ProtNLM2 — OpenScientist adjudication of borderline and disputed predictions

Warnings (4)

OpenScientist adjudication of borderline and disputed ProtNLM2 predictions

← back to ProtNLM2 Evaluation

Bottom line

Every ProtNLM2 prediction the ARGO-ProtNLM-50 review had scored UNC (genuinely
on the fence — "cannot validate or refute from the evidence at hand") was re-tested as an
independent, blinded gene-function hypothesis by an OpenScientist agent (structure +
comparative genomics; the suspected answer was withheld from the prompt). 15 UNC terms
across 11 genes; 10 genes adjudicated (14 terms), 1 gene failed upstream.

The result splits cleanly by prediction type:

This mirrors and sharpens the main evaluation's finding that ProtNLM2 "captures broad
functional categories but lacks resolution": where it names a compartment it is usually
right; where it names a specific mechanism or process it tends to over-generalize from
family/ortholog associations.

A second round then tested the opposite population — the 10 genes whose predictions the
review had disputed (NPI/PLI, "the prediction is wrong") — as the same blinded
hypotheses, to check our own rejections. 9 of 10 were independently confirmed as
misassignments; 1 (GADMO/tbc1d14) was partially overturned
(see the dispute-confirmation
section). Together the two rounds show the adjudicator moves in both directions: it upholds
well-reasoned rejections and catches an over-harsh one.

Round 1 — borderline (UNC) verdicts

Gene UNC prediction(s) Verdict New assessment Why
ARTAN/A0A2U1PS28 chloroplast (GO:0009507) Supported UNC → COR ~78% id to Arabidopsis chloroplastic paralog Q9FNM5 vs ~47% to the mitochondrial one, + chloroplast transit-peptide signature. The GOA/HAMAP mitochondrial annotation is the error.
DROVI/B4MAQ2 cytoplasm (GO:0005737) Supported (incomplete) UNC → COR It is Exportin-5; cytoplasm is valid (obligatory cytoplasmic phase) but undersells a Ran-GTP nucleocytoplasmic shuttle.
CALMI/A0A4W3GVU1 nucleus (GO:0005634) Supported UNC → COR It is ESRP1 (paralog ESRP2 ruled out); human ortholog is IDA-nuclear.
STRCO/Q9L243 deoxyribonucleotide catabolism (GO:0009264) Over-annotated UNC → NPI A generic HAD phosphohydrolase (PF18143 RNA-repair family), not a dNMP-specific 5′-nucleotidase; the process term has no pathway support.
AQUCT/A0A2G9RZF1 single fertilization (GO:0007338) Refuted UNC → NPI A 156-aa CUB-domain-only protein with no serine-protease domain or catalytic triad; real ovochymases are large multidomain S1 proteases. Family over-annotation.
ORYSI/B8BAB0 pollen maturation (GO:0010152) Refuted UNC → NPI Belongs to the PG1β-like cell-wall/pectin BURP clade, not the anther-specific RAFTIN clade that supplies the reproductive precedent. Wrong-subfamily over-generalization.
WHEAT/A0A3B6RKV1 photomorphogenesis, GA signaling, red light, seed germination (GO:0010099/0010476/0010114/0010030) Split UNC → NPI ×4 A catalytically competent JMJ22 co-ortholog (correcting the "KDM5/JARID" mislabel — it is a JMJD6-type histone arginine demethylase), but the four terms are Arabidopsis IMP redundant-pair phenotypes not transferable to wheat by orthology alone.
PHATC/B7FXQ8 salt stress, H₂O₂ (GO:0009651, GO:0042542) Over-annotated UNC → NPI ×2 A bona fide small-HSP/ACD chaperone whose only defensible family process is response to heat; salt is weakly plausible only by cross-kingdom analogy and H₂O₂ is partly contradicted by sHSP precedent.
ORYSJ/Q6YYC5 K63-linked ubiquitination (GO:0070534) Undecidable kept UNC (lead) A real RGLG-family RING E3 ligase, but chain-linkage specificity is E2-determined and not decidable from sequence/orthology — needs an in-vitro chain-linkage assay.
SOYBN/C6T1A2 neg. reg. ABA signaling (GO:0009788) Undecidable kept UNC (lead) A real Q-type (QALGGH) C2H2 zinc-finger TF (MF scaffold solid), but the specific ABA-negative-regulation role is overexpression-only and should not be a direct annotation.
ABRPR/A0A8B8L1Z3 endoplasmic reticulum (GO:0005783) unchanged Run failed 4× with a transient upstream error (Request ID: …); gene-specific, not attempted further.

