problem.
Evidence alignment: Strong overlap. PN cites the single ScienceDirect "Beclin 2 Functions in Autophagy, Degradation of GPCRs, and Metabolism" = review's central PMID:23954414. Review adds PMID:28218432, 36719945, 37409929, 34152938, 32865519 (none in PN). Convergent on the founding paper; review is far richer.
Verdict: Fully consistent; PN captures the shared C1 role, review additionally captures the BECN2-specific GPCR branch. No edits needed.