Consistency: Fully consistent. Deep research (falcon), review YAML, PN annotation, and PN-node mapping all converge on FBXW2 as a non-catalytic SCF(FBXW2) substrate receptor. Description, core_functions (2x GO:1990756) and the MODIFY of the catalytic GO:0004842→GO:1990756 all match the PN projected term. No contradictions.
PN story / NEW pressure: PN asserts only the generic Cul1-receptor adaptor role, which the review already captures: GO:1990756 is introduced via MODIFY (transferase IEA/TAS) — it is NOT in current GOA, so projection is correctly new_to_goa. Term verified real via OLS. No additional NEW term needed; substrate biology (GCM1, SKP2, β-catenin, TAK1, p65, Moesin, EGFR) is well captured by description/core_functions and the GO:0031146 catabolic process. Conclusion: already captured / ADD GO:1990756 (which the review's MODIFY already proposes).
Mapping strategy: Correct. Group-level GO:1990756 is neither broader nor narrower than the review's core MF; this is the recurring correct call (adaptor activity replacing catalytic transferase). Status/scope appropriate; class-level GO:0061630 correctly held as too_broad. No node change warranted.
Evidence alignment: PN cites only PMID:15340381 (Cardozo & Pagano SCF review) "/ rev" — a family review, not in the review's reference list but consistent with it. Review draws substrate evidence from PMID:23651062 (GCM1/RACK1, HIGH) and PMID:35414786 (SKP2/β-catenin/TAK1, HIGH) plus falcon. Divergence is benign: PN uses the family-level review, the YAML uses gene-specific primary literature.
Verdict: CONSISTENT — no edits. Validated F-box receptor; GO:1990756 ADD already handled by review MODIFY.