Round 2 — dispute confirmation (NPI / PLI)

The 10 genes whose predictions the review had disputed were re-tested the same way
(neutral hypothesis: "is this prediction supported or a misassignment?"). This checks whether
our own rejections hold up — and, as ARTAN showed in round 1, an independent agent can overturn
a call.

Gene Disputed prediction (our call) OpenScientist finding Outcome
ARATH/F4JLB7 (RIC7) kinase activity + phosphorylation (PLI) no kinase catalytic domain (LRR-RLP; no VAIK/HRD/DFG) confirmed
MYTGA/A0A8B6GS20 PI3P phosphatase (NPI) catalytically dead pseudophosphatase (MTMR9-like) confirmed
ASPOR/Q2U1U6 O-glycosyl hydrolase (NPI) polysaccharide lyase, not GH (and too small for either) confirmed
WHEAT/F6LAX4 6 animal/mitotic terms (NPI×6) PP2A A scaffold; terms taxon-inappropriate or B56-subunit (not A) confirmed
CAEEL/mcm-4 (A0A061AL94) transcription initiation (NPI) 74-aa MCM4 replicative-helicase fragment confirmed
XENTR/A0A8J0SCI2 DNA-binding TF activator (NPI) naked C2H2 array, no effector domain → direction not sequence-determinable confirmed
DANRE/dnajc6 (A0A8M9QG43) dephosphorylation (NPI) PTEN-fold pseudophosphatase, no intact catalytic site confirmed
DROPS/A0A6I8W8A2 ligase activity (NPI) 169-aa RCC1 β-propeller, no HECT/RING (HERC3 name-transfer) confirmed
XENNA/D3VIU4 ligand-gated ion channel (NPI) periplasmic SBP; iGluR fold homology but no TM pore confirmed
GADMO/tbc1d14 (A0A8C5FPT8) autophagosome (NPI) defensible — human ortholog has a curated IDA overturned → CNN

9 confirmed, 1 overturned. The overturn is the informative case: our review flatly rejected
autophagosome for tbc1d14, but human TBC1D14 (Q9P2M4) carries a direct experimental IDA
autophagosome annotation
(Longatti et al. 2012, PMID:22613832) plus a phylogenetic IBA
(verified independently via QuickGO). The prediction is correct-by-orthology, so it was
reassessed NPI → CNN — with the caveat, on which both analyses agree, that autophagosome is a
secondary, condition-dependent localization (the recycling endosome is primary and the role is
regulatory). The other nine confirmations recapitulate the main evaluation's failure modes:
paralog/pseudoenzyme over-annotation, cross-kingdom / taxon-inappropriate transfer, over-specific
directional child terms, and RefSeq-name-transfer artifacts.

Curation leads beyond the predictions

Three findings warrant fixes to the genes' own reviews, independent of the ProtNLM assessment:

Method & provenance

Each hypothesis was run with just gene-hypothesis-research openscientist <ORG> <GENE>
(3 iterations, 7200 s job timeout), framed neutrally as computational_prediction, with the
suspected answer withheld. No local *-bioinformatics/RESULTS.md was fed to the agent (there
was none for these genes). Full reports and provenance artifacts live under each gene's
genes/<ORG>/<GENE>/<GENE>-hypotheses/<slug>/openscientist.md. Verdicts are wired into the
per-gene *-protnlm-predictions-review.yaml files with verbatim supporting_text quotes.

Caveat: all verdicts rest on computational evidence (sequence, structure, orthology,
comparative genomics) without new wet-lab data; the two COR localization upgrades and the
ARTAN curation lead are strong but should be curator-confirmed